Bone and muscle loss after spinal cord injury: organ interactions.
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Does Upper Extremity Training Influence Body Composition after Spinal Cord Injury?The bone-fat interface: basic and clinical implications of marrow adiposityMultiple organ dysfunction and systemic inflammation after spinal cord injury: a complex relationshipThe Signature of MicroRNA Dysregulation in Muscle Paralyzed by Spinal Cord Injury Includes Downregulation of MicroRNAs that Target Myostatin SignalingAutomatic and quantitative assessment of regional muscle volume by multi-atlas segmentation using whole-body water-fat MRI.Bone marrow fat accumulation accelerated by high fat diet is suppressed by exercise.Age-related prevalence of low testosterone in men with spinal cord injury.Electrical stimulation modulates Wnt signaling and regulates genes for the motor endplate and calcium binding in muscle of rats with spinal cord transection.The effects of electrical stimulation on body composition and metabolic profile after spinal cord injury--Part II.Impact on bone and muscle area after spinal cord injury.Electroacupuncture in the repair of spinal cord injury: inhibiting the Notch signaling pathway and promoting neural stem cell proliferation.Feasibility of MR-Based Body Composition Analysis in Large Scale Population Studies.Skeletal effects of long-term caloric restriction in rhesus monkeysWhole-body adipose tissue and lean muscle volumes and their distribution across gender and age: MR-derived normative values in a normal-weight Swiss population.The central nervous system (CNS)-independent anti-bone-resorptive activity of muscle contraction and the underlying molecular and cellular signatures.The effects of aging and electrical stimulation exercise on bone after spinal cord injury.Connexin 43 deficiency attenuates loss of trabecular bone and prevents suppression of cortical bone formation during unloading.Proteomic and bioinformatic analyses of spinal cord injury‑induced skeletal muscle atrophy in ratsLow-dose baclofen therapy raised plasma insulin-like growth factor-1 concentrations, but not into the normal range in a predictable and sustained manner in men with chronic spinal cord injury.Lower-extremity muscle atrophy and fat infiltration after chronic spinal cord injury.Decoding Lower Limb Muscle Activity and Kinematics from Cortical Neural Spike Trains during Monkey Performing Stand and Squat Movements.Influences of nutrition and adiposity on bone mineral density in individuals with chronic spinal cord injury: A cross-sectional, observational study.Spinal cord injury sequelae alter drug pharmacokinetics: an overview.Generation and applications of human pluripotent stem cells induced into neural lineages and neural tissues.Potential role of oxidative stress on the prescription of rehabilitation interventions in spinal cord injury.Neuronal involvement in muscular atrophy.Myostatin inhibits osteoblastic differentiation by suppressing osteocyte-derived exosomal microRNA-218: A novel mechanism in muscle-bone communication.Diabetes and disordered bone metabolism (diabetic osteodystrophy): time for recognition.New perspectives on the development of muscle contractures following central motor lesions.Vitamin D and spinal cord injury: should we care?Brief communication: the effects of disuse on the mechanical properties of bone: what unloading tells us about the adaptive nature of skeletal tissue.Anabolic steroids reduce spinal cord injury-related bone loss in rats associated with increased Wnt signaling.Nandrolone normalizes determinants of muscle mass and fiber type after spinal cord injury.Plasticity of TRPV1-Expressing Sensory Neurons Mediating Autonomic Dysreflexia Following Spinal Cord Injury.Temporal modifications in bone following spinal cord injury in rats.Early surgical decompression within 8 hours for traumatic spinal cord injury: Is it beneficial? A meta-analysis.Precision of dual-energy X-ray absorptiometry of the knee and heel: methodology and implications for research to reduce bone mineral loss after spinal cord injury.Focal adhesion kinase signaling is decreased 56 days following spinal cord injury in rat gastrocnemius.A lifestyle intervention program for successfully addressing major cardiometabolic risks in persons with SCI: a three-subject case series.Low-Force Muscle Activity Regulates Energy Expenditure after Spinal Cord Injury.
P2860
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P2860
Bone and muscle loss after spinal cord injury: organ interactions.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Bone and muscle loss after spinal cord injury: organ interactions.
@ast
Bone and muscle loss after spinal cord injury: organ interactions.
@en
Bone and muscle loss after spinal cord injury: organ interactions.
@nl
type
label
Bone and muscle loss after spinal cord injury: organ interactions.
@ast
Bone and muscle loss after spinal cord injury: organ interactions.
@en
Bone and muscle loss after spinal cord injury: organ interactions.
@nl
prefLabel
Bone and muscle loss after spinal cord injury: organ interactions.
@ast
Bone and muscle loss after spinal cord injury: organ interactions.
@en
Bone and muscle loss after spinal cord injury: organ interactions.
@nl
P2093
P2860
P1476
Bone and muscle loss after spinal cord injury: organ interactions.
@en
P2093
Christopher Cardozo
Weiping Qin
William A Bauman
P2860
P356
10.1111/J.1749-6632.2010.05806.X
P407
P577
2010-11-01T00:00:00Z