Defective ribosomal products are the major source of antigenic peptides endogenously generated from influenza A virus neuraminidase.
about
DRiPs solidify: progress in understanding endogenous MHC class I antigen processingMHC class I antigen presentation of DRiP-derived peptides from a model antigen is not dependent on the AAA ATPase p97Biogenesis of influenza a virus hemagglutinin cross-protective stem epitopesDifferent Polyubiquitinated Bodies in Human Dendritic Cells: IL-4 Causes PaCS During Differentiation while LPS or IFNα Induces DALIS During MaturationSimilar intracellular peptide profile of TAP1/β2 microglobulin double-knockout mice and C57BL/6 wild-type mice as revealed by peptidomic analysis.Signal peptides and trans-membrane regions are broadly immunogenic and have high CD8+ T cell epitope densities: Implications for vaccine development.Hydrophobicity as a driver of MHC class I antigen processing.Influenza A viral replication is blocked by inhibition of the inositol-requiring enzyme 1 (IRE1) stress pathway.Ubiquitous Autofragmentation of Fluorescent Proteins Creates Abundant Defective Ribosomal Products (DRiPs) for Immunosurveillance.Enhanced Recognition of HIV-1 Cryptic Epitopes Restricted by HLA Class I Alleles Associated With a Favorable Clinical Outcome.RNA polymerase II inhibitors dissociate antigenic peptide generation from normal viral protein synthesis: a role for nuclear translation in defective ribosomal product synthesis?Distinct pathways generate peptides from defective ribosomal products for CD8+ T cell immunosurveillance.Defining Viral Defective Ribosomal Products: Standard and Alternative Translation Initiation Events Generate a Common Peptide from Influenza A Virus M2 and M1 mRNAsInfluenza A virus hemagglutinin trimerization completes monomer folding and antigenicity.The role of the proteasome in the generation of MHC class I ligands and immune responses.Translating DRiPs: progress in understanding viral and cellular sources of MHC class I peptide ligands.Running the gauntlet: from peptide generation to antigen presentation by MHC class I.Influenza vaccines: T-cell responses deserve more attention.Alternative Start Sites Downstream of Non-Sense Mutations Drive Antigen Presentation and Tolerance Induction to C-Terminal Epitopes.Characterization of T-cell responses to cryptic epitopes in recipients of a noncodon-optimized HIV-1 vaccineExtended Culture of Bone Marrow with Granulocyte Macrophage-Colony Stimulating Factor Generates Immunosuppressive Cells.
P2860
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P2860
Defective ribosomal products are the major source of antigenic peptides endogenously generated from influenza A virus neuraminidase.
description
2009 nî lūn-bûn
@nan
2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Defective ribosomal products a ...... fluenza A virus neuraminidase.
@ast
Defective ribosomal products a ...... fluenza A virus neuraminidase.
@en
Defective ribosomal products a ...... fluenza A virus neuraminidase.
@nl
type
label
Defective ribosomal products a ...... fluenza A virus neuraminidase.
@ast
Defective ribosomal products a ...... fluenza A virus neuraminidase.
@en
Defective ribosomal products a ...... fluenza A virus neuraminidase.
@nl
prefLabel
Defective ribosomal products a ...... fluenza A virus neuraminidase.
@ast
Defective ribosomal products a ...... fluenza A virus neuraminidase.
@en
Defective ribosomal products a ...... fluenza A virus neuraminidase.
@nl
P2093
P2860
P356
P1476
Defective ribosomal products a ...... fluenza A virus neuraminidase.
@en
P2093
Brian P Dolan
Jack R Bennink
Kazuyo Takeda
P2860
P304
P356
10.4049/JIMMUNOL.0901907
P407
P577
2009-12-28T00:00:00Z