The FGFR4-G388R polymorphism promotes mitochondrial STAT3 serine phosphorylation to facilitate pituitary growth hormone cell tumorigenesis
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Mitochondrial Stat3, the Need for Design ThinkingLandscape of Familial Isolated and Young-Onset Pituitary Adenomas: Prospective Diagnosis in AIP Mutation Carriers.Metabolic glucose status and pituitary pathology portend therapeutic outcomes in acromegaly.Functional FGFR4 Gly388Arg polymorphism contributes to cancer susceptibility: Evidence from meta-analysis.Regulation and function of signal transducer and activator of transcription 3.Is fibroblast growth factor receptor 4 a suitable target of cancer therapy?FGFR4 profile as a prognostic marker in squamous cell carcinoma of the mouth and oropharynx.Advances in understanding pituitary tumorsToward a new STATe: the role of STATs in mitochondrial function.STAT3 upregulation in pituitary somatotroph adenomas induces growth hormone hypersecretion.Stat3: friend or foe in colitis and colitis-associated cancer?Fibroblast growth factor receptor 4 (FGFR4): a targetable regulator of drug resistance in colorectal cancer.FGFR4 polymorphic alleles modulate mitochondrial respiration: A novel target for somatostatin analog action in pituitary tumorsThe cancer-associated FGFR4-G388R polymorphism enhances pancreatic insulin secretion and modifies the risk of diabetes.A call to ARMS: targeting the PAX3-FOXO1 gene in alveolar rhabdomyosarcoma.Genomic approaches to problems in pituitary neoplasia.STAT3-Mediated Metabolic Reprograming in Cellular Transformation and Implications for Drug ResistanceGenomic analyses identify agents regulating somatotroph and lactotroph functions.FGFR4 polymorphic variants modulate phenotypic features of Cushing disease.A history of pituitary pathology.Honokiol Decreases Lung Cancer Metastasis through Inhibition of the STAT3 Signaling Pathway.Aggressive Pituitary Tumors or Localized Pituitary Carcinomas: Defining Pituitary TumorsThe Role of Mediators of Cell Invasiveness, Motility, and Migration in the Pathogenesis of Silent Corticotroph AdenomasNucleus, Mitochondrion, or Reticulum? STAT3 à La Carte
P2860
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P2860
The FGFR4-G388R polymorphism promotes mitochondrial STAT3 serine phosphorylation to facilitate pituitary growth hormone cell tumorigenesis
description
2011 nî lūn-bûn
@nan
2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
The FGFR4-G388R polymorphism p ...... wth hormone cell tumorigenesis
@ast
The FGFR4-G388R polymorphism p ...... wth hormone cell tumorigenesis
@en
The FGFR4-G388R polymorphism p ...... wth hormone cell tumorigenesis
@nl
type
label
The FGFR4-G388R polymorphism p ...... wth hormone cell tumorigenesis
@ast
The FGFR4-G388R polymorphism p ...... wth hormone cell tumorigenesis
@en
The FGFR4-G388R polymorphism p ...... wth hormone cell tumorigenesis
@nl
prefLabel
The FGFR4-G388R polymorphism p ...... wth hormone cell tumorigenesis
@ast
The FGFR4-G388R polymorphism p ...... wth hormone cell tumorigenesis
@en
The FGFR4-G388R polymorphism p ...... wth hormone cell tumorigenesis
@nl
P2093
P2860
P1433
P1476
The FGFR4-G388R polymorphism p ...... wth hormone cell tumorigenesis
@en
P2093
Axel Ullrich
Shereen Ezzat
Thomas Mayr
Toru Tateno
P2860
P304
P356
10.1371/JOURNAL.PGEN.1002400
P577
2011-12-08T00:00:00Z