Thioredoxin 1 negatively regulates angiotensin II-induced cardiac hypertrophy through upregulation of miR-98/let-7 - Wikidata - wikimedia - TriplyDB

Thioredoxin 1 negatively regulates angiotensin II-induced cardiac hypertrophy through upregulation of miR-98/let-7

Thioredoxin 1 negatively regulates angiotensin II-induced cardiac hypertrophy through upregulation of miR-98/let-7

http://www.wikidata.org/entity/Q34156481

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Overview of MicroRNAs in Cardiac Hypertrophy, Fibrosis, and Apoptosis MicroRNAs Based Therapy of Hypertrophic Cardiomyopathy: The Road Traveled So Far The function of miRNA in cardiac hypertrophy Epigenetic mechanisms underlying cardiac degeneration and regeneration Regulation of cell survival and death by pyridine nucleotides Non-coding RNAs: the "dark matter" of cardiovascular pathophysiology.The miR-19a/b family positively regulates cardiomyocyte hypertrophy by targeting atrogin-1 and MuRF-1 Thioredoxins, glutaredoxins, and peroxiredoxins--molecular mechanisms and health significance: from cofactors to antioxidants to redox signaling Sirtuin 6 protects cardiomyocytes from hypertrophy in vitro via inhibition of NF-κB-dependent transcriptional activity O-GlcNAcylation involvement in high glucose-induced cardiac hypertrophy via ERK1/2 and cyclin D2 Cardiac hypertrophy is negatively regulated by miR-541.Non-coding RNAs in cardiac hypertrophy.A cardiac-enriched microRNA, miR-378, blocks cardiac hypertrophy by targeting Ras signaling.Upregulation of TRB2 induced by miR-98 in the early lesions of large artery of type-2 diabetic rat.MicroRNAs and cardiac sarcoplasmic reticulum calcium ATPase-2 in human myocardial infarction: expression and bioinformatic analysis.Strategies to deliver microRNAs as potential therapeutics in the treatment of cardiovascular pathology.Circulating miR-30a, miR-195 and let-7b associated with acute myocardial infarction Modulation of signaling mechanisms in the heart by thioredoxin 1.Restriction of big hearts by a small RNA AMP activated protein kinase is indispensable for myocardial adaptation to caloric restriction in mice.Identification of thioredoxin target protein networks in cardiac tissues of a transgenic mouse.The Role of MicroRNAs in Regulatory T Cells and in the Immune Response.MicroRNAs in Human Diseases: From Cancer to Cardiovascular Disease.The miRNA Transcriptome Directly Reflects the Physiological and Biochemical Differences between Red, White, and Intermediate Muscle Fiber Types.Cardiac hypertrophy is positively regulated by MicroRNA miR-23a.The Human Myotrophin Variant Attenuates MicroRNA-Let-7 Binding Ability but Not Risk of Left Ventricular Hypertrophy in Human Essential Hypertension.Expression Profiling of Circulating MicroRNAs in Canine Myxomatous Mitral Valve Disease.MicroRNA therapeutics in cardiovascular medicine.Cardiovascular redox and ox stress proteomics.Enhanced Cardiac Regenerative Ability of Stem Cells After Ischemia-Reperfusion Injury: Role of Human CD34+ Cells Deficient in MicroRNA-377.Serum thioredoxin 1 level has close relation with myocardial damage amount in acute myocardial infarction patients.Metabolic stress in the myocardium: adaptations of gene expression.A cardiac myocyte-restricted Lin28/let-7 regulatory axis promotes hypoxia-mediated apoptosis by inducing the AKT signaling suppressor PIK3IP1.Glucocorticoids suppress T cell function by up-regulating microRNA-98 miR-218 Involvement in Cardiomyocyte Hypertrophy Is Likely through Targeting REST.Angiotensin II and oxidative stress in the failing heart Thioredoxin 1 is essential for sodium sulfide-mediated cardioprotection in the setting of heart failure Let-7 in cardiovascular diseases, heart development and cardiovascular differentiation from stem cells.Let-7a Is an Antihypertrophic Regulator in the Heart via Targeting Calmodulin.Cardinal roles of miRNA in cardiac development and disease.

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Thioredoxin 1 negatively regulates angiotensin II-induced cardiac hypertrophy through upregulation of miR-98/let-7

http://www.wikidata.org/entity/Q34156481

description

2010 nî lūn-bûn

@nan

2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Thioredoxin 1 negatively regul ...... h upregulation of miR-98/let-7

@ast

Thioredoxin 1 negatively regul ...... h upregulation of miR-98/let-7

@en

Thioredoxin 1 negatively regul ...... h upregulation of miR-98/let-7

@nl

type

label

Thioredoxin 1 negatively regul ...... h upregulation of miR-98/let-7

@ast

Thioredoxin 1 negatively regul ...... h upregulation of miR-98/let-7

@en

Thioredoxin 1 negatively regul ...... h upregulation of miR-98/let-7

@nl

prefLabel

Thioredoxin 1 negatively regul ...... h upregulation of miR-98/let-7

@ast

Thioredoxin 1 negatively regul ...... h upregulation of miR-98/let-7

@en

Thioredoxin 1 negatively regul ...... h upregulation of miR-98/let-7

@nl

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CIRCRESAHA.110.228437

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Circulation Research

P1476

Thioredoxin 1 negatively regul ...... h upregulation of miR-98/let-7

@en

P2093

Junichi Sadoshima

http://www.w3.org/2001/XMLSchema#string

Maha Abdellatif

http://www.w3.org/2001/XMLSchema#string

Peiyong Zhai

http://www.w3.org/2001/XMLSchema#string

Tetsuro Ago

http://www.w3.org/2001/XMLSchema#string

Yanfei Yang

http://www.w3.org/2001/XMLSchema#string

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Mammalian thioredoxin is a direct inhibitor of apoptosis signal-regulating kinase (ASK) 1.

MicroRNA-98 and let-7 confer cholangiocyte expression of cytokine-inducible Src homology 2-containing protein in response to microbial challenge.

Selective blockade of microRNA processing by Lin28

let-7 Overexpression leads to an increased fraction of cells in G2/M, direct down-regulation of Cdc34, and stabilization of Wee1 kinase in primary fibroblasts

Widespread microRNA repression by Myc contributes to tumorigenesis

Disrupting the pairing between let-7 and Hmga2 enhances oncogenic transformation

A signature pattern of stress-responsive microRNAs that can evoke cardiac hypertrophy and heart failure

Thioredoxin and ventricular remodeling

RAS is regulated by the let-7 microRNA family

The let-7 MicroRNA family members mir-48, mir-84, and mir-241 function together to regulate developmental timing in Caenorhabditis elegans

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305-313

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scholarly article

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10.1161/CIRCRESAHA.110.228437

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3

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P478

108

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2010-12-23T00:00:00Z

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21183740

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3249645

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