Paxillin regulates androgen- and epidermal growth factor-induced MAPK signaling and cell proliferation in prostate cancer cells. - Wikidata - wikimedia - TriplyDB

Paxillin regulates androgen- and epidermal growth factor-induced MAPK signaling and cell proliferation in prostate cancer cells.

Paxillin regulates androgen- and epidermal growth factor-induced MAPK signaling and cell proliferation in prostate cancer cells.

http://www.wikidata.org/entity/Q34121332

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Targeting membrane androgen receptors in tumors Androgen actions on endothelium functions and cardiovascular diseases Overlapping nongenomic and genomic actions of thyroid hormone and steroids Minireview: Recent advances in extranuclear steroid receptor actions Spatially defined EGF receptor activation reveals an F-actin-dependent phospho-Erk signaling complex.Overexpression of paxillin induced by miR-137 suppression promotes tumor progression and metastasis in colorectal cancer Translating extranuclear steroid receptor signaling to clinical medicine.The regulation of spermatogenesis by androgens.Significance of talin in cancer progression and metastasis.The focal complex of epithelial cells provides a signalling platform for interleukin-8 induction in response to bacterial pathogens Understanding extranuclear (nongenomic) androgen signaling: what a frog oocyte can tell us about human biology Stimulus-specific activation and actin dependency of distinct, spatially separated ERK1/2 fractions in A7r5 smooth muscle cells.Abnormal expression of paxillin correlates with tumor progression and poor survival in patients with gastric cancer.Minireview: Extranuclear steroid receptors: roles in modulation of cell functions.GPCR/EGFR cross talk is conserved in gonadal and adrenal steroidogenesis but is uniquely regulated by matrix metalloproteinases 2 and 9 in the ovary.Diagnosis and treatment of sexual dysfunctions in late-onset hypogonadism Androgen receptor non-nuclear regulation of prostate cancer cell invasion mediated by Src and matriptase.Up-regulation of Orai1 expression and store operated Ca(2+) entry following activation of membrane androgen receptors in MCF-7 breast tumor cells.Paxillin mediates extranuclear and intranuclear signaling in prostate cancer proliferation.Protein kinase Cα and Src kinase support human prostate-distributed dihydrotestosterone-metabolizing UDP-glucuronosyltransferase 2B15 activity.Inhibition of SGK1 enhances mAR-induced apoptosis in MCF-7 breast cancer cells.Stable Isotope Labeling with Amino Acids (SILAC)-Based Proteomics of Primary Human Kidney Cells Reveals a Novel Link between Male Sex Hormones and Impaired Energy Metabolism in Diabetic Kidney Disease.Discovery of a Novel Inhibitor of the Protein Tyrosine Phosphatase Shp2.Nuclear transport of paxillin depends on focal adhesion dynamics and FAT domains Paxillin and steroid signaling: from frog to human.Androgens regulate ovarian follicular development by increasing follicle stimulating hormone receptor and microRNA-125b expression.β-arrestin 2-dependent activation of ERK1/2 is required for ADP-induced paxillin phosphorylation at Ser(83) and microglia chemotaxis.The actin cytoskeleton in rapid steroid hormone actions.The molecular mechanisms underlying the ERα-36-mediated signaling in breast cancer Nuclear receptors outside the nucleus: extranuclear signalling by steroid receptors.Marine natural products-inspired phenylmethylene hydantoins with potent in vitro and in vivo antitumor activities via suppression of Brk and FAK signaling.Phosphorylation of paxillin confers cisplatin resistance in non-small cell lung cancer via activating ERK-mediated Bcl-2 expression.An ion-current-based, comprehensive and reproducible proteomic strategy for comparative characterization of the cellular responses to novel anti-cancer agents in a prostate cell model.The adaptor protein and Arf GTPase-activating protein Cat-1/Git-1 is required for cellular transformation.ARFGAP3, an androgen target gene, promotes prostate cancer cell proliferation and migration.Testosterone signaling and the regulation of spermatogenesis.Abi3bp is a multifunctional autocrine/paracrine factor that regulates mesenchymal stem cell biology.A novel androgen signalling pathway uses dihydrotestosterone, but not testosterone, to activate the EGF receptor signalling cascade in prostate stromal cells.Transfer printing of transfected cell microarrays from poly(ethylene glycol)-oleyl surfaces onto biological hydrogels.Paxillin actions in the nucleus.

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P2860

Paxillin regulates androgen- and epidermal growth factor-induced MAPK signaling and cell proliferation in prostate cancer cells.

http://www.wikidata.org/entity/Q34121332

description

2010 nî lūn-bûn

@nan

2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Paxillin regulates androgen- a ...... tion in prostate cancer cells.

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Paxillin regulates androgen- a ...... tion in prostate cancer cells.

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Paxillin regulates androgen- a ...... tion in prostate cancer cells.

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Journal of Biological Chemistry

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Paxillin regulates androgen- a ...... tion in prostate cancer cells.

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Aritro Sen

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Donald B Defranco

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Ganesh V Raj

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Katherine O'Malley

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Stephen R Hammes

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Zhou Wang

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P2860

Newest findings on the oldest oncogene; how activated src does it

Brk activates rac1 and promotes cell migration and invasion by phosphorylating paxillin

Testosterone activates mitogen-activated protein kinase and the cAMP response element binding protein transcription factor in Sertoli cells

Steroid-induced androgen receptor-oestradiol receptor beta-Src complex triggers prostate cancer cell proliferation

Paxillin comes of age

Hormonal control of androgen receptor function through SIRT1

Erlotinib effectively inhibits JAK2V617F activity and polycythemia vera cell growth

The spatiotemporal pattern of Src activation at lipid rafts revealed by diffusion-corrected FRET imaging

Beta1 integrins activate a MAPK signalling pathway in neural stem cells that contributes to their maintenance

Changes in androgen receptor nongenotropic signaling correlate with transition of LNCaP cells to androgen independence

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