NOX4 mediates BMP4-induced upregulation of TRPC1 and 6 protein expressions in distal pulmonary arterial smooth muscle cells - Wikidata - wikimedia - TriplyDB

NOX4 mediates BMP4-induced upregulation of TRPC1 and 6 protein expressions in distal pulmonary arterial smooth muscle cells

NOX4 mediates BMP4-induced upregulation of TRPC1 and 6 protein expressions in distal pulmonary arterial smooth muscle cells

http://www.wikidata.org/entity/Q34156155

about

Calcium and ROS: A mutual interplay The Role of Transient Receptor Potential Channel 6 Channels in the Pulmonary Vasculature NADPH oxidases-do they play a role in TRPC regulation under hypoxia?Critical role of X-box binding protein 1 in NADPH oxidase 4-triggered cardiac hypertrophy is mediated by receptor interacting protein kinase 1.Therapeutic potential of NADPH oxidase 1/4 inhibitors.Bortezomib alleviates experimental pulmonary hypertension by regulating intracellular calcium homeostasis in PASMCs Hydrogen peroxide is a critical regulator of the hypoxia-induced alterations of store-operated Ca2+ entry into rat pulmonary arterial smooth muscle cells.

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NOX4 mediates BMP4-induced upregulation of TRPC1 and 6 protein expressions in distal pulmonary arterial smooth muscle cells

http://www.wikidata.org/entity/Q34156155

description

2014 nî lūn-bûn

@nan

2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

@zh-hk

2014年論文

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2014年論文

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2014年论文

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name

NOX4 mediates BMP4-induced upr ...... y arterial smooth muscle cells

@ast

NOX4 mediates BMP4-induced upr ...... y arterial smooth muscle cells

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NOX4 mediates BMP4-induced upr ...... y arterial smooth muscle cells

@nl

type

label

NOX4 mediates BMP4-induced upr ...... y arterial smooth muscle cells

@ast

NOX4 mediates BMP4-induced upr ...... y arterial smooth muscle cells

@en

NOX4 mediates BMP4-induced upr ...... y arterial smooth muscle cells

@nl

prefLabel

NOX4 mediates BMP4-induced upr ...... y arterial smooth muscle cells

@ast

NOX4 mediates BMP4-induced upr ...... y arterial smooth muscle cells

@en

NOX4 mediates BMP4-induced upr ...... y arterial smooth muscle cells

@nl

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NOX4 mediates BMP4-induced upr ...... y arterial smooth muscle cells

@en

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Jian Wang

http://www.w3.org/2001/XMLSchema#string

Jie Zhang

http://www.w3.org/2001/XMLSchema#string

Lichun Tian

http://www.w3.org/2001/XMLSchema#string

Qian Jiang

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Wenju Lu

http://www.w3.org/2001/XMLSchema#string

Xin Fu

http://www.w3.org/2001/XMLSchema#string

Xiuqing Chen

http://www.w3.org/2001/XMLSchema#string

Yuqin Chen

http://www.w3.org/2001/XMLSchema#string

P2860

Direct signaling by the BMP type II receptor via the cytoskeletal regulator LIMK1

Bone morphogenetic protein 4 enhances canonical transient receptor potential expression, store-operated Ca2+ entry, and basal [Ca2+]i in rat distal pulmonary arterial smooth muscle cells

An abnormal mitochondrial-hypoxia inducible factor-1alpha-Kv channel pathway disrupts oxygen sensing and triggers pulmonary arterial hypertension in fawn hooded rats: similarities to human pulmonary arterial hypertension

The relationships among reactive oxygen species, hypoxia-inducible factor 1alpha and cell proliferation in rat pulmonary arterial smooth muscle cells under hypoxia.

The NADPH oxidase subunit NOX4 is a new target gene of the hypoxia-inducible factor-1.

Classical transient receptor potential 1 and 6 contribute to hypoxic pulmonary hypertension through differential regulation of pulmonary vascular functions.

Human urotensin II is a novel activator of NADPH oxidase in human pulmonary artery smooth muscle cells.

ACCF/AHA 2009 expert consensus document on pulmonary hypertension a report of the American College of Cardiology Foundation Task Force on Expert Consensus Documents and the American Heart Association developed in collaboration with the American Coll

Effects of chronic exposure to cigarette smoke on canonical transient receptor potential expression in rat pulmonary arterial smooth muscle.

Role of mitochondrial reactive oxygen species in hypoxia-dependent increase in intracellular calcium in pulmonary artery myocytes.

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scholarly article

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10.1371/JOURNAL.PONE.0107135

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