Pathogenic and nonpathogenic hantaviruses differentially regulate endothelial cell responses. - Wikidata - wikimedia - TriplyDB

Pathogenic and nonpathogenic hantaviruses differentially regulate endothelial cell responses.

Pathogenic and nonpathogenic hantaviruses differentially regulate endothelial cell responses.

http://www.wikidata.org/entity/Q34161805

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Alpha/beta interferon (IFN-alpha/beta)-independent induction of IFN-lambda1 (interleukin-29) in response to Hantaan virus infection Depletion of Alveolar Macrophages Does Not Prevent Hantavirus Disease Pathogenesis in Golden Syrian Hamsters What Do We Know about How Hantaviruses Interact with Their Different Hosts?Hantaviral Proteins: Structure, Functions, and Role in Hantavirus Infection Dengue Virus Induces Novel Changes in Gene Expression of Human Umbilical Vein Endothelial Cells Hantavirus protein interactions regulate cellular functions and signaling responses.Peptide antagonists that inhibit Sin Nombre virus and hantaan virus entry through the beta3-integrin receptor Andes virus nucleocapsid protein interrupts protein kinase R dimerization to counteract host interference in viral protein synthesis.Thrombocytopenia and acute renal failure in Puumala hantavirus infections Immunological mechanisms mediating hantavirus persistence in rodent reservoirs.Hypoxia induces permeability and giant cell responses of Andes virus-infected pulmonary endothelial cells by activating the mTOR-S6K signaling pathway.Vascular events in viral hemorrhagic fevers: a comparative study of dengue and hantaviruses.Pathophysiology of hantavirus pulmonary syndrome in rhesus macaques.New World hantaviruses activate IFNlambda production in type I IFN-deficient vero E6 cells.Seoul virus suppresses NF-kappaB-mediated inflammatory responses of antigen presenting cells from Norway rats.Hantavirus interferon regulation and virulence determinants β2 integrin mediates hantavirus-induced release of neutrophil extracellular traps.Hantaviruses induce antiviral and pro-inflammatory innate immune responses in astrocytic cells and the brain Hantaviruses in the americas and their role as emerging pathogens Pathogenesis and host response in Syrian hamsters following intranasal infection with Andes virus.Differential antiviral response of endothelial cells after infection with pathogenic and nonpathogenic hantaviruses Induction of innate immune response genes by Sin Nombre hantavirus does not require viral replication.Andes virus regulation of cellular microRNAs contributes to hantavirus-induced endothelial cell permeability.Cardiopulmonary involvement in Puumala hantavirus infection.Characterization of two substrains of Puumala virus that show phenotypes that are different from each other and from the original strain Experimental Andes virus infection in deer mice: characteristics of infection and clearance in a heterologous rodent host VEGFR2 and Src kinase inhibitors suppress Andes virus-induced endothelial cell permeability The C-terminal 42 residues of the Tula virus Gn protein regulate interferon induction.The pathogenic NY-1 hantavirus G1 cytoplasmic tail inhibits RIG-I- and TBK-1-directed interferon responses.Involvement of the Akt/NF-κB pathways in the HTNV-mediated increase of IL-6, CCL5, ICAM-1, and VCAM-1 in HUVECs.Characterization of the host response to pichinde virus infection in the Syrian golden hamster by species-specific kinome analysis Genomic profiling of host responses to Lassa virus: therapeutic potential from primate to man Effect of Puumala hantavirus infection on human umbilical vein endothelial cell hemostatic function: platelet interactions, increased tissue factor expression and fibrinolysis regulator release.Andes and Prospect Hill hantaviruses differ in early induction of interferon although both can downregulate interferon signaling.Degrons at the C terminus of the pathogenic but not the nonpathogenic hantavirus G1 tail direct proteasomal degradation Induction of pathogenic sets of genes in macrophages and neurons in NeuroAIDS.Insights into host responses against pathogens from transcriptional profiling.Andes virus infection of lymphatic endothelial cells causes giant cell and enhanced permeability responses that are rapamycin and vascular endothelial growth factor C sensitive.Hantavirus regulation of type I interferon responses.Elevated VEGF Levels in Pulmonary Edema Fluid and PBMCs from Patients with Acute Hantavirus Pulmonary Syndrome.

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P2860

Pathogenic and nonpathogenic hantaviruses differentially regulate endothelial cell responses.

http://www.wikidata.org/entity/Q34161805

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2002 nî lūn-bûn

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Proceedings of the National Academy of Sciences of the United States of America

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Pathogenic and nonpathogenic h ...... te endothelial cell responses.

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Erich R Mackow

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Erika Geimonen

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Irina N Gavrilovskaya

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Salih S Kocer

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Sherry Neff

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Tracy Raymond

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P2860

Role of alphavbeta3 integrin in the activation of vascular endothelial growth factor receptor-2.

Neuropilin-1 is expressed by endothelial and tumor cells as an isoform-specific receptor for vascular endothelial growth factor

Antiviral actions of interferons

Vascular permeability factor/vascular endothelial growth factor, microvascular hyperpermeability, and angiogenesis

Hantavirus infection induces the expression of RANTES and IP-10 without causing increased permeability in human lung microvascular endothelial cells

Hantaviruses: A Global Disease Problem

Tumor necrosis factor receptor-associated factor (TRAF) family: adapter proteins that mediate cytokine signaling

Tumor necrosis factor receptor-associated factors (TRAFs)

The splice variants of vascular endothelial growth factor (VEGF) and their receptors

Signal transduction and signal modulation by cell adhesion receptors: the role of integrins, cadherins, immunoglobulin-cell adhesion molecules, and selectins

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10.1073/PNAS.192298899

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99

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