Mutant DLX 3 disrupts odontoblast polarization and dentin formation. - Wikidata - wikimedia - TriplyDB

Mutant DLX 3 disrupts odontoblast polarization and dentin formation.

Mutant DLX 3 disrupts odontoblast polarization and dentin formation.

http://www.wikidata.org/entity/Q34155863

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Tricho-dento-osseous syndrome: diagnosis and dental management Neural crest deletion of Dlx3 leads to major dentin defects through down-regulation of Dspp Mutation in SSUH2 Causes Autosomal-Dominant Dentin Dysplasia Type I Identification of cells at early and late stages of polarization during odontoblast differentiation using pOBCol3.6GFP and pOBCol2.3GFP transgenic mice Transcriptional factor DLX3 promotes the gene expression of enamel matrix proteins during amelogenesis.MicroRNA 665 Regulates Dentinogenesis through MicroRNA-Mediated Silencing and Epigenetic Mechanisms The transcription factor DLX3 regulates the osteogenic differentiation of human dental follicle precursor cells.Senescence: novel insight into DLX3 mutations leading to enhanced bone formation in Tricho-Dento-Osseous syndrome.The amazing odontoblast: activity, autophagy, and aging.Mineral features of connective dental hard tissues in hypoplastic amelogenesis imperfecta.Odontoblasts: Specialized hard-tissue-forming cells in the dentin-pulp complex.Effects of neurotrophin receptor-mediated MAGE homology on proliferation and odontoblastic differentiation of mouse dental pulp cells.Specific inactivation of Twist1 in the mandibular arch neural crest cells affects the development of the ramus and reveals interactions with hand2.Craniofacial variations in the tricho-dento-osseous syndrome.DLX3-Dependent Regulation of Ion Transporters and Carbonic Anhydrases is Crucial for Enamel Mineralization.

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P2860

Mutant DLX 3 disrupts odontoblast polarization and dentin formation.

http://www.wikidata.org/entity/Q34155863

description

2010 nî lūn-bûn

@nan

2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Mutant DLX 3 disrupts odontoblast polarization and dentin formation.

@ast

Mutant DLX 3 disrupts odontoblast polarization and dentin formation.

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Mutant DLX 3 disrupts odontoblast polarization and dentin formation.

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type

label

Mutant DLX 3 disrupts odontoblast polarization and dentin formation.

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Mutant DLX 3 disrupts odontoblast polarization and dentin formation.

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Mutant DLX 3 disrupts odontoblast polarization and dentin formation.

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prefLabel

Mutant DLX 3 disrupts odontoblast polarization and dentin formation.

@ast

Mutant DLX 3 disrupts odontoblast polarization and dentin formation.

@en

Mutant DLX 3 disrupts odontoblast polarization and dentin formation.

@nl

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J.YDBIO.2010.05.499

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Developmental Biology

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Mutant DLX 3 disrupts odontoblast polarization and dentin formation.

@en

P2093

I S Song

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J Q Feng

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N Haruyama

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P G Robey

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P Gautam

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S J Choi

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T Gao

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Thomas C Hart

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P2860

Identification of a mutation in DLX3 associated with tricho-dento-osseous (TDO) syndrome

WNT10A mutations are a frequent cause of a broad spectrum of ectodermal dysplasias with sex-biased manifestation pattern in heterozygotes

Dlx3 is a crucial regulator of hair follicle differentiation and cycling

Clinical features of tricho-dento-osseous syndrome and presentation of three new cases: an addition to clinical heterogeneity

Increased bone density associated with DLX3 mutation in the tricho-dento-osseous syndrome

Wnt10a regulates dentin sialophosphoprotein mRNA expression and possibly links odontoblast differentiation and tooth morphogenesis

Differential recruitment of Dishevelled provides signaling specificity in the planar cell polarity and Wingless signaling pathways

Reassessing the Dlx code: the genetic regulation of branchial arch skeletal pattern and development

A common DLX3 gene mutation is responsible for tricho-dento-osseous syndrome in Virginia and North Carolina families.

Regulation and function of Dlx3 in vertebrate development.

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682-692

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10.1016/J.YDBIO.2010.05.499

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