Curcumin inhibits hepatic protein-tyrosine phosphatase 1B and prevents hypertriglyceridemia and hepatic steatosis in fructose-fed rats. - Wikidata - wikimedia - TriplyDB

Curcumin inhibits hepatic protein-tyrosine phosphatase 1B and prevents hypertriglyceridemia and hepatic steatosis in fructose-fed rats.

Curcumin inhibits hepatic protein-tyrosine phosphatase 1B and prevents hypertriglyceridemia and hepatic steatosis in fructose-fed rats.

http://www.wikidata.org/entity/Q34103434

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Herbal products: benefits, limits, and applications in chronic liver disease Herbal medicines for the treatment of nonalcoholic steatohepatitis: current scenario and future prospects Natural products possessing protein tyrosine phosphatase 1B (PTP1B) inhibitory activity found in the last decades Simple sugar intake and hepatocellular carcinoma: epidemiological and mechanistic insight Reactive oxygen species-induced TXNIP drives fructose-mediated hepatic inflammation and lipid accumulation through NLRP3 inflammasome activation High Dietary Fructose: Direct or Indirect Dangerous Factors Disturbing Tissue and Organ Functions Hepatic metabolic effects of Curcuma longa extract supplement in high-fructose and saturated fat fed rats Temporal relationship between diet-induced steatosis and onset of insulin/leptin resistance in male Wistar rats.ACE Reduces Metabolic Abnormalities in a High-Fat Diet Mouse Model The Role of PTP1B O-GlcNAcylation in Hepatic Insulin Resistance Fish oil and indomethacin in combination potently reduce dyslipidemia and hepatic steatosis in LDLR(-/-) mice.Quercetin Preserves β -Cell Mass and Function in Fructose-Induced Hyperinsulinemia through Modulating Pancreatic Akt/FoxO1 Activation Modest fructose beverage intake causes liver injury and fat accumulation in marginal copper deficient rats.Cinnamaldehyde and allopurinol reduce fructose-induced cardiac inflammation and fibrosis by attenuating CD36-mediated TLR4/6-IRAK4/1 signaling to suppress NLRP3 inflammasome activation.Novel curcumin derivative CNB-001 mitigates obesity-associated insulin resistance.Why it is necessary to translate curcumin into clinical practice for the prevention and treatment of metabolic syndrome?Curcumin and liver disease.A comprehensive overview of hepatoprotective natural compounds: mechanism of action and clinical perspectives.Turmeric Supplementation Improves Serum Glucose Indices and Leptin Levels in Patients with Nonalcoholic Fatty Liver Diseases.Curcumin reverses the depressive-like behavior and insulin resistance induced by chronic mild stress.Dose-related antihyperglycemic and hypolipidemic effects of two novel thiazolidin-4-ones in a rodent model of metabolic syndrome.Curcumin attenuates palmitate-induced apoptosis in MIN6 pancreatic β-cells through PI3K/Akt/FoxO1 and mitochondrial survival pathways.Curcumin and insulin resistance-Molecular targets and clinical evidences.Turmeric curcumin inhibits entry of all hepatitis C virus genotypes into human liver cells.Novel role of mitochondrial manganese superoxide dismutase in STAT3 dependent pluripotency of mouse embryonic stem cells.Decrease of microRNA-122 causes hepatic insulin resistance by inducing protein tyrosine phosphatase 1B, which is reversed by licorice flavonoid.Protein tyrosine phosphatase 1B inhibitors from natural sources.Betaine recovers hypothalamic neural injury by inhibiting astrogliosis and inflammation in fructose-fed rats.Activation of Nrf2 signaling by natural products-can it alleviate diabetes?Inducible liver-specific knockdown of protein tyrosine phosphatase 1B improves glucose and lipid homeostasis in adult mice.Curcumin inhibits hyperlipidemia and hepatic fat accumulation in high-fructose-fed male Wistar rats.Curcumin protects against fructose-induced podocyte insulin signaling impairment through upregulation of miR-206.Ferulic acid exerts its antidiabetic effect by modulating insulin-signalling molecules in the liver of high-fat diet and fructose-induced type-2 diabetic adult male rat.Molecular Mechanisms Underlying Curcumin-Mediated Therapeutic Effects in Type 2 Diabetes and Cancer.Curcumin, Cardiometabolic Health and Dementia Paeoniflorin Ameliorates Fructose-Induced Insulin Resistance and Hepatic Steatosis by Activating LKB1/AMPK and AKT Pathways

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P2860

Curcumin inhibits hepatic protein-tyrosine phosphatase 1B and prevents hypertriglyceridemia and hepatic steatosis in fructose-fed rats.

http://www.wikidata.org/entity/Q34103434

description

2010 nî lūn-bûn

@nan

2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Curcumin inhibits hepatic prot ...... teatosis in fructose-fed rats.

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Curcumin inhibits hepatic prot ...... teatosis in fructose-fed rats.

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Curcumin inhibits hepatic prot ...... teatosis in fructose-fed rats.

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Curcumin inhibits hepatic prot ...... teatosis in fructose-fed rats.

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Curcumin inhibits hepatic prot ...... teatosis in fructose-fed rats.

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Curcumin inhibits hepatic prot ...... teatosis in fructose-fed rats.

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Curcumin inhibits hepatic prot ...... teatosis in fructose-fed rats.

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Curcumin inhibits hepatic prot ...... teatosis in fructose-fed rats.

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Jian-Mei Li

http://www.w3.org/2001/XMLSchema#string

Ling-Dong Kong

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Qing-Hua Hu

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Yu-Cheng Li

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P2860

Tyrosine dephosphorylation and deactivation of insulin receptor substrate-1 by protein-tyrosine phosphatase 1B. Possible facilitation by the formation of a ternary complex with the Grb2 adaptor protein

Protein-tyrosine phosphatase-1B negatively regulates insulin signaling in l6 myocytes and Fao hepatoma cells

PTP1B regulates leptin signal transduction in vivo

Fructose-induced leptin resistance exacerbates weight gain in response to subsequent high-fat feeding

Fructose consumption as a risk factor for non-alcoholic fatty liver disease

A RAPID METHOD OF TOTAL LIPID EXTRACTION AND PURIFICATION

Curcumin eliminates leptin's effects on hepatic stellate cell activation via interrupting leptin signaling

Protein-tyrosine phosphatase 1B as new activator for hepatic lipogenesis via sterol regulatory element-binding protein-1 gene expression

Suppressor of cytokine signaling 1 (SOCS-1) and SOCS-3 cause insulin resistance through inhibition of tyrosine phosphorylation of insulin receptor substrate proteins by discrete mechanisms

PTP1B antisense oligonucleotide lowers PTP1B protein, normalizes blood glucose, and improves insulin sensitivity in diabetic mice.

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10.1002/HEP.23524

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5

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41896732

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20222050

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