Quiescent haematopoietic stem cells are activated by IFN-gamma in response to chronic infection. - Wikidata - wikimedia - TriplyDB

Quiescent haematopoietic stem cells are activated by IFN-gamma in response to chronic infection.

Quiescent haematopoietic stem cells are activated by IFN-gamma in response to chronic infection.

http://www.wikidata.org/entity/Q34112414

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Human Peripheral CD4(+) Vδ1(+) γδT Cells Can Develop into αβT Cells Hypercholesterolemia Tunes Hematopoietic Stem/Progenitor Cells for Inflammation and Atherosclerosis Cutting the brakes on hematopoietic regeneration by blocking TGFβ to limit chemotherapy-induced myelosuppression DNA Damage Response in Hematopoietic Stem Cell Ageing Bone Marrow T Cells and the Integrated Functions of Recirculating and Tissue-Resident Memory T Cells Single-cell analysis tools for drug discovery and development The regulation of inflammation by interferons and their STATs Differential effects of type I and II interferons on myeloid cells and resistance to intracellular bacterial infections The complex and central role of interferon-γ in graft-versus-host disease and graft-versus-tumor activity Hematopoietic stem cell niche maintenance during homeostasis and regeneration Inflammatory modulation of HSCs: viewing the HSC as a foundation for the immune response Diversification of dendritic cell subsets: Emerging roles for STAT proteins TLRs control hematopoiesis during infection Infection-induced changes in hematopoiesis Paneth cells in intestinal homeostasis and tissue injury Drosophila E-cadherin functions in hematopoietic progenitors to maintain multipotency and block differentiation Modulation of bleomycin-induced lung fibrosis by pegylated hyaluronidase and dopamine receptor antagonist in mice Extramedullary myelopoiesis in malaria depends on mobilization of myeloid-restricted progenitors by IFN-γ induced chemokines Hematopoietic Stem Cell Regulation by Type I and II Interferons in the Pathogenesis of Acquired Aplastic Anemia Stress and Non-Stress Roles of Inflammatory Signals during HSC Emergence and Maintenance Inflamm-Aging of Hematopoiesis, Hematopoietic Stem Cells, and the Bone Marrow Microenvironment Inflammation Shapes Stem Cells and Stemness during Infection and Beyond Correlates of tuberculosis risk: predictive biomarkers for progression to active tuberculosis Aging-associated inflammation promotes selection for adaptive oncogenic events in B cell progenitors Systematic tracking of altered haematopoiesis during sporozoite-mediated malaria development reveals multiple response points Dynamic variation in cycling of hematopoietic stem cells in steady state and inflammation M-CSF instructs myeloid lineage fate in single haematopoietic stem cells Fgd5 identifies hematopoietic stem cells in the murine bone marrow.Systemic inoculation of Escherichia coli causes emergency myelopoiesis in zebrafish larval caudal hematopoietic tissue Transient activation of hematopoietic stem and progenitor cells by IFNγ during acute bacterial infection.Improved HSC reconstitution and protection from inflammatory stress and chemotherapy in mice lacking granzyme B.The bone marrow at the crossroads of blood and immunity The hematopoietic stem cell niche.Ubiquitination of hnRNPA1 by TRAF6 links chronic innate immune signaling with myelodysplasia.mTORC1 controls the adaptive transition of quiescent stem cells from G0 to G(Alert)The journey from stem cell to macrophage.Infection mobilizes hematopoietic stem cells through cooperative NOD-like receptor and Toll-like receptor signaling.Chronic variable stress activates hematopoietic stem cells.T cells from Programmed Death-1 deficient mice respond poorly to Mycobacterium tuberculosis infection Conversion of danger signals into cytokine signals by hematopoietic stem and progenitor cells for regulation of stress-induced hematopoiesis.

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Quiescent haematopoietic stem cells are activated by IFN-gamma in response to chronic infection.

http://www.wikidata.org/entity/Q34112414

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Quiescent haematopoietic stem ...... response to chronic infection.

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Quiescent haematopoietic stem ...... response to chronic infection.

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Quiescent haematopoietic stem ...... response to chronic infection.

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type

label

Quiescent haematopoietic stem ...... response to chronic infection.

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Quiescent haematopoietic stem ...... response to chronic infection.

@en

Quiescent haematopoietic stem ...... response to chronic infection.

@nl

prefLabel

Quiescent haematopoietic stem ...... response to chronic infection.

@ast

Quiescent haematopoietic stem ...... response to chronic infection.

@en

Quiescent haematopoietic stem ...... response to chronic infection.

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Quiescent haematopoietic stem ...... response to chronic infection.

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David C Weksberg

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Katherine Y King

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Nathan C Boles

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P2860

Functional role of type I and type II interferons in antiviral defense

Flk-2 is a marker in hematopoietic stem cell differentiation: a simple method to isolate long-term stem cells

Molecular signatures of proliferation and quiescence in hematopoietic stem cells

Mechanisms of type-I- and type-II-interferon-mediated signalling

Adult hematopoietic stem and progenitor cells require either Lyl1 or Scl for survival

Identification of genes differentially regulated by interferon alpha, beta, or gamma using oligonucleotide arrays

SLAM family receptors distinguish hematopoietic stem and progenitor cells and reveal endothelial niches for stem cells

Targeted disruption of the mouse Stat1 gene results in compromised innate immunity to viral disease

Hematopoietic stem cells do not engraft with absolute efficiencies

Aging hematopoietic stem cells decline in function and exhibit epigenetic dysregulation.

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10.1038/NATURE09135

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7299

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Margaret A Goodell

Megan T Baldridge

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2010-06-01T00:00:00Z

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44660785

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1005476743

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20535209

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2935898

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