about
Phase and antigenic variation in bacteriaPathogenesis of Helicobacter pylori infectionColonize, evade, flourish: how glyco-conjugates promote virulence of Helicobacter pyloriPeriplasmic cleavage and modification of the 1-phosphate group of Helicobacter pylori lipid AHelicobacter pylori does not require Lewis X or Lewis Y expression to colonize C3H/HeJ mice.Identification and characterization of Helicobacter pylori genes essential for gastric colonizationA novel 3-deoxy-D-manno-octulosonic acid (Kdo) hydrolase that removes the outer Kdo sugar of Helicobacter pylori lipopolysaccharide.Helicobacter pylori lipopolysaccharide modification, Lewis antigen expression, and gastric colonization are cholesterol-dependent.Comparative genomics and proteomics of Helicobacter mustelae, an ulcerogenic and carcinogenic gastric pathogen.Phenotypic variation of Helicobacter pylori isolates from geographically distinct regions detected by lectin typingAccuracy of Helicobacter pylori diagnostic tests in patients with bleeding peptic ulcer: a systematic review and meta-analysis.Relationship of anti-Lewis x and anti-Lewis y antibodies in serum samples from gastric cancer and chronic gastritis patients to Helicobacter pylori-mediated autoimmunityRemoval of the outer Kdo from Helicobacter pylori lipopolysaccharide and its impact on the bacterial surfacepathogenesis of Helicobacter pylori-induced gastric inflammation.Structural studies of FlaA1 from Helicobacter pylori reveal the mechanism for inverting 4,6-dehydratase activity.O-antigen structural variation: mechanisms and possible roles in animal/plant-microbe interactions.Helicobacter pylori with a truncated lipopolysaccharide O chain fails to induce gastritis in SCID mice injected with splenocytes from wild-type C57BL/6J mice.Pathogens target DC-SIGN to influence their fate DC-SIGN functions as a pathogen receptor with broad specificity.Helicobacter pylori vs coronary heart disease - searching for connections.Persistent bacterial infections: the interface of the pathogen and the host immune system.Anti-Lewis X antibody promotes Helicobacter pylori adhesion to gastric epithelial cellsThe Effects of Hormones and Vaginal Microflora on the Glycome of the Female Genital Tract: Cervical-Vaginal FluidInduction of Mincle by Helicobacter pylori and consequent anti-inflammatory signaling denote a bacterial survival strategyImplication of antigenic conversion of Helicobacter pylori lipopolysaccharides that involve interaction with surfactant protein D.Protein glycosylation in diverse cell systems: implications for modification and analysis of recombinant proteins.Expression of Lewis(b) blood group antigen in Helicobacter pylori does not interfere with bacterial adhesion propertyThe schistosome in the mammalian host: understanding the mechanisms of adaptation.Carbohydrate-dependent defense mechanisms against Helicobacter pylori infectionRoles of gastric mucin-type O-glycans in the pathogenesis of Helicobacter pylori infection.Selective upregulation of endothelial E-selectin in response to Helicobacter pylori-induced gastritis.Relationship between Helicobacter pylori virulence factors and regulatory cytokines as predictors of clinical outcome.Helicobacter pylori: phenotypes, genotypes and virulence genes.Strategies used by helicobacter pylori to establish persistent infectionComparative analysis of the interaction of Helicobacter pylori with human dendritic cells, macrophages, and monocytes.Limited role of lipopolysaccharide Lewis antigens in adherence of Helicobacter pylori to the human gastric epithelium.Induction of cyclooxygenase 2 by Escherichia coli but not Helicobacter pylori lipopolysaccharide in gastric epithelial cells in vitro.The Helicobacter heilmannii hofE and hofF Genes are Essential for Colonization of the Gastric Mucosa and Play a Role in IL-1β-Induced Gastric MUC13 Expression.Comparison of the Serological Reactivity of Lipopolysaccharides from Japanese and Western Strains of Helicobacter pylori to Sera from H. pylori-Positive Humans.Influence of monoclonal anti-Lewis b, anti-H type 1, and anti-sialyl Lewis x antibodies on binding of Helicobacter pylori to MUC1 mucin.Helicobacter pylori modulates the T helper cell 1/T helper cell 2 balance through phase-variable interaction between lipopolysaccharide and DC-SIGN.
P2860
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P2860
description
2000 nî lūn-bûn
@nan
2000 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Why Helicobacter pylori has Lewis antigens.
@ast
Why Helicobacter pylori has Lewis antigens.
@en
Why Helicobacter pylori has Lewis antigens.
@nl
type
label
Why Helicobacter pylori has Lewis antigens.
@ast
Why Helicobacter pylori has Lewis antigens.
@en
Why Helicobacter pylori has Lewis antigens.
@nl
prefLabel
Why Helicobacter pylori has Lewis antigens.
@ast
Why Helicobacter pylori has Lewis antigens.
@en
Why Helicobacter pylori has Lewis antigens.
@nl
P2093
P1476
Why Helicobacter pylori has Lewis antigens.
@en
P2093
Appelmelk BJ
Monteiro MA
Vandenbroucke-Grauls CM
P304
P356
10.1016/S0966-842X(00)01875-8
P577
2000-12-01T00:00:00Z