Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations - Wikidata - wikimedia - TriplyDB

Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations

Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations

http://www.wikidata.org/entity/Q34121201

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Mouse models of acute exacerbations of allergic asthma GM-CSF produced by the airway epithelium is required for sensitization to cockroach allergen Understanding Interleukin 33 and Its Roles in Eosinophil Development Pulmonary ORMDL3 is critical for induction of Alternaria-induced allergic airways disease.Early local immune defences in the respiratory tract IL-33 and thymic stromal lymphopoietin mediate immune pathology in response to chronic airborne allergen exposure Oxidation of the alarmin IL-33 regulates ST2-dependent inflammation.Mold allergens in respiratory allergy: from structure to therapy.Interleukin-33 in Tissue Homeostasis, Injury, and Inflammation CARMA3 Is Critical for the Initiation of Allergic Airway Inflammation.Pediatric severe asthma with fungal sensitization is mediated by steroid-resistant IL-33 Porphyromonas gingivalis Gingipain-Dependently Enhances IL-33 Production in Human Gingival Epithelial Cells.The novel PAR2 ligand C391 blocks multiple PAR2 signalling pathways in vitro and in vivo.Pulmonary receptor for advanced glycation end-products promotes asthma pathogenesis through IL-33 and accumulation of group 2 innate lymphoid cells Aspirin-Exacerbated Respiratory Disease Involves a Cysteinyl Leukotriene-Driven IL-33-Mediated Mast Cell Activation Pathway Ovarian cancer treatment with a tumor-targeting and gene expression-controllable lipoplex The histone deacetylase inhibitor trichostatin A suppresses murine innate allergic inflammation by blocking group 2 innate lymphoid cell (ILC2) activation.Alternative splicing of interleukin-33 and type 2 inflammation in asthma.Allergens stimulate store-operated calcium entry and cytokine production in airway epithelial cells.IL-33 mediates reactive eosinophilopoiesis in response to airborne allergen exposure Fatal asthma; is it still an epidemic?Is allergic sensitization relevant in severe asthma? Which allergens may be culprit?Cockroach protease allergen induces allergic airway inflammation via epithelial cell activation.IL-1 and IL-36 are dominant cytokines in generalized pustular psoriasis.ILC2s and fungal allergy.Update in Asthma 2014.Group 2 innate lymphoid cells in disease.Early life factors that affect allergy development.Airway epithelial anion secretion and barrier function following exposure to fungal aeroallergens: role of oxidative stress.Oxidative Stress: Promoter of Allergic Sensitization to Protease Allergens?Airway epithelial dual oxidase 1 mediates allergen-induced IL-33 secretion and activation of type 2 immune responses.Innate Immune Responses to Fungal Allergens.Human Helminths and Allergic Disease: The Hygiene Hypothesis and Beyond.Mouse models of severe asthma: Understanding the mechanisms of steroid resistance, tissue remodelling and disease exacerbation.Allergic airway inflammation: key players beyond the Th2 cell pathway.Development of allergic immunity in early life.IL-33: biological properties, functions, and roles in airway disease.Role of Type 2 Innate Lymphoid Cells in Allergic Diseases.The Innate Cytokines IL-25, IL-33, and TSLP Cooperate in the Induction of Type 2 Innate Lymphoid Cell Expansion and Mucous Metaplasia in Rhinovirus-Infected Immature Mice.Different clinical effect of several types of airborne allergens on the severity of bronchial hyperreactivity.

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Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations

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2014 nî lūn-bûn

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2014 թուականի Մարտին հրատարակուած գիտական յօդուած

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2014 թվականի մարտին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations

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Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations

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Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations

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Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations

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Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations

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Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations

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Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations

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Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations

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Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations

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Alternaria-derived serine protease activity drives IL-33-mediated asthma exacerbations

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Gaynor A Campbell

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Robert J Snelgrove

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Samia Akthar

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Simone A Walker

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Teresa Peiró

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P2860

IL-33 amplifies the polarization of alternatively activated macrophages that contribute to airway inflammation

The IL-1-like cytokine IL-33 is inactivated after maturation by caspase-1

Mold sensitization is common amongst patients with severe asthma requiring multiple hospital admissions

Innate production of T(H)2 cytokines by adipose tissue-associated c-Kit(+)Sca-1(+) lymphoid cells

Alternaria alternata serine proteases induce lung inflammation and airway epithelial cell activation via PAR2.

Predictive markers of asthma exacerbation during stepwise dose reduction of inhaled corticosteroids.

The IL-1-like cytokine IL-33 is constitutively expressed in the nucleus of endothelial cells and epithelial cells in vivo: a novel 'alarmin'?

Increased expression of IL-33 in severe asthma: evidence of expression by airway smooth muscle cells.

IL-9-deficient mice establish fundamental roles for IL-9 in pulmonary mastocytosis and goblet cell hyperplasia but not T cell development.

Sensitisation to airborne moulds and severity of asthma: cross sectional study from European Community respiratory health survey

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