The choice of alternative 5' splice sites in influenza virus M1 mRNA is regulated by the viral polymerase complex.
about
The 3' splice site of influenza A segment 7 mRNA can exist in two conformations: a pseudoknot and a hairpinThe influenza A segment 7 mRNA 3' splice site pseudoknot/hairpin familyIdentification of a novel splice variant form of the influenza A virus M2 ion channel with an antigenically distinct ectodomainRNA-dependent activation of primer RNA production by influenza virus polymerase: different regions of the same protein subunit constitute the two required RNA-binding sites.The ion channel activity of the influenza virus M2 protein affects transport through the Golgi apparatusCellular RNA binding proteins NS1-BP and hnRNP K regulate influenza A virus RNA splicingInfluenza C virus NS1 protein upregulates the splicing of viral mRNAs.Recruitment of RED-SMU1 complex by Influenza A Virus RNA polymerase to control Viral mRNA splicingInfluenza virus ns1 protein induces apoptosis in cultured cells.Secondary structure of a conserved domain in an intron of influenza A M1 mRNAIndividual influenza A virus mRNAs show differential dependence on cellular NXF1/TAP for their nuclear export.Structural Analysis of the Roles of Influenza A Virus Membrane-Associated Proteins in Assembly and MorphologyNovel exploitation of a nuclear function by influenza virus: the cellular SF2/ASF splicing factor controls the amount of the essential viral M2 ion channel protein in infected cells.An A14U Substitution in the 3' Noncoding Region of the M Segment of Viral RNA Supports Replication of Influenza Virus with an NS1 Deletion by Modulating Alternative Splicing of M Segment mRNAsThe influenza A virus spliced messenger RNA M mRNA3 is not required for viral replication in tissue cultureAdaptive mutation in influenza A virus non-structural gene is linked to host switching and induces a novel protein by alternative splicingInfluenza matrix protein 2 alters CFTR expression and function through its ion channel activity.Mutations at alternative 5' splice sites of M1 mRNA negatively affect influenza A virus viability and growth rate.An NS-segment exonic splicing enhancer regulates influenza A virus replication in mammalian cells.Biogenesis, assembly, and export of viral messenger ribonucleoproteins in the influenza A virus infected cell.Influenza viruses and mRNA splicing: doing more with lessThree-dimensional model for the isolated recombinant influenza virus polymerase heterotrimer.A compensatory mutagenesis study of a conserved hairpin in the M gene segment of influenza A virus shows its role in virus replication.Nuclear export of influenza A virus mRNAs requires ongoing RNA polymerase II activity.Influenza virus polymerase confers independence of the cellular cap-binding factor eIF4E for viral mRNA translation.Establishment of a chimeric, replication-deficient influenza A virus vector by modulation of splicing efficiency.Influenza A Virus M2 Protein: Roles from Ingress to Egress.The award of the "Interbrew-Baillet Latour de la Santé-2002" prize to Robert M. Krug for outstanding contributions to influenza virus research.
P2860
Q21134545-7F260F98-F439-478F-B630-3AB74E0E147DQ24605070-FE114910-7991-4456-A376-2E388DEEF991Q27341132-A616643B-97BC-49C7-B2B9-1E6895DAAA22Q27867719-A0CDF930-1FE7-440B-A4AC-8B5808023D34Q28282474-3F5C4162-C506-4AD8-B44A-AE17FF4C4A28Q28534367-8557B440-DAEC-4357-AE27-5BC340F6FFE2Q33614671-B84FDB61-DBF3-4CC6-AA79-CF5765CDD369Q33748942-DA9DC6EF-7930-4E63-97EC-2B812521C5C1Q33853489-56467417-F137-4964-B91A-FEDD0A4AF7D3Q34070147-9AC1224D-D8D3-40C0-B64F-CE2C2E760531Q34641761-55FBAD8C-6C37-4F04-AD52-BF90B9FBE68DQ35666756-FAE41546-9592-45E6-AEAC-86BC5F223C50Q35910336-662DC461-5F32-4471-AA39-008504E3C7EDQ36086374-BE448E71-775E-464A-A830-7782D7D93A30Q36368449-F6B887C3-55BD-4AF1-9814-208EC8FCC026Q36779746-0AA07A56-E30E-45DD-AE8B-B61311277540Q36837735-CC845198-F8BC-401A-A336-B75FABEB9F8CQ36949707-10044252-DAB4-4BBA-9C4F-57F765FBA4C5Q37719285-9655A5CA-1B06-46E5-9A7A-772A82E031F3Q38117553-E0B820CB-4032-4035-9544-68DEC3E7BEA7Q38211561-4BCBFE5C-EEBC-4F3F-8553-F67BFC2FE652Q40129576-0A408709-5D91-485C-A803-BB243DBFC44AQ40140942-8037279B-DA3E-4F8A-A69D-1A8AEF40AFB1Q40203496-7CFD8FA1-9291-4FEF-88E9-C243788B656AQ42084631-DE61F7CD-B8D4-47F2-A733-A3C9FF218F10Q42707023-F61155A5-E24D-43EC-982E-7719AC0AEFEEQ45323658-872D9A18-C29B-4DFD-BB9F-4F67FA667988Q45731045-EDA41997-FFBE-4F0C-8D88-EE41BCCED3A8
P2860
The choice of alternative 5' splice sites in influenza virus M1 mRNA is regulated by the viral polymerase complex.
description
1995 nî lūn-bûn
@nan
1995 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
name
The choice of alternative 5' s ...... the viral polymerase complex.
@ast
The choice of alternative 5' s ...... the viral polymerase complex.
@en
The choice of alternative 5' s ...... the viral polymerase complex.
@nl
type
label
The choice of alternative 5' s ...... the viral polymerase complex.
@ast
The choice of alternative 5' s ...... the viral polymerase complex.
@en
The choice of alternative 5' s ...... the viral polymerase complex.
@nl
prefLabel
The choice of alternative 5' s ...... the viral polymerase complex.
@ast
The choice of alternative 5' s ...... the viral polymerase complex.
@en
The choice of alternative 5' s ...... the viral polymerase complex.
@nl
P2093
P2860
P356
P1476
The choice of alternative 5' s ...... the viral polymerase complex.
@en
P2093
P2860
P304
P356
10.1073/PNAS.92.14.6324
P407
P577
1995-07-01T00:00:00Z