Chronic hypoxia inhibits sex steroid hormone-mediated attenuation of ovine uterine arterial myogenic tone in pregnancy.
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Gestational hypoxia up-regulates protein kinase C and inhibits calcium-activated potassium channels in ovine uterine arteries.Epigenetic upregulation of large-conductance Ca2+-activated K+ channel expression in uterine vascular adaptation to pregnancyChronic hypoxia during gestation enhances uterine arterial myogenic tone via heightened oxidative stress.Gestational hypoxia increases reactive oxygen species and inhibits steroid hormone-mediated upregulation of Ca(2+)-activated K(+) channel function in uterine arteries.Pregnancy upregulates large-conductance Ca(2+)-activated K(+) channel activity and attenuates myogenic tone in uterine arteries.Chronic hypoxia differentially up-regulates protein kinase C-mediated ovine uterine arterial contraction via actin polymerization signaling in pregnancy.Hypoxia Represses ER-α Expression and Inhibits Estrogen-Induced Regulation of Ca2+-Activated K+ Channel Activity and Myogenic Tone in Ovine Uterine Arteries: Causal Role of DNA MethylationChronic hypoxia during gestation causes epigenetic repression of the estrogen receptor-α gene in ovine uterine arteries via heightened promoter methylation.Adaptive increases in expression and vasodilator activity of estrogen receptor subtypes in a blood vessel-specific pattern during pregnancy.Chronic hypoxia suppresses pregnancy-induced upregulation of large-conductance Ca2+-activated K+ channel activity in uterine arteriesPregnancy-associated adaptations in [Ca2+]i-dependent and Ca2+ sensitization mechanisms of venous contraction: implications in pregnancy-related venous disorders.Chronic hypoxia inhibits pregnancy-induced upregulation of SKCa channel expression and function in uterine arteries.Gestational hypoxia induces preeclampsia-like symptoms via heightened endothelin-1 signaling in pregnant rats.Pregnancy Reprograms Large-Conductance Ca2+-Activated K+ Channel in Uterine Arteries: Roles of Ten-Eleven Translocation Methylcytosine Dioxygenase 1-Mediated Active Demethylation.Direct effect of chronic hypoxia in suppressing large conductance Ca(2+)-activated K(+) channel activity in ovine uterine arteries via increasing oxidative stress.Chronic hypoxia upregulates DNA methyltransferase and represses large conductance Ca2+-activated K+ channel function in ovine uterine arteries.MicroRNA-210 Targets Ten-Eleven Translocation Methylcytosine Dioxygenase 1 and Suppresses Pregnancy-Mediated Adaptation of Large Conductance Ca2+-Activated K+ Channel Expression and Function in Ovine Uterine Arteries.
P2860
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P2860
Chronic hypoxia inhibits sex steroid hormone-mediated attenuation of ovine uterine arterial myogenic tone in pregnancy.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Chronic hypoxia inhibits sex s ...... al myogenic tone in pregnancy.
@ast
Chronic hypoxia inhibits sex s ...... al myogenic tone in pregnancy.
@en
Chronic hypoxia inhibits sex s ...... al myogenic tone in pregnancy.
@nl
type
label
Chronic hypoxia inhibits sex s ...... al myogenic tone in pregnancy.
@ast
Chronic hypoxia inhibits sex s ...... al myogenic tone in pregnancy.
@en
Chronic hypoxia inhibits sex s ...... al myogenic tone in pregnancy.
@nl
prefLabel
Chronic hypoxia inhibits sex s ...... al myogenic tone in pregnancy.
@ast
Chronic hypoxia inhibits sex s ...... al myogenic tone in pregnancy.
@en
Chronic hypoxia inhibits sex s ...... al myogenic tone in pregnancy.
@nl
P2093
P2860
P1433
P1476
Chronic hypoxia inhibits sex s ...... ial myogenic tone in pregnancy
@en
P2093
Katherine Chang
Lawrence D Longo
Shumei Yang
Xiaohui Huang
P2860
P304
P356
10.1161/HYPERTENSIONAHA.110.155812
P407
P577
2010-07-26T00:00:00Z