Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain. - Wikidata - wikimedia - TriplyDB

Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain.

Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain.

http://www.wikidata.org/entity/Q34178989

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The pharmacology of cyclic nucleotide-gated channels: emerging from the darkness Delayed functional expression of neuronal chemokine receptors following focal nerve demyelination in the rat: a mechanism for the development of chronic sensitization of peripheral nociceptors Noise-induced plasticity of KCNQ2/3 and HCN channels underlies vulnerability and resilience to tinnitus.Local anesthetic inhibits hyperpolarization-activated cationic currents Alterations in the spontaneous discharge patterns of single units in the dorsal cochlear nucleus following intense sound exposure.The pelvis-kidney junction contains HCN3, a hyperpolarization-activated cation channel that triggers ureter peristalsis.Urothelial Tight Junction Barrier Dysfunction Sensitizes Bladder Afferents Chronic cervical radiculopathic pain is associated with increased excitability and hyperpolarization-activated current ( Ih) in large-diameter dorsal root ganglion neurons Contribution of primary afferent channels to neuropathic pain.Modulators of calcium influx regulate membrane excitability in rat dorsal root ganglion neurons Immunostaining for the α3 isoform of the Na+/K+-ATPase is selective for functionally identified muscle spindle afferents in vivo.HCN1 and HCN2 in Rat DRG neurons: levels in nociceptors and non-nociceptors, NT3-dependence and influence of CFA-induced skin inflammation on HCN2 and NT3 expression Hyperpolarization-activated cation current (Ih) is an ethanol target in midbrain dopamine neurons of mice.Control of somatic membrane potential in nociceptive neurons and its implications for peripheral nociceptive transmission.Arthritis and pain. Future targets to control osteoarthritis pain HCN channels in behavior and neurological disease: too hyper or not active enough?Pharmacogenetics of new analgesics.Comprehensive RNA-Seq expression analysis of sensory ganglia with a focus on ion channels and GPCRs in Trigeminal ganglia Differential modulation of repetitive firing and synchronous network activity in neocortical interneurons by inhibition of A-type K(+) channels and Ih.cAMP control of HCN2 channel Mg2+ block reveals loose coupling between the cyclic nucleotide-gating ring and the pore.Corticospinal-specific HCN expression in mouse motor cortex: I(h)-dependent synaptic integration as a candidate microcircuit mechanism involved in motor control Sympathetic sprouting near sensory neurons after nerve injury occurs preferentially on spontaneously active cells and is reduced by early nerve block.Inhibition of Hyperpolarization-Activated Cation Current in Medium-Sized DRG Neurons Contributed to the Antiallodynic Effect of Methylcobalamin in the Rat of a Chronic Compression of the DRG.Bursting activity in myelinated sensory neurons plays a key role in pain behavior induced by localized inflammation of the rat sensory ganglion Cocaine sensitization inhibits the hyperpolarization-activated cation current Ih and reduces cell size in dopamine neurons of the ventral tegmental area Local inflammation in rat dorsal root ganglion alters excitability and ion currents in small-diameter sensory neurons.A Novel Carbamoyloxy Arylalkanoyl Arylpiperazine Compound (SKL-NP) Inhibits Hyperpolarization-Activated Cyclic Nucleotide-Gated (HCN) Channel Currents in Rat Dorsal Root Ganglion Neurons.Upregulation of Ih expressed in IB4-negative Aδ nociceptive DRG neurons contributes to mechanical hypersensitivity associated with cervical radiculopathic pain.Systemic antiinflammatory corticosteroid reduces mechanical pain behavior, sympathetic sprouting, and elevation of proinflammatory cytokines in a rat model of neuropathic pain Expression and properties of hyperpolarization-activated current in rat dorsal root ganglion neurons with known sensory function Activity-dependent slowing of conduction velocity in uninjured L4 C fibers increases after an L5 spinal nerve injury in the rat.Lidocaine Inhibits HCN Currents in Rat Spinal Substantia Gelatinosa Neurons.HCN4 subunit expression in fast-spiking interneurons of the rat spinal cord and hippocampus.HCN1 channels as targets for anesthetic and nonanesthetic propofol analogs in the amelioration of mechanical and thermal hyperalgesia in a mouse model of neuropathic pain.A role for polymodal C-fiber afferents in nonhistaminergic itch A novel intrinsic analgesic mechanism: the enhancement of the conduction failure along polymodal nociceptive C-fibers Hyperpolarization-activated current (I(h)) contributes to excitability of primary sensory neurons in rats.Methylcobalamin: a potential vitamin of pain killer.ZD 7288, an HCN channel blocker, attenuates chronic visceral pain in irritable bowel syndrome-like rats.Deconstructing the neuropathic pain phenotype to reveal neural mechanisms.

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P2860

Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain.

http://www.wikidata.org/entity/Q34178989

description

2003 nî lūn-bûn

@nan

2003 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի փետրվարին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

@zh-hk

2003年論文

@zh-mo

2003年論文

@zh-tw

2003年论文

@wuu

name

Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain.

@ast

Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain.

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Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain.

@nl

type

label

Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain.

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Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain.

@en

Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain.

@nl

prefLabel

Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain.

@ast

Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain.

@en

Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain.

@nl

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JNEUROSCI.23-04-01169.2003

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Journal of Neuroscience

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Neuronal hyperpolarization-activated pacemaker channels drive neuropathic pain

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Adrienne E Dubin

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Alexander A Velumian

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Changlu Liu

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Chester Kuei

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Doo Hyun Lee

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Hong-Qing Guo

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Lin Luo

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Matthew P Butler

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Sandra R Chaplan

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Sean M Brown

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1169-1178

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scholarly article

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10.1523/JNEUROSCI.23-04-01169.2003

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2003-02-01T00:00:00Z

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12598605

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neuropathic pain

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6742242

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