Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis. - Wikidata - wikimedia - TriplyDB

Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

http://www.wikidata.org/entity/Q34112184

about

Smoking and increased Alzheimer's disease risk: a review of potential mechanisms Acyclovir or Aβ42 peptides attenuate HSV-1-induced miRNA-146a levels in human primary brain cells.Alpha-synuclein repeat variants and survival in Parkinson's disease Microglial KCa3.1 Channels as a Potential Therapeutic Target for Alzheimer's Disease Glycogen synthase kinase 3: a point of integration in Alzheimer's disease and a therapeutic target?Correlation of Alzheimer disease neuropathologic changes with cognitive status: a review of the literature β-Amyloid oligomers in aging and Alzheimer's disease.Phosphorylation of tau protein as the link between oxidative stress, mitochondrial dysfunction, and connectivity failure: implications for Alzheimer's disease.The role of inflammatory processes in Alzheimer's disease.Oxidative Stress and Aberrant Cell Cycle in Alzheimer's Disease Lymphocytes: Diagnostic Prospects.Intracerebroventricular Streptozotocin Injections as a Model of Alzheimer's Disease: in Search of a Relevant Mechanism.Involvement of insulin resistance in D-galactose-induced age-related dementia in rats: Protective role of metformin and saxagliptin.Smoking cessation and Alzheimer's disease: facts, fallacies and promise.The Anti-Amyloid-β and Neuroprotective Properties of a Novel Tricyclic Pyrone Molecule.Amyloid Beta and tau proteins as therapeutic targets for Alzheimer's disease treatment: rethinking the current strategy.Phosphorylation of tau protein at sites Ser(396-404) is one of the earliest events in Alzheimer's disease and Down syndrome.Mortality and morbidity hazards associated with cognitive status in seniors: a Canadian population prospective cohort study.Effect of smoking cessation on the risk of dementia: a longitudinal study

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Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

http://www.wikidata.org/entity/Q34112184

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2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

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Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

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Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

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Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

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Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

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Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

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Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

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Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

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Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

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Expert Review of Neurotherapeutics: a key contribution to decision making in the treatment of neurologic and neuropsychiatric disorders

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Causes versus effects: the increasing complexities of Alzheimer's disease pathogenesis.

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Gustavo Basurto-Islas

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Hyoung-gon Lee

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Rudy J Castellani

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Siddhartha Mondragón-Rodríguez

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P2860

Cellular prion protein mediates impairment of synaptic plasticity by amyloid-beta oligomers

Caspase cleavage of tau: linking amyloid and neurofibrillary tangles in Alzheimer's disease

Hyperphosphorylation induces self-assembly of tau into tangles of paired helical filaments/straight filaments

Apolipoprotein E and its receptors in Alzheimer's disease: pathways, pathogenesis and therapy

Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

A single mutation at the sheet switch region results in conformational changes favoring lambda6 light-chain fibrillogenesis

Neuropathological stageing of Alzheimer-related changes

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

A presenilin-1-dependent gamma-secretase-like protease mediates release of Notch intracellular domain

Presenilin is required for activity and nuclear access of Notch in Drosophila

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683-691

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10.1586/ERN.10.27

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5

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10

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Mark Anthony Smith

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247850659

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20420489

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Alzheimer's disease

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2922904

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