Cardiac contractile dysfunction during acute hyperglycemia due to impairment of SERCA by polyol pathway-mediated oxidative stress.
about
Pathogenesis and neuroimaging of cerebral large and small vessel disease in type 2 diabetes: A possible link between cerebral and retinal microvascular abnormalitiesAdvanced glycation end-products: modifiable environmental factors profoundly mediate insulin resistanceHow exercise may amend metabolic disturbances in diabetic cardiomyopathyCardiac dysfunction and oxidative stress in the metabolic syndrome: an update on antioxidant therapiesRadiation protection following nuclear power accidents: a survey of putative mechanisms involved in the radioprotective actions of taurine during and after radiation exposureGlucose regulation of load-induced mTOR signaling and ER stress in mammalian heart.Oleanolic acid: a novel cardioprotective agent that blunts hyperglycemia-induced contractile dysfunction.Redox signaling in cardiac myocytes.Glucose metabolism and cardiac hypertrophyCa+2/calmodulin-dependent protein kinase mediates glucose toxicity-induced cardiomyocyte contractile dysfunction.Insulin resistance improves metabolic and contractile efficiency in stressed rat heart.Is the lack of adiponectin associated with increased ER/SR stress and inflammation in the heart?Loss of endoplasmic reticulum Ca2+ homeostasis: contribution to neuronal cell death during cerebral ischemia.Glucose-induced cell signaling in the pathogenesis of diabetic cardiomyopathy.Impairment of calcium ATPases by high glucose and potential pharmacological protection.Use of Ophiocordyceps sinensis (syn. Cordyceps sinensis) combined with angiotensin-converting enzyme inhibitors (ACEI)/angiotensin receptor blockers (ARB) versus ACEI/ARB alone in the treatment of diabetic kidney disease: a meta-analysis.The regulation of sarco(endo)plasmic reticulum calcium-ATPases (SERCA).Damaging effects of hyperglycemia on cardiovascular function: spotlight on glucose metabolic pathways.Electrophysiological basis of metabolic-syndrome-induced cardiac dysfunction.PKC-mediated toxicity of elevated glucose concentration on cardiomyocyte function.Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and activation of SERCA2a.Biomarkers and echocardiography for evaluating the improvement of the ventricular diastolic function after surgical relief of hydronephrosis.Exercise Amaliorates Metabolic Disturbances and Oxidative Stress in Diabetic Cardiomyopathy: Possible Underlying Mechanisms.Detrimental effects of acute hyperglycaemia on the rat heart.
P2860
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P2860
Cardiac contractile dysfunction during acute hyperglycemia due to impairment of SERCA by polyol pathway-mediated oxidative stress.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
name
Cardiac contractile dysfunctio ...... way-mediated oxidative stress.
@ast
Cardiac contractile dysfunctio ...... way-mediated oxidative stress.
@en
Cardiac contractile dysfunctio ...... way-mediated oxidative stress.
@nl
type
label
Cardiac contractile dysfunctio ...... way-mediated oxidative stress.
@ast
Cardiac contractile dysfunctio ...... way-mediated oxidative stress.
@en
Cardiac contractile dysfunctio ...... way-mediated oxidative stress.
@nl
prefLabel
Cardiac contractile dysfunctio ...... way-mediated oxidative stress.
@ast
Cardiac contractile dysfunctio ...... way-mediated oxidative stress.
@en
Cardiac contractile dysfunctio ...... way-mediated oxidative stress.
@nl
P2093
P2860
P1476
Cardiac contractile dysfunctio ...... way-mediated oxidative stress.
@en
P2093
Gennadi M Kravtsov
Sookja K Chung
Stephen Sum Man Chung
Wai Ho Tang
Wing Tim Cheng
Xiao Yong Tong
Xiu Yun Hou
P2860
P304
P356
10.1152/AJPCELL.00137.2010
P577
2010-06-23T00:00:00Z