Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival. - Wikidata - wikimedia - TriplyDB

Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.

Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.

http://www.wikidata.org/entity/Q34146480

about

Beyond generalized hair cells: molecular cues for hair cell types The genetics of hair cell development and regeneration Segregating neural and mechanosensory fates in the developing ear: patterning, signaling, and transcriptional control.Neurog1 can partially substitute for Atoh1 function in hair cell differentiation and maintenance during organ of Corti development The quest for restoring hearing: Understanding ear development more completely.Inner ear development: building a spiral ganglion and an organ of Corti out of unspecified ectoderm Evolution and development of the tetrapod auditory system: an organ of Corti-centric perspective.Evolving gene regulatory networks into cellular networks guiding adaptive behavior: an outline how single cells could have evolved into a centralized neurosensory system.The Role of Atonal Factors in Mechanosensory Cell Specification and Function The myc road to hearing restoration.Correct timing of proliferation and differentiation is necessary for normal inner ear development and auditory hair cell viability.Understanding the evolution and development of neurosensory transcription factors of the ear to enhance therapeutic translation.Opportunities and limits of the one gene approach: the ability of Atoh1 to differentiate and maintain hair cells depends on the molecular context The role of her4 in inner ear development and its relationship with proneural genes and Notch signalling.Pressure in the cochlea during infrared irradiation.Atoh1 directs hair cell differentiation and survival in the late embryonic mouse inner ear.The role of Atonal transcription factors in the development of mechanosensitive cells.Can the 'neuron theory' be complemented by a universal mechanism for generic neuronal differentiation.Gene, cell, and organ multiplication drives inner ear evolution.Using Drosophila to study mechanisms of hereditary hearing loss.Multichannel optrodes for photonic stimulation

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P2860

Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.

http://www.wikidata.org/entity/Q34146480

description

2012 nî lūn-bûn

@nan

2012 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի հունվարին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.

@ast

Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.

@en

Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.

@nl

type

label

Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.

@ast

Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.

@en

Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.

@nl

prefLabel

Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.

@ast

Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.

@en

Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.

@nl

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JOURNAL.PONE.0030853

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Expression of Neurog1 instead of Atoh1 can partially rescue organ of Corti cell survival.

@en

P2093

Benjamin Kopecky

http://www.w3.org/2001/XMLSchema#string

Israt Jahan

http://www.w3.org/2001/XMLSchema#string

Jennifer Kersigo

http://www.w3.org/2001/XMLSchema#string

Jeremy S Duncan

http://www.w3.org/2001/XMLSchema#string

Ken A Morris

http://www.w3.org/2001/XMLSchema#string

Kirk W Beisel

http://www.w3.org/2001/XMLSchema#string

Lilian E Calisto

http://www.w3.org/2001/XMLSchema#string

Ning Pan

http://www.w3.org/2001/XMLSchema#string

P2860

Divergent functions of the proneural genes Mash1 and Ngn2 in the specification of neuronal subtype identity

The Notch ligand JAG1 is required for sensory progenitor development in the mammalian inner ear

Math5 is required for retinal ganglion cell and optic nerve formation

Canal cristae growth and fiber extension to the outer hair cells of the mouse ear require Prox1 activity

Hey2 regulation by FGF provides a Notch-independent mechanism for maintaining pillar cell fate in the organ of Corti

Atoh1 null mice show directed afferent fiber growth to undifferentiated ear sensory epithelia followed by incomplete fiber retention

Neurogenin and NeuroD direct transcriptional targets and their regulatory enhancers

Notch ligands with contrasting functions: Jagged1 and Delta1 in the mouse inner ear

The role of Math1 in inner ear development: Uncoupling the establishment of the sensory primordium from hair cell fate determination

HPRT-deficient (Lesch-Nyhan) mouse embryos derived from germline colonization by cultured cells

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10.1371/JOURNAL.PONE.0030853

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