Mechanisms contributing to cerebral infarct size after stroke: gender, reperfusion, T lymphocytes, and Nox2-derived superoxide - Wikidata - wikimedia - TriplyDB

Mechanisms contributing to cerebral infarct size after stroke: gender, reperfusion, T lymphocytes, and Nox2-derived superoxide

Mechanisms contributing to cerebral infarct size after stroke: gender, reperfusion, T lymphocytes, and Nox2-derived superoxide

http://www.wikidata.org/entity/Q34171222

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A combined pre-clinical meta-analysis and randomized confirmatory trial approach to improve data validity for therapeutic target validation Cerebral Small Vessel Disease: Targeting Oxidative Stress as a Novel Therapeutic Strategy?Antimicrobial peptides and complement in neonatal hypoxia-ischemia induced brain damage Neuroprotection after stroke by targeting NOX4 as a source of oxidative stress Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular system Inflammatory responses in hypoxic ischemic encephalopathy Cross-talk between the inflammatory response, sympathetic activation and pulmonary infection in the ischemic stroke Inflammation after Ischemic Stroke: The Role of Leukocytes and Glial Cells Oxidative Stress in Hypoxic-Ischemic Encephalopathy: Molecular Mechanisms and Therapeutic Strategies.The immunology of stroke: from mechanisms to translation Molecular dialogs between the ischemic brain and the peripheral immune system: dualistic roles in injury and repair Targeting microglial activation in stroke therapy: pharmacological tools and gender effects.Innate inflammatory responses in stroke: mechanisms and potential therapeutic targets.Nox2 oxidase activity accounts for the oxidative stress and vasomotor dysfunction in mouse cerebral arteries following ischemic stroke.Brain immune cell composition and functional outcome after cerebral ischemia: comparison of two mouse strains NADPH oxidase-2: linking glucose, acidosis, and excitotoxicity in stroke.Role of dihydrotestosterone in post-stroke peripheral immunosuppression after cerebral ischemia Therapy with recombinant T-cell receptor ligand reduces infarct size and infiltrating inflammatory cells in brain after middle cerebral artery occlusion in mice Increased brain injury and worsened neurological outcome in interleukin-4 knockout mice after transient focal cerebral ischemia Sex-independent neuroprotection with minocycline after experimental thromboembolic stroke.Estrogen neuroprotection and the critical period hypothesis.Importance of T lymphocytes in brain injury, immunodeficiency, and recovery after cerebral ischemia.Oestrogen signalling and neuroprotection in cerebral ischaemia.Combating oxidative stress in vascular disease: NADPH oxidases as therapeutic targets The 1027th target candidate in stroke: Will NADPH oxidase hold up?Reperfusion rather than ischemia drives the formation of ubiquitin aggregates after middle cerebral artery occlusion Effect of Inducible Co-Stimulatory Molecule siRNA in Cerebral Infarction Rat Models Over-expression of DSCR1 protects against post-ischemic neuronal injury Neuroprotective effect of an angiotensin receptor type 2 agonist following cerebral ischemia in vitro and in vivo.Blood biomarkers role in acute ischemic stroke patients: higher is worse or better?Post-stroke inflammation and the potential efficacy of novel stem cell therapies: focus on amnion epithelial cells.Method parameters' impact on mortality and variability in mouse stroke experiments: a meta-analysis.Histopathological Investigation of Different MCAO Modalities and Impact of Autologous Bone Marrow Mononuclear Cell Administration in an Ovine Stroke Model.Interaction between sex and apolipoprotein e genetic background in a murine model of intracerebral hemorrhage.Phenotypic changes in immune cell subsets reflect increased infarct volume in male vs. female mice.Leukocyte infiltration in experimental stroke Pharmacologically induced hypothermia via TRPV1 channel agonism provides neuroprotection following ischemic stroke when initiated 90 min after reperfusion.Perioperative organ injury.Immune cell infiltration in malignant middle cerebral artery infarction: comparison with transient cerebral ischemia.NADPH oxidases as therapeutic targets in ischemic stroke.

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Mechanisms contributing to cerebral infarct size after stroke: gender, reperfusion, T lymphocytes, and Nox2-derived superoxide

http://www.wikidata.org/entity/Q34171222

description

2010 nî lūn-bûn

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2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2010 թվականի փետրվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Mechanisms contributing to cer ...... s, and Nox2-derived superoxide

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Mechanisms contributing to cer ...... s, and Nox2-derived superoxide

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Mechanisms contributing to cer ...... s, and Nox2-derived superoxide

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Mechanisms contributing to cer ...... s, and Nox2-derived superoxide

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Mechanisms contributing to cer ...... s, and Nox2-derived superoxide

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Mechanisms contributing to cer ...... s, and Nox2-derived superoxide

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Journal of Cerebral Blood Flow & Metabolism

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Mechanisms contributing to cer ...... s, and Nox2-derived superoxide

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Anja E Mast

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Anna K Walduck

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Brad R S Broughton

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Christopher G Sobey

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Elizabeth Guida

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Grant R Drummond

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Henry Diep

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Katherine A Jackman

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Stavros Selemidis

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Vanessa H Brait

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P2860

Effect of neutrophil depletion on gelatinase expression, edema formation and hemorrhagic transformation after focal ischemic stroke.

T cells express a phagocyte-type NADPH oxidase that is activated after T cell receptor stimulation

Expression of nitric oxide synthase (NOS)-2 following permanent focal ischemia and the role of nitric oxide in infarct generation in male, female and NOS-2 gene-deficient mice.

Effect of suture size and carotid clip application upon blood flow and infarct volume after permanent and temporary middle cerebral artery occlusion in mice.

Vascular cell adhesion molecule-1 (VCAM-1) gene transcription and expression are regulated through an antioxidant-sensitive mechanism in human vascular endothelial cells.

The role of leukocytes following cerebral ischemia: pathogenic variable or bystander reaction to emerging infarct?

Reduced susceptibility to ischemic brain injury and N-methyl-D-aspartate-mediated neurotoxicity in cyclooxygenase-2-deficient mice.

Role of the T cell in the genesis of angiotensin II induced hypertension and vascular dysfunction.

Stroke and T-cells.

Surface expression of alpha 4 integrin by CD4 T cells is required for their entry into brain parenchyma

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1306-1317

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10.1038/JCBFM.2010.14

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7

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30

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20145655

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