Obligatory role of ASK1 in the apoptotic surge of K+ currents. - Wikidata - wikimedia - TriplyDB

Obligatory role of ASK1 in the apoptotic surge of K+ currents.

Obligatory role of ASK1 in the apoptotic surge of K+ currents.

http://www.wikidata.org/entity/Q34164795

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Suppression of a pro-apoptotic K+ channel as a mechanism for hepatitis C virus persistence Glucose-dependent insulinotropic polypeptide signaling in pancreatic β-cells and adipocytes Oxidation of KCNB1 K(+) channels in central nervous system and beyond Voltage-gated potassium channels at the crossroads of neuronal function, ischemic tolerance, and neurodegeneration.Apoptotic surface delivery of K+ channels.The neurophysiology and pathology of brain zinc.Microglia induce neurotoxicity via intraneuronal Zn(2+) release and a K(+) current surge.Convergent Ca2+ and Zn2+ signaling regulates apoptotic Kv2.1 K+ currents The role of intracellular zinc release in aging, oxidative stress, and Alzheimer's disease.Enhancement of TWIK-related acid-sensitive potassium channel 3 (TASK3) two-pore domain potassium channel activity by tumor necrosis factor α.Pancreatic β-cell prosurvival effects of the incretin hormones involve post-translational modification of Kv2.1 delayed rectifier channels.Regulation of apoptotic potassium currents by coordinated zinc-dependent signalling.Zn2+-induced disruption of neuronal mitochondrial function: Synergism with Ca2+, critical dependence upon cytosolic Zn2+ buffering, and contributions to neuronal injury.Targeting a Potassium Channel/Syntaxin Interaction Ameliorates Cell Death in Ischemic Stroke.

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Obligatory role of ASK1 in the apoptotic surge of K+ currents.

http://www.wikidata.org/entity/Q34164795

description

2005 nî lūn-bûn

@nan

2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2005年の論文

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2005年論文

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2005年論文

@zh-hant

2005年論文

@zh-hk

2005年論文

@zh-mo

2005年論文

@zh-tw

2005年论文

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name

Obligatory role of ASK1 in the apoptotic surge of K+ currents.

@ast

Obligatory role of ASK1 in the apoptotic surge of K+ currents.

@en

Obligatory role of ASK1 in the apoptotic surge of K+ currents.

@nl

type

label

Obligatory role of ASK1 in the apoptotic surge of K+ currents.

@ast

Obligatory role of ASK1 in the apoptotic surge of K+ currents.

@en

Obligatory role of ASK1 in the apoptotic surge of K+ currents.

@nl

prefLabel

Obligatory role of ASK1 in the apoptotic surge of K+ currents.

@ast

Obligatory role of ASK1 in the apoptotic surge of K+ currents.

@en

Obligatory role of ASK1 in the apoptotic surge of K+ currents.

@nl

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P356

J.NEULET.2005.06.024

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Neuroscience Letters

P1476

Obligatory role of ASK1 in the apoptotic surge of K+ currents.

@en

P2093

Elias Aizenman

http://www.w3.org/2001/XMLSchema#string

Mandar A Aras

http://www.w3.org/2001/XMLSchema#string

P2860

Activation of apoptosis signal-regulating kinase 1 by the stress-induced activating phosphorylation of pre-formed oligomer

ASK1 is essential for endoplasmic reticulum stress-induced neuronal cell death triggered by expanded polyglutamine repeats

Induction of apoptosis by ASK1, a mammalian MAPKKK that activates SAPK/JNK and p38 signaling pathways

Mammalian thioredoxin is a direct inhibitor of apoptosis signal-regulating kinase (ASK) 1.

MAPKK-independent activation of p38alpha mediated by TAB1-dependent autophosphorylation of p38alpha

p38 activation is required upstream of potassium current enhancement and caspase cleavage in thiol oxidant-induced neuronal apoptosis

Peroxynitrite-induced neuronal apoptosis is mediated by intracellular zinc release and 12-lipoxygenase activation

MAPK cascade signalling and synaptic plasticity

Potassium is a critical regulator of apoptotic enzymes in vitro and in vivo.

Activation of apoptosis signal-regulating kinase 1 (ASK1) by tumor necrosis factor receptor-associated factor 2 requires prior dissociation of the ASK1 inhibitor thioredoxin.

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136-140

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scholarly article

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10.1016/J.NEULET.2005.06.024

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3

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387

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7738583

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16006035

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apoptotic process

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2947746

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