Mice with an absent stress response are protected against ischemic renal injury. - Wikidata - wikimedia - TriplyDB

Mice with an absent stress response are protected against ischemic renal injury.

Mice with an absent stress response are protected against ischemic renal injury.

http://www.wikidata.org/entity/Q34111676

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Albumin stimulates renal tubular inflammation through an HSP70-TLR4 axis in mice with early diabetic nephropathy.Heat shock factor 1 induces crystallin-αB to protect against cisplatin nephrotoxicity.Heat shock proteins in the kidney.

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Mice with an absent stress response are protected against ischemic renal injury.

http://www.wikidata.org/entity/Q34111676

description

2014 nî lūn-bûn

@nan

2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

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name

Mice with an absent stress response are protected against ischemic renal injury.

@ast

Mice with an absent stress response are protected against ischemic renal injury.

@en

Mice with an absent stress response are protected against ischemic renal injury.

@nl

type

label

Mice with an absent stress response are protected against ischemic renal injury.

@ast

Mice with an absent stress response are protected against ischemic renal injury.

@en

Mice with an absent stress response are protected against ischemic renal injury.

@nl

prefLabel

Mice with an absent stress response are protected against ischemic renal injury.

@ast

Mice with an absent stress response are protected against ischemic renal injury.

@en

Mice with an absent stress response are protected against ischemic renal injury.

@nl

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P1433

Kidney International

P1476

Mice with an absent stress response are protected against ischemic renal injury.

@en

P2093

Calvin B Williams

http://www.w3.org/2001/XMLSchema#string

Dipica Haribhai

http://www.w3.org/2001/XMLSchema#string

Melody Miller

http://www.w3.org/2001/XMLSchema#string

Rajasree Sreedharan

http://www.w3.org/2001/XMLSchema#string

Scott K Van Why

http://www.w3.org/2001/XMLSchema#string

Shaoying Chen

http://www.w3.org/2001/XMLSchema#string

P2860

HSF1 is required for extra-embryonic development, postnatal growth and protection during inflammatory responses in mice

The chaperone function of hsp70 is required for protection against stress-induced apoptosis

Regulatory T cells contribute to the protective effect of ischemic preconditioning in the kidney.

Regulatory T cells suppress innate immunity in kidney ischemia-reperfusion injury

NKT cell activation mediates neutrophil IFN-gamma production and renal ischemia-reperfusion injury.

Prior heat stress inhibits apoptosis in adenosine triphosphate-depleted renal tubular cells.

Heat-shock proteins, molecular chaperones, and the stress response: evolutionary and ecological physiology.

Selective renal overexpression of human heat shock protein 27 reduces renal ischemia-reperfusion injury in mice.

Mouse heat shock transcription factor 1 deficiency alters cardiac redox homeostasis and increases mitochondrial oxidative damage

Activation of heat-shock transcription factor by graded reductions in renal ATP, in vivo, in the rat.

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515-524

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scholarly article

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10.1038/KI.2014.73

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3

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86

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