NikR mediates nickel-responsive transcriptional induction of urease expression in Helicobacter pylori.
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Interplay of metal ions and ureasePathogenesis of Helicobacter pylori infectionCoping with low pH: molecular strategies in neutralophilic bacteriaStructural and mechanistic insights into Helicobacter pylori NikR activationHolo-Ni(II) Hp NikR Is an Asymmetric Tetramer Containing Two Different Nickel-Binding SitesHolo-Ni2+ Helicobacter pylori NikR contains four square-planar nickel-binding sites at physiological pHNi(II) coordination to mixed sites modulates DNA binding of HpNikR via a long-range effect.Acid-induced activation of the urease promoters is mediated directly by the ArsRS two-component system of Helicobacter pylori.Helicobacter pylori HP1512 is a nickel-responsive NikR-regulated outer membrane proteinNikR mediates nickel-responsive transcriptional repression of the Helicobacter pylori outer membrane proteins FecA3 (HP1400) and FrpB4 (HP1512).Dissecting the role of DNA sequence in Helicobacter pylori NikR/DNA recognition.The N-terminal arm of the Helicobacter pylori Ni2+-dependent transcription factor NikR is required for specific DNA binding.Characterization of the ArsRS regulon of Helicobacter pylori, involved in acid adaptationBuilt shallow to maintain homeostasis and persistent infection: insight into the transcriptional regulatory network of the gastric human pathogen Helicobacter pylori.Analysis of protein expression regulated by the Helicobacter pylori ArsRS two-component signal transduction system.Helicobacter pylori gene silencing in vivo demonstrates urease is essential for chronic infection.In vitro analysis of protein-operator interactions of the NikR and fur metal-responsive regulators of coregulated genes in Helicobacter pyloriA cis-encoded antisense small RNA regulated by the HP0165-HP0166 two-component system controls expression of ureB in Helicobacter pylori.Helicobacter pylori 5'ureB-sRNA, a cis-encoded antisense small RNA, negatively regulates ureAB expression by transcription terminationIdentification by RNA profiling and mutational analysis of the novel copper resistance determinants CrdA (HP1326), CrdB (HP1327), and CzcB (HP1328) in Helicobacter pylori.Nickel represses the synthesis of the nickel permease NixA of Helicobacter pyloriThe novel Helicobacter pylori CznABC metal efflux pump is required for cadmium, zinc, and nickel resistance, urease modulation, and gastric colonizationMua (HP0868) is a nickel-binding protein that modulates urease activity in Helicobacter pylori.Hierarchical regulation of the NikR-mediated nickel response in Helicobacter pyloriCharacterization in Helicobacter pylori of a Nickel Transporter Essential for Colonization That Was Acquired during Evolution by Gastric Helicobacter Species.Helicobacter pylori stores nickel to aid its host colonization.Nickel-binding and accessory proteins facilitating Ni-enzyme maturation in Helicobacter pylori.Metal-responsive gene regulation and metal transport in Helicobacter species.The role of ExbD in periplasmic pH homeostasis in Helicobacter pyloriHydrogen Metabolism in Helicobacter pylori Plays a Role in Gastric Carcinogenesis through Facilitating CagA Translocation.Regulatory circuits in Helicobacter pylori : network motifs and regulators involved in metal-dependent responses.Nickel-responsive transcriptional regulators.Survival of Helicobacter pylori in gastric acidic territory.The nickel-responsive regulator NikR controls activation and repression of gene transcription in Helicobacter pylori.Surface plasmon resonance and isothermal titration calorimetry to monitor the Ni(II)-dependent binding of Helicobacter pylori NikR to DNA.Acid-responsive gene induction of ammonia-producing enzymes in Helicobacter pylori is mediated via a metal-responsive repressor cascade.Crosstalk between the HpArsRS two-component system and HpNikR is necessary for maximal activation of urease transcription.On the interaction of Helicobacter pylori NikR, a Ni(II)-responsive transcription factor, with the urease operator: in solution and in silico studies.Nickel-responsive regulation of two novel Helicobacter pylori NikR-targeted genes.The Helicobacter pylori CrdRS two-component regulation system (HP1364/HP1365) is required for copper-mediated induction of the copper resistance determinant CrdA.
P2860
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P2860
NikR mediates nickel-responsive transcriptional induction of urease expression in Helicobacter pylori.
description
2002 nî lūn-bûn
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2002 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
NikR mediates nickel-responsiv ...... ession in Helicobacter pylori.
@ast
NikR mediates nickel-responsiv ...... ession in Helicobacter pylori.
@en
NikR mediates nickel-responsiv ...... ession in Helicobacter pylori.
@nl
type
label
NikR mediates nickel-responsiv ...... ession in Helicobacter pylori.
@ast
NikR mediates nickel-responsiv ...... ession in Helicobacter pylori.
@en
NikR mediates nickel-responsiv ...... ession in Helicobacter pylori.
@nl
prefLabel
NikR mediates nickel-responsiv ...... ession in Helicobacter pylori.
@ast
NikR mediates nickel-responsiv ...... ession in Helicobacter pylori.
@en
NikR mediates nickel-responsiv ...... ession in Helicobacter pylori.
@nl
P2093
P2860
P1476
NikR mediates nickel-responsiv ...... ession in Helicobacter pylori.
@en
P2093
Arnoud H M van Vliet
Beverly J Davies
Charles W Penn
Jeroen Stoof
Johannes G Kusters
Manfred Kist
Sophie W Poppelaars
P2860
P304
P356
10.1128/IAI.70.6.2846-2852.2002
P407
P577
2002-06-01T00:00:00Z