The peritoneum is both a source and target of TGF-β in women with endometriosis.
about
Endometriosis: where are we and where are we going?Implications of immune dysfunction on endometriosis associated infertilityRelevant human tissue resources and laboratory models for use in endometriosis research.Epithelial-to-Mesenchymal Transition in the Female Reproductive Tract: From Normal Functioning to Disease Pathology.Enhanced epithelial to mesenchymal transition (EMT) and upregulated MYC in ectopic lesions contribute independently to endometriosis.Peritoneal VEGF-A expression is regulated by TGF-β1 through an ID1 pathway in women with endometriosis.PAI-1 secretion of endometrial and endometriotic cells is Smad2/3- and ERK1/2-dependent and influences cell adhesionFunctional expression of aryl hydrocarbon receptor on mast cells populating human endometriotic tissues.Effects of matrix stiffness on epithelial to mesenchymal transition-like processes of endometrial epithelial cells: Implications for the pathogenesis of endometriosis.ID2 mediates the transforming growth factor-β1-induced Warburg-like effect seen in the peritoneum of women with endometriosis.TNFα-induced IKKβ complex activation influences epithelial, but not stromal cell survival in endometriosis.Soft matrices inhibit cell proliferation and inactivate the fibrotic phenotype of deep endometriotic stromal cells in vitro.Transforming growth factor β1 signaling coincides with epithelial-mesenchymal transition and fibroblast-to-myofibroblast transdifferentiation in the development of adenomyosis in mice.Transforming growth factor β1 enhances adhesion of endometrial cells to mesothelium by regulating integrin expression.Reduced Transforming Growth Factor-β Activity in the Endometrium of Women With Heavy Menstrual Bleeding.Research Priorities for Endometriosis.Phenotypic characterization of adenomyosis occurring at the inner and outer myometrium.TGF-beta1 from Endometriomas Promotes Fibrosis in Surrounding Ovarian Tissues via Smad2/3 Signaling.Suppressive regulatory T cells and latent transforming growth factor-β-expressing macrophages are altered in the peritoneal fluid of patients with endometriosis.The role of TGF-β in the pathophysiology of peritoneal endometriosis.mir-126 rs4636297 and TGFβRI rs334348 functional gene variants are associated with susceptibility to endometriosis and its severity.Hypoxia-inducible factor 1α-induced epithelial-mesenchymal transition of endometrial epithelial cells may contribute to the development of endometriosis.Cancer driver mutations in endometriosis: Variations on the major theme of fibrogenesisThe Endometriotic Tumor Microenvironment in Ovarian Cancer
P2860
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P2860
The peritoneum is both a source and target of TGF-β in women with endometriosis.
description
2014 nî lūn-bûn
@nan
2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
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2014年学术文章
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2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
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2014年學術文章
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name
The peritoneum is both a source and target of TGF-β in women with endometriosis.
@ast
The peritoneum is both a source and target of TGF-β in women with endometriosis.
@en
The peritoneum is both a source and target of TGF-β in women with endometriosis.
@nl
type
label
The peritoneum is both a source and target of TGF-β in women with endometriosis.
@ast
The peritoneum is both a source and target of TGF-β in women with endometriosis.
@en
The peritoneum is both a source and target of TGF-β in women with endometriosis.
@nl
prefLabel
The peritoneum is both a source and target of TGF-β in women with endometriosis.
@ast
The peritoneum is both a source and target of TGF-β in women with endometriosis.
@en
The peritoneum is both a source and target of TGF-β in women with endometriosis.
@nl
P2093
P2860
P1433
P1476
The peritoneum is both a source and target of TGF-β in women with endometriosis
@en
P2093
Jeremy K Brown
Vicky J Young
W Colin Duncan
P2860
P304
P356
10.1371/JOURNAL.PONE.0106773
P407
P577
2014-09-10T00:00:00Z