The E2F1-3 transcription factors are essential for cellular proliferation. - Wikidata - wikimedia - TriplyDB

The E2F1-3 transcription factors are essential for cellular proliferation.

The E2F1-3 transcription factors are essential for cellular proliferation.

http://www.wikidata.org/entity/Q34102966

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Restriction beyond the restriction point: mitogen requirement for G2 passage Prolactin-induced protein (PIP) regulates proliferation of luminal A type breast cancer cells in an estrogen-independent manner E2F-7: a distinctive E2F family member with an unusual organization of DNA-binding domains Cdc2: a monopotent or pluripotent CDK?Human TFDP3, a novel DP protein, inhibits DNA binding and transactivation by E2F Characterization of E2F8, a novel E2F-like cell-cycle regulated repressor of E2F-activated transcription.Functional high-throughput screening identifies the miR-15 microRNA family as cellular restriction factors for Salmonella infection The transcriptional repressor gene Mad3 is a novel target for regulation by E2F1 Rapamycin potentiates transforming growth factor beta-induced growth arrest in nontransformed, oncogene-transformed, and human cancer cells.Interaction of YY1 with E2Fs, mediated by RYBP, provides a mechanism for specificity of E2F function E2F7, a novel E2F featuring DP-independent repression of a subset of E2F-regulated genes miR-17 and miR-20a temper an E2F1-induced G1 checkpoint to regulate cell cycle progression E2f1, E2f2, and E2f3 control E2F target expression and cellular proliferation via a p53-dependent negative feedback loop Epstein-Barr virus LMP1 blocks p16INK4a-RB pathway by promoting nuclear export of E2F4/5 Regulation of E2F1 by BRCT domain-containing protein TopBP1 Retinoblastoma family genes NPAT expression is regulated by E2F and is essential for cell cycle progression E2F mediates cell cycle-dependent transcriptional repression in vivo by recruitment of an HDAC1/mSin3B corepressor complex Impaired DNA replication within progenitor cell pools promotes leukemogenesis.The E2F2 transcription factor sustains hepatic glycerophospholipid homeostasis in mice A role for E2F activities in determining the fate of Myc-induced lymphomagenesis Targeted Cancer Therapy: Vital Oncogenes and a New Molecular Genetic Paradigm for Cancer Initiation Progression and Treatment Tumor-suppressive miR-34a induces senescence-like growth arrest through modulation of the E2F pathway in human colon cancer cells Aberrant regulation of survivin by the RB/E2F family of proteins The association of GSK3 beta with E2F1 facilitates nerve growth factor-induced neural cell differentiation Cloning and characterization of mouse E2F8, a novel mammalian E2F family member capable of blocking cellular proliferation Identification and characterization of E2F7, a novel mammalian E2F family member capable of blocking cellular proliferation The DP-1 transcription factor is required for keratinocyte growth and epidermal stratification E2F-3B is a physiological target of cyclin A E2F7 represses a network of oscillating cell cycle genes to control S-phase progression TGFβ and CCN2/CTGF mediate actin related gene expression by differential E2F1/CREB activation.Division of labour between Myc and G1 cyclins in cell cycle commitment and pace control CREB inhibits AP-2alpha expression to regulate the malignant phenotype of melanoma.Genome-wide differential gene expression in immortalized DF-1 chicken embryo fibroblast cell line Contrasting roles of E2F2 and E2F3 in cardiac neovascularization.A functional genetic screen identifies TFE3 as a gene that confers resistance to the anti-proliferative effects of the retinoblastoma protein and transforming growth factor-beta.Cell size checkpoint control by the retinoblastoma tumor suppressor pathway Rb-mediated neuronal differentiation through cell-cycle-independent regulation of E2f3a.Context-dependent requirement for dE2F during oncogenic proliferation Identification of a novel risk locus for multiple sclerosis at 13q31.3 by a pooled genome-wide scan of 500,000 single nucleotide polymorphisms

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The E2F1-3 transcription factors are essential for cellular proliferation.

http://www.wikidata.org/entity/Q34102966

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2001 nî lūn-bûn

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2001 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2001 թվականի նոյեմբերին հրատարակված գիտական հոդված

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The E2F1-3 transcription factors are essential for cellular proliferation.

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The E2F1-3 transcription factors are essential for cellular proliferation.

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The E2F1-3 transcription factors are essential for cellular proliferation.

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The E2F1-3 transcription factors are essential for cellular proliferation.

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The E2F1-3 transcription factors are essential for cellular proliferation.

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Chong GT

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