Slug inhibits proliferation of human prostate cancer cells via downregulation of cyclin D1 expression.
about
The Role of Connexins in Wound Healing and Repair: Novel Therapeutic ApproachesInduction of Slug by Chronic Exposure to Single-Walled Carbon Nanotubes Promotes Tumor Formation and Metastasis.Slug, a unique androgen-regulated transcription factor, coordinates androgen receptor to facilitate castration resistance in prostate cancerConcomitant Notch activation and p53 deletion trigger epithelial-to-mesenchymal transition and metastasis in mouse gut.Combination of rapamycin, CI-1040, and 17-AAG inhibits metastatic capacity of prostate cancer via Slug inhibition.EPLIN downregulation promotes epithelial-mesenchymal transition in prostate cancer cells and correlates with clinical lymph node metastasis.SLUG promotes prostate cancer cell migration and invasion via CXCR4/CXCL12 axis.Integrated Analysis Reveals together miR-182, miR-200c and miR-221 Can Help in the Diagnosis of Prostate Cancer.Epithelial-mesenchymal transition can suppress major attributes of human epithelial tumor-initiating cells.Inhibition of Snail Family Transcriptional Repressor 2 (SNAI2) Enhances Multidrug Resistance of Hepatocellular Carcinoma CellsSnail/Gfi-1 (SNAG) family zinc finger proteins in transcription regulation, chromatin dynamics, cell signaling, development, and disease3D Porous Chitosan-Alginate Scaffolds as an In Vitro Model for Evaluating Nanoparticle-Mediated Tumor Targeting and Gene Delivery to Prostate Cancer.An evolutionary explanation for the perturbation of the dynamics of metastatic tumors induced by surgery and acute inflammation.Genotypes of cancer stem cells characterized by epithelial-to-mesenchymal transition and proliferation related functions.Slug inhibits the proliferation and tumor formation of human cervical cancer cells by up-regulating the p21/p27 proteins and down-regulating the activity of the Wnt/β-catenin signaling pathway via the trans-suppression Akt1/p-Akt1 expression.Prostate tumor cell plasticity: a consequence of the microenvironment.SNAIL transcription factor increases the motility and invasive capacity of prostate cancer cells.TMPRSS4 induces invasion and proliferation of prostate cancer cells through induction of Slug and cyclin D1.Frequently rearranged and overexpressed δ-catenin is responsible for low sensitivity of prostate cancer cells to androgen receptor and β-catenin antagonists.The PCAT3/PCAT9-miR-203-SNAI2 axis functions as a key mediator for prostate tumor growth and progression.
P2860
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P2860
Slug inhibits proliferation of human prostate cancer cells via downregulation of cyclin D1 expression.
description
2010 nî lūn-bûn
@nan
2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Slug inhibits proliferation of ...... ation of cyclin D1 expression.
@ast
Slug inhibits proliferation of ...... ation of cyclin D1 expression.
@en
Slug inhibits proliferation of ...... ation of cyclin D1 expression.
@nl
type
label
Slug inhibits proliferation of ...... ation of cyclin D1 expression.
@ast
Slug inhibits proliferation of ...... ation of cyclin D1 expression.
@en
Slug inhibits proliferation of ...... ation of cyclin D1 expression.
@nl
prefLabel
Slug inhibits proliferation of ...... ation of cyclin D1 expression.
@ast
Slug inhibits proliferation of ...... ation of cyclin D1 expression.
@en
Slug inhibits proliferation of ...... ation of cyclin D1 expression.
@nl
P2093
P2860
P356
P1433
P1476
Slug inhibits proliferation of ...... ation of cyclin D1 expression.
@en
P2093
Berna Uygur
Calvin Vary
Diana Romero
Lijian Shao
Qiang Ding
Wen-Shu Wu
Zhonghui Zhang
P2860
P304
P356
10.1002/PROS.21213
P577
2010-12-01T00:00:00Z