Meganucleases can restore the reading frame of a mutated dystrophin.
about
In Vivo Delivery Systems for Therapeutic Genome EditingTargeted gene disruption to cure HIV5'-Cytosine-Phosphoguanine (CpG) Methylation Impacts the Activity of Natural and Engineered MeganucleasesAdvances in gene therapy for muscular dystrophiesAnimal models of Duchenne muscular dystrophy: from basic mechanisms to gene therapyRescuing the failing heart by targeted gene transferHigh throughput screening in duchenne muscular dystrophy: from drug discovery to functional genomicsCurrent status of pharmaceutical and genetic therapeutic approaches to treat DMD.Homing endonucleases: from genetic anomalies to programmable genomic clippersIn vivo genome editing improves muscle function in a mouse model of Duchenne muscular dystrophy.Reading frame correction by targeted genome editing restores dystrophin expression in cells from Duchenne muscular dystrophy patients.Targeting sarcoplasmic reticulum calcium ATPase by gene therapy.Genome Editing Gene Therapy for Duchenne Muscular Dystrophy.Current Translational Research and Murine Models For Duchenne Muscular Dystrophy.The Development of TALE Nucleases for Biotechnology.Current advances in cell therapy strategies for muscular dystrophies.Homing endonucleases: from microbial genetic invaders to reagents for targeted DNA modification.Cell-matrix interactions in muscle disease.Molecular scissors for in situ cellular repair.Repair or replace? Exploiting novel gene and cell therapy strategies for muscular dystrophies.Gene-based therapies of neuromuscular disorders: an update and the pivotal role of patient organizations in their discovery and implementation.New developments in the use of gene therapy to treat Duchenne muscular dystrophy.Genetic correction using engineered nucleases for gene therapy applications.Emerging gene editing strategies for Duchenne muscular dystrophy targeting stem cells.Gene therapies that restore dystrophin expression for the treatment of Duchenne muscular dystrophy.Male-sterile maize plants produced by targeted mutagenesis of the cytochrome P450-like gene (MS26) using a re-designed I-CreI homing endonuclease.Endonucleases: tools to correct the dystrophin gene.Genome engineering: a new approach to gene therapy for neuromuscular disorders.An integration-defective lentivirus-based resource for site-specific targeting of an edited safe-harbour locus in the human genome.Venturing into the New Science of NucleasesRational design of a serum-free culture medium for the growth of human myoblasts destined to cell therapy
P2860
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P2860
Meganucleases can restore the reading frame of a mutated dystrophin.
description
2010 nî lūn-bûn
@nan
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Meganucleases can restore the reading frame of a mutated dystrophin.
@ast
Meganucleases can restore the reading frame of a mutated dystrophin.
@en
Meganucleases can restore the reading frame of a mutated dystrophin.
@nl
type
label
Meganucleases can restore the reading frame of a mutated dystrophin.
@ast
Meganucleases can restore the reading frame of a mutated dystrophin.
@en
Meganucleases can restore the reading frame of a mutated dystrophin.
@nl
prefLabel
Meganucleases can restore the reading frame of a mutated dystrophin.
@ast
Meganucleases can restore the reading frame of a mutated dystrophin.
@en
Meganucleases can restore the reading frame of a mutated dystrophin.
@nl
P2093
P356
P1433
P1476
Meganucleases can restore the reading frame of a mutated dystrophin.
@en
P2093
C Pichavant
J P Tremblay
J Rousseau
P Chapdelaine
P2860
P2888
P304
P356
10.1038/GT.2010.26
P577
2010-04-15T00:00:00Z
P5875
P6179
1023491052