Virulence control in group A Streptococcus by a two-component gene regulatory system: global expression profiling and in vivo infection modeling.
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Microevolution of group A streptococci in vivo: capturing regulatory networks engaged in sociomicrobiology, niche adaptation, and hypervirulenceA genome-wide analysis of small regulatory RNAs in the human pathogen group A StreptococcusPhages and the evolution of bacterial pathogens: from genomic rearrangements to lysogenic conversionTrxR, a new CovR-repressed response regulator that activates the Mga virulence regulon in group A StreptococcusA streptococcal protease that degrades CXC chemokines and impairs bacterial clearance from infected tissuesGenome-wide analysis of group a streptococci reveals a mutation that modulates global phenotype and disease specificity.Stress Physiology of Lactic Acid BacteriaThe Mechanisms of Virulence Regulation by Small Noncoding RNAs in Low GC Gram-Positive PathogensFunctional analysis of the Mycobacterium tuberculosis MprAB two-component signal transduction systemcovR Mediated Antibiofilm Activity of 3-Furancarboxaldehyde Increases the Virulence of Group A StreptococcusStreptococcus pyogenes SpyCEP influences host-pathogen interactions during infection in a murine air pouch modelThermoregulation of capsule production by Streptococcus pyogenesA Two-Component Regulatory System Impacts Extracellular Membrane-Derived Vesicle Production in Group A StreptococcusYvqE and CovRS of Group A Streptococcus Play a Pivotal Role in Viability and Phenotypic Adaptations to Multiple Environmental StressesThreonine phosphorylation prevents promoter DNA binding of the Group B Streptococcus response regulator CovR.Regulation of gbpC expression in Streptococcus mutans.Regulation of CovR expression in Group B Streptococcus impacts blood-brain barrier penetration.Evolutionary Constraints Shaping Streptococcus pyogenes-Host Interactions.Distinct single amino acid replacements in the control of virulence regulator protein differentially impact streptococcal pathogenesisPhylogenomic analysis of natural selection pressure in Streptococcus genomes.The Abi-domain protein Abx1 interacts with the CovS histidine kinase to control virulence gene expression in group B Streptococcus.Novel algorithms reveal streptococcal transcriptomes and clues about undefined genesSalK/SalR, a two-component signal transduction system, is essential for full virulence of highly invasive Streptococcus suis serotype 2.Transcriptome adaptation of group B Streptococcus to growth in human amniotic fluid.The transcriptome of the nosocomial pathogen Enterococcus faecalis V583 reveals adaptive responses to growth in blood.Genome-wide analyses of gene expression during mouse endochondral ossificationSerotype- and strain- dependent contribution of the sensor kinase CovS of the CovRS two-component system to Streptococcus pyogenes pathogenesisA combination of independent transcriptional regulators shapes bacterial virulence gene expression during infectionHighly frequent mutations in negative regulators of multiple virulence genes in group A streptococcal toxic shock syndrome isolates.Dual-site phosphorylation of the control of virulence regulator impacts group a streptococcal global gene expression and pathogenesisClindamycin-induced CovS-mediated regulation of the production of virulent exoproteins streptolysin O, NAD glycohydrolase, and streptokinase in Streptococcus pyogenes.Inactivation of DNA-binding response regulator Sak189 abrogates beta-antigen expression and affects virulence of Streptococcus agalactiaeEmerging role of the interleukin-8 cleaving enzyme SpyCEP in clinical Streptococcus pyogenes infectionWiring the streptococcal network for alternative lifestylesStreptococcal collagen-like protein A and general stress protein 24 are immunomodulating virulence factors of group A Streptococcus.Streptolysin S contributes to group A streptococcal translocation across an epithelial barrier.Regulation of virulence by a two-component system in group B streptococcus.Activation of the SMU.1882 transcription by CovR in Streptococcus mutans.Impact of the SpeB protease on binding of the complement regulatory proteins factor H and factor H-like protein 1 by Streptococcus pyogenes.Extracellular deoxyribonuclease made by group A Streptococcus assists pathogenesis by enhancing evasion of the innate immune response.
P2860
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P2860
Virulence control in group A Streptococcus by a two-component gene regulatory system: global expression profiling and in vivo infection modeling.
description
2002 nî lūn-bûn
@nan
2002 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Virulence control in group A S ...... nd in vivo infection modeling.
@ast
Virulence control in group A S ...... nd in vivo infection modeling.
@en
Virulence control in group A S ...... nd in vivo infection modeling.
@nl
type
label
Virulence control in group A S ...... nd in vivo infection modeling.
@ast
Virulence control in group A S ...... nd in vivo infection modeling.
@en
Virulence control in group A S ...... nd in vivo infection modeling.
@nl
prefLabel
Virulence control in group A S ...... nd in vivo infection modeling.
@ast
Virulence control in group A S ...... nd in vivo infection modeling.
@en
Virulence control in group A S ...... nd in vivo infection modeling.
@nl
P2093
P2860
P356
P1476
Virulence control in group A S ...... nd in vivo infection modeling.
@en
P2093
Cristi A Lux Migliaccio
Daniel E Sturdevant
Gerald J Adams
June R Scott
Kimmo Virtaneva
Laura M Smoot
Michael J Federle
Morag R Graham
Stephen F Porcella
P2860
P304
13855-13860
P356
10.1073/PNAS.202353699
P407
P577
2002-10-07T00:00:00Z