The nonphagocytic NADPH oxidase Duox1 mediates a positive feedback loop during T cell receptor signaling. - Wikidata - wikimedia - TriplyDB

The nonphagocytic NADPH oxidase Duox1 mediates a positive feedback loop during T cell receptor signaling.

The nonphagocytic NADPH oxidase Duox1 mediates a positive feedback loop during T cell receptor signaling.

http://www.wikidata.org/entity/Q34135661

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Reactive Oxygen Species Regulate T Cell Immune Response in the Tumor Microenvironment Clinical significance of T cell metabolic reprogramming in cancer T cells and reactive oxygen species The role of selenium in inflammation and immunity: from molecular mechanisms to therapeutic opportunities Multiple sclerosis: molecular mechanisms and therapeutic opportunities Mechanisms of activation of the transcription factor Nrf2 by redox stressors, nutrient cues, and energy status and the pathways through which it attenuates degenerative disease Dual oxidase regulates neutrophil recruitment in allergic airways Peroxiredoxin 6 (Prdx6) supports NADPH oxidase1 (Nox1)-based superoxide generation and cell migration.VHL-dependent alterations in the secretome of renal cell carcinoma: Association with immune cell response?Hydrogen Peroxide Triggers a Dual Signaling Axis To Selectively Suppress Activated Human T Lymphocyte Migration.Reactive oxygen species are required for driving efficient and sustained aerobic glycolysis during CD4+ T cell activation.NLRP3 inflammasome activation and interleukin-1β release in macrophages require calcium but are independent of calcium-activated NADPH oxidases Chaperone-mediated autophagy regulates T cell responses through targeted degradation of negative regulators of T cell activation.TCR-triggered extracellular superoxide production is not required for T-cell activation.Superoxide production by macrophages and T cells is critical for the induction of autoreactivity and type 1 diabetes Programmed death-1 controls T cell survival by regulating oxidative metabolism.Glucose deprivation activates a metabolic and signaling amplification loop leading to cell death Prolonged production of reactive oxygen species in response to B cell receptor stimulation promotes B cell activation and proliferation.Decreased STAT5 phosphorylation and GATA-3 expression in NOX2-deficient T cells: role in T helper development DUOX1 mediates persistent epithelial EGFR activation, mucous cell metaplasia, and airway remodeling during allergic asthma.Oxidative stress induced by HIV-1 reverse transcriptase modulates the enzyme's performance in gene immunization.Key roles of autophagy in regulating T-cell function Nox enzymes in allergic airway inflammation.Regulation of protein tyrosine phosphatases by reversible oxidation.Reactive oxygen species and the neuronal fate.Protein tyrosine phosphatase structure-function relationships in regulation and pathogenesis.Mitochondria as oxidative signaling organelles in T-cell activation: physiological role and pathological implications.Ncf1 polymorphism reveals oxidative regulation of autoimmune chronic inflammation.Single-cell resolution of intracellular T cell Ca2+ dynamics in response to frequency-based H2O2 stimulation.Redox balance of mouse medullary CD4 single-positive thymocytes.IFNβ/TNFα synergism induces a non-canonical STAT2/IRF9-dependent pathway triggering a novel DUOX2 NADPH oxidase-mediated airway antiviral response.The role of metabolic reprogramming in T cell fate and function.Paradoxical roles of dual oxidases in cancer biology.Metabolic orchestration of T lineage differentiation and function.An automated programmable platform enabling multiplex dynamic stimuli delivery and cellular response monitoring for high-throughput suspension single-cell signaling studies.Hypoxic culture conditions enhance the generation of regulatory T cells.Regulation of redox balance in cancer and T cells.DUAL OXIDASE: A NOVEL THERAPEUTIC TARGET IN ALLERGIC DISEASE.Redox-sensitive signaling in inflammatory T cells and in autoimmune disease.

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The nonphagocytic NADPH oxidase Duox1 mediates a positive feedback loop during T cell receptor signaling.

http://www.wikidata.org/entity/Q34135661

description

2010 nî lūn-bûn

@nan

2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի օգոստոսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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The nonphagocytic NADPH oxidas ...... ing T cell receptor signaling.

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Science Signaling

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Françoise Miot

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Haiyan Chen

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Jaeyul Kwon

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Kristen E Shatynski

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Mark S Williams

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Stanislas Morand

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Xavier de Deken

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P2860

Tyrosine 319 in the interdomain B of ZAP-70 is a binding site for the Src homology 2 domain of Lck

Cloning of two human thyroid cDNAs encoding new members of the NADPH oxidase family

Involvement of Rac1 in activation of multicomponent Nox1- and Nox3-based NADPH oxidases

Identification of a phospholipase C-gamma1 (PLC-gamma1) SH3 domain-binding site in SLP-76 required for T-cell receptor-mediated activation of PLC-gamma1 and NFAT

HVCN1 modulates BCR signal strength via regulation of BCR-dependent generation of reactive oxygen species

Structural basis for relief of autoinhibition of the Dbl homology domain of proto-oncogene Vav by tyrosine phosphorylation

Structural basis for the inhibition of tyrosine kinase activity of ZAP-70

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

The role of membrane-associated adaptors in T cell receptor signalling

Docking protein Gab2 is phosphorylated by ZAP-70 and negatively regulates T cell receptor signaling by recruitment of inhibitory molecules

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