Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice. - Wikidata - wikimedia - TriplyDB

Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice.

Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice.

http://www.wikidata.org/entity/Q34185758

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Interleukin-10: a pleiotropic regulator in pregnancy.Novel Toll-like receptor-4 antagonist (+)-naloxone protects mice from inflammation-induced preterm birth.Type I interferons regulate susceptibility to inflammation-induced preterm birth.Evidence for CB2 receptor involvement in LPS-induced reduction of cAMP intracellular levels in uterine explants from pregnant mice: pathophysiological implications.Targeting Cannabinoid Signaling in the Immune System: "High"-ly Exciting Questions, Possibilities, and Challenges.Endocannabinoid signaling in female reproductive events: a potential therapeutic target?Differential outcomes of TLR2 engagement in inflammation-induced preterm birth.

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Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice.

http://www.wikidata.org/entity/Q34185758

description

2014 nî lūn-bûn

@nan

2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice.

@ast

Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice.

@en

Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice.

@nl

type

label

Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice.

@ast

Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice.

@en

Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice.

@nl

prefLabel

Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice.

@ast

Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice.

@en

Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice.

@nl

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Cnr2 deficiency confers resistance to inflammation-induced preterm birth in mice

@en

P2093

Monica Cappelletti

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Senad Divanovic

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Sudhansu K Dey

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Xiaofei Sun

http://www.w3.org/2001/XMLSchema#string

Yingju Li

http://www.w3.org/2001/XMLSchema#string

P2860

Cannabinoid ligand-receptor signaling in the mouse uterus

Is lipid signaling through cannabinoid 2 receptors part of a protective system?

Cannabinoid-induced mesenteric vasodilation through an endothelial site distinct from CB1 or CB2 receptors

Negative regulation of Toll-like receptor 4 signaling by the Toll-like receptor homolog RP105

Increased mortality, hypoactivity, and hypoalgesia in cannabinoid CB1 receptor knockout mice

Structure of the rat interleukin 6 gene and its expression in macrophage-derived cells

Epidemiology and causes of preterm birth

Differential uterine expression of estrogen and progesterone receptors correlates with uterine preparation for implantation and decidualization in the mouse.

Il-10 is a central regulator of cyclooxygenase-2 expression and prostaglandin production.

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4006-4014

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scholarly article

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10.1210/EN.2014-1387

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10

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155

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Christopher L Karp

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2014-07-22T00:00:00Z

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264127492

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25051450

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4164934

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