Enhancement of reactive oxygen species production and chlamydial infection by the mitochondrial Nod-like family member NLRX1. - Wikidata - wikimedia - TriplyDB

Enhancement of reactive oxygen species production and chlamydial infection by the mitochondrial Nod-like family member NLRX1.

Enhancement of reactive oxygen species production and chlamydial infection by the mitochondrial Nod-like family member NLRX1.

http://www.wikidata.org/entity/Q34144581

about

NLRX1/NOD5 deficiency does not affect MAVS signalling Periodontal Disease-Induced Atherosclerosis and Oxidative Stress The Role of the Immune Response in Chlamydia trachomatis Infection of the Male Genital Tract: A Double-Edged Sword NOD-Like Receptors: Master Regulators of Inflammation and Cancer The intracellular bacteria Chlamydia hijack peroxisomes and utilize their enzymatic capacity to produce bacteria-specific phospholipids Danger signals, inflammasomes, and the intricate intracellular lives of chlamydiae Holding the inflammatory system in check: NLRs keep it cool NLRs, inflammasomes, and viral infection Modeling-Enabled Characterization of Novel NLRX1 Ligands NLRX1 protein attenuates inflammatory responses to infection by interfering with the RIG-I-MAVS and TRAF6-NF-κB signaling pathways Activation of an NLRP3 inflammasome restricts Mycobacterium kansasii infection Laser-mediated rupture of chlamydial inclusions triggers pathogen egress and host cell necrosis.The potential role of infectious agents and pelvic inflammatory disease in ovarian carcinogenesis Alarmins, inflammasomes and immunity.Opportunistic Pathogen Porphyromonas gingivalis Modulates Danger Signal ATP-Mediated Antibacterial NOX2 Pathways in Primary Epithelial Cells.Rac1 regulates the NLRP3 inflammasome which mediates IL-1beta production in Chlamydophila pneumoniae infected human mononuclear cells.Imbalanced oxidative stress causes chlamydial persistence during non-productive human herpes virus co-infection.Modeling the Regulatory Mechanisms by Which NLRX1 Modulates Innate Immune Responses to Helicobacter pylori Infection P2X4 assembles with P2X7 and pannexin-1 in gingival epithelial cells and modulates ATP-induced reactive oxygen species production and inflammasome activation Nlrx1 regulates neuronal cell death.Female reproductive hormones and biomarkers of oxidative stress in genital Chlamydia infection in tubal factor infertility.Emerging significance of NLRs in inflammatory bowel disease.Detection of a microbial metabolite by STING regulates inflammasome activation in response to Chlamydia trachomatis infection.Porphyromonas gingivalis-nucleoside-diphosphate-kinase inhibits ATP-induced reactive-oxygen-species via P2X7 receptor/NADPH-oxidase signalling and contributes to persistence.NLRX1 Sequesters STING to Negatively Regulate the Interferon Response, Thereby Facilitating the Replication of HIV-1 and DNA Viruses.NLRX1 has a tail to tell.The Innate Immune Receptor NLRX1 Functions as a Tumor Suppressor by Reducing Colon Tumorigenesis and Key Tumor-Promoting Signals.Beyond the inflammasome: regulatory NOD-like receptor modulation of the host immune response following virus exposure.Salvianolic Acid B Restored Impaired Barrier Function via Downregulation of MLCK by microRNA-1 in Rat Colitis Model Chlamydia cell biology and pathogenesis The expanding role of NLRs in antiviral immunity.Functions of NOD-Like Receptors in Human Diseases.NLRX1 accelerates cisplatin-induced ototoxity in HEI-OC1 cells via promoting generation of ROS and activation of JNK signaling pathway.Chlamydia trachomatis and HPV co-infections in HIV negative men from a multi-ethnic area of Northern Italy at high prevalence of cervical malignancies.Subversion of Cell-Autonomous Host Defense by Chlamydia Infection.The role of reactive-oxygen-species in microbial persistence and inflammation.Mitochondria in innate immunity.Non-Inflammasome Forming NLRs in Inflammation and Tumorigenesis.Mitochondrial Regulation of Inflammasome Activation in Chronic Obstructive Pulmonary Disease.Role of Innate Genes in HIV Replication.

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Enhancement of reactive oxygen species production and chlamydial infection by the mitochondrial Nod-like family member NLRX1.

http://www.wikidata.org/entity/Q34144581

description

2010 nî lūn-bûn

@nan

2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Enhancement of reactive oxygen ...... Nod-like family member NLRX1.

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Enhancement of reactive oxygen ...... Nod-like family member NLRX1.

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Enhancement of reactive oxygen ...... Nod-like family member NLRX1.

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type

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Enhancement of reactive oxygen ...... Nod-like family member NLRX1.

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Enhancement of reactive oxygen ...... Nod-like family member NLRX1.

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Enhancement of reactive oxygen ...... Nod-like family member NLRX1.

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Enhancement of reactive oxygen ...... Nod-like family member NLRX1.

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Enhancement of reactive oxygen ...... Nod-like family member NLRX1.

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Enhancement of reactive oxygen ...... Nod-like family member NLRX1.

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JBC.M110.137885

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Journal of Biological Chemistry

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Enhancement of reactive oxygen ...... l Nod-like family member NLRX1

@en

P2093

Ali A Abdul-Sater

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Bhavni Singh

http://www.w3.org/2001/XMLSchema#string

Fraser Soares

http://www.w3.org/2001/XMLSchema#string

Ivan Tattoli

http://www.w3.org/2001/XMLSchema#string

Matthew A Pettengill

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Najwane Saïd-Sadier

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Simone Lipinski

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Stephen E Girardin

http://www.w3.org/2001/XMLSchema#string

Verissa M Lam

http://www.w3.org/2001/XMLSchema#string

P2860

Nod1-mediated endothelial cell activation by Chlamydophila pneumoniae

An N-terminal addressing sequence targets NLRX1 to the mitochondrial matrix

NLRX1 is a mitochondrial NOD-like receptor that amplifies NF-kappaB and JNK pathways by inducing reactive oxygen species production

Redox mechanisms in hepatic chronic wound healing and fibrogenesis

Homologs of gp91phox: cloning and tissue expression of Nox3, Nox4, and Nox5

Thioredoxin-interacting protein links oxidative stress to inflammasome activation

Recruitment of BAD by the Chlamydia trachomatis vacuole correlates with host-cell survival.

Stimulator of IFN gene is critical for induction of IFN-beta during Chlamydia muridarum infection

Closing in on Chlamydia and its intracellular bag of tricks.

Intracellular survival by Chlamydia.

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41637-41645

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10.1074/JBC.M110.137885

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53

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285

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Philip Rosenstiel

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2010-10-19T00:00:00Z

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20959452

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3009891

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