Staphylococcus aureus agr and sarA functions are required for invasive infection but not inflammatory responses in the lung. - Wikidata - wikimedia - TriplyDB

Staphylococcus aureus agr and sarA functions are required for invasive infection but not inflammatory responses in the lung.

Staphylococcus aureus agr and sarA functions are required for invasive infection but not inflammatory responses in the lung.

http://www.wikidata.org/entity/Q34116496

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Castanea sativa (European Chestnut) Leaf Extracts Rich in Ursene and Oleanene Derivatives Block Staphylococcus aureus Virulence and Pathogenesis without Detectable Resistance Quorum-sensing regulation in staphylococci-an overview Host airway proteins interact with Staphylococcus aureus during early pneumonia Staphylococcus aureus elicits marked alterations in the airway proteome during early pneumonia.Importance of the global regulators Agr and SaeRS in the pathogenesis of CA-MRSA USA300 infection.The SaeR/S gene regulatory system induces a pro-inflammatory cytokine response during Staphylococcus aureus infection.Peptide signaling in the staphylococci.Nox2 modification of LDL is essential for optimal apolipoprotein B-mediated control of agr type III Staphylococcus aureus quorum-sensing.Relationship of plcR-regulated factors to Bacillus endophthalmitis virulence.Establishment of a bacterial infection model using the European honeybee, Apis mellifera L.Surface proteins and exotoxins are required for the pathogenesis of Staphylococcus aureus pneumonia.Erosion from Staphylococcus aureus biofilms grown under physiologically relevant fluid shear forces yields bacterial cells with reduced avidity to collagen Evolutionary Trade-Offs Underlie the Multi-faceted Virulence of Staphylococcus aureus Staphylococcus aureus Inhibits IL-8 Responses Induced by Pseudomonas aeruginosa in Airway Epithelial Cells.Serum Lipoproteins Are Critical for Pulmonary Innate Defense against Staphylococcus aureus Quorum Sensing.Random mutagenesis and topology analysis of the autoinducing peptide biosynthesis proteins in Staphylococcus aureus.Immunopathogenesis of Staphylococcus aureus pulmonary infection.Cell surface O-glycans limit Staphylococcus aureus adherence to corneal epithelial cells.Evolution and pathogenesis of Staphylococcus aureus: lessons learned from genotyping and comparative genomics.Neutrophil depletion causes a fatal defect in murine pulmonary Staphylococcus aureus clearance.RpiRc Is a Pleiotropic Effector of Virulence Determinant Synthesis and Attenuates Pathogenicity in Staphylococcus aureus.Innate immune response of corneal epithelial cells to Staphylococcus aureus infection: role of peptidoglycan in stimulating proinflammatory cytokine secretion Inactivation of a bacterial virulence pheromone by phagocyte-derived oxidants: new role for the NADPH oxidase in host defense Targeting agr- and agr-Like quorum sensing systems for development of common therapeutics to treat multiple gram-positive bacterial infections.Investigational therapies targeting quorum-sensing for the treatment of Staphylococcus aureus infections.Virulence factor targeting of the bacterial pathogen Staphylococcus aureus for vaccine and therapeutics.A human pathogenic bacterial infection model using the two-spotted cricket, Gryllus bimaculatus.Impact of Staphylococcus aureus regulatory mutations that modulate biofilm formation in the USA300 strain LAC on virulence in a murine bacteremia model.Staphylococcus aureus AgrA binding to the RNAIII-agr regulatory region.TLR2 is mobilized into an apical lipid raft receptor complex to signal infection in airway epithelial cells.Dynamic interaction between airway epithelial cells and Staphylococcus aureus.Peptidoglycan from Staphylococcus aureus induces the overexpression of TRLs 1-8 mRNA in corneal fibroblasts, but its lipoteichoic acid and muramyl dipeptide only induced the overexpression of TLR5 or TRL9.Fate of mutation rate depends on agr locus expression during oxacillin-mediated heterogeneous-homogeneous selection in methicillin-resistant Staphylococcus aureus clinical strains.An agr-like two-component regulatory system in Lactobacillus plantarum is involved in production of a novel cyclic peptide and regulation of adherence.Changes in the agr locus affect enteritis caused by methicillin-resistant Staphylococcus aureus.Genetic requirements for Staphylococcus aureus nitric oxide resistance and virulence.

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Staphylococcus aureus agr and sarA functions are required for invasive infection but not inflammatory responses in the lung.

http://www.wikidata.org/entity/Q34116496

description

2002 nî lūn-bûn

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2002 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2002 թվականի հունվարին հրատարակված գիտական հոդված

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2002年の論文

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Staphylococcus aureus agr and ...... mmatory responses in the lung.

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Staphylococcus aureus agr and ...... mmatory responses in the lung.

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Staphylococcus aureus agr and ...... mmatory responses in the lung.

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IAI.70.1.127-133.2002

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Infection and Immunity

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Staphylococcus aureus agr and ...... mmatory responses in the lung.

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Alice Prince

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Ambrose Cheung

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Geoffrey Heyer

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Grace Soong

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Robert Adamo

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Shahryar Saba

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William Rush

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P2860

Cutting edge: recognition of Gram-positive bacterial cell wall components by the innate immune system occurs via Toll-like receptor 2

Heterologously expressed Staphylococcus aureus fibronectin-binding proteins are sufficient for invasion of host cells

Staphylococcus aureus genetic loci impacting growth and survival in multiple infection environments.

Peptidoglycan and lipoteichoic acid from Staphylococcus aureus induce tumor necrosis factor alpha, interleukin 6 (IL-6), and IL-10 production in both T cells and monocytes in a human whole blood model.

Cell density control of staphylococcal virulence mediated by an octapeptide pheromone.

Diminished virulence of a sar-/agr- mutant of Staphylococcus aureus in the rabbit model of endocarditis.

Collagen binding of Staphylococcus aureus is a virulence factor in experimental endocarditis.

Capsular polysaccharide types 5 and 8 of Staphylococcus aureus bind specifically to human epithelial (KB) cells, endothelial cells, and monocytes and induce release of cytokines.

Contribution of specific Pseudomonas aeruginosa virulence factors to pathogenesis of pneumonia in a neonatal mouse model of infection

Staphylococcal accessory regulator (sar) in conjunction with agr contributes to Staphylococcus aureus virulence in endophthalmitis

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127-133

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scholarly article

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10.1128/IAI.70.1.127-133.2002

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1

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70

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2002-01-01T00:00:00Z

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11748173

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127645

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