The pentraxins, C-reactive protein and serum amyloid P component, are cleared and catabolized by hepatocytes in vivo.
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Serum amyloid P component prevents proteolysis of the amyloid fibrils of Alzheimer disease and systemic amyloidosisTargeting C-Reactive Protein in Inflammatory Disease by Preventing Conformational ChangesMolecular dissection of Alzheimer's disease neuropathology by depletion of serum amyloid P componentDietary modulation and structure prediction of rat mucosal pentraxin (Mptx) protein and loss of function in humansTransgenic human C-reactive protein is not proatherogenic in apolipoprotein E-deficient mice.Systemic catabolism of Alzheimer's Abeta40 and Abeta42.Association of kidney function with inflammatory and procoagulant markers in a diverse cohort: a cross-sectional analysis from the Multi-Ethnic Study of Atherosclerosis (MESA).Parasitic loads in tissues of mice infected with Trypanosoma cruzi and treated with AmBisome.Electronegative low-density lipoprotein increases C-reactive protein expression in vascular endothelial cells through the LOX-1 receptorA quantitative proteomic approach to prion disease biomarker research: delving into the glycoproteome.C-reactive protein and cardiovascular disease: new insights from an old molecule.Reductions in Plasma Cystatin C After Initiation of Antiretroviral Therapy Are Associated With Reductions in Inflammation: ACTG A5224s.Role of serum amyloid P component in bacterial infection: protection of the host or protection of the pathogenPentraxins and Fc receptorsLack of effect of a single injection of human C-reactive protein on murine lupus or nephrotoxic nephritis.C-reactive protein and the biology of disease.Modeling C-reactive protein kinetic profiles for use as a clinical prediction tool in patients with Staphylococcus aureus bacteremia.C-reactive protein and the acute phase reaction in geriatric patients.Advanced age influences the dynamic changes in circulating C-reactive protein following injury.Autoimmunity and glomerulonephritis in mice with targeted deletion of the serum amyloid P component gene: SAP deficiency or strain combination?Amyloid P component. A critical review
P2860
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P2860
The pentraxins, C-reactive protein and serum amyloid P component, are cleared and catabolized by hepatocytes in vivo.
description
1994 nî lūn-bûn
@nan
1994 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1994 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
name
The pentraxins, C-reactive pro ...... olized by hepatocytes in vivo.
@ast
The pentraxins, C-reactive pro ...... olized by hepatocytes in vivo.
@en
The pentraxins, C-reactive pro ...... olized by hepatocytes in vivo.
@nl
type
label
The pentraxins, C-reactive pro ...... olized by hepatocytes in vivo.
@ast
The pentraxins, C-reactive pro ...... olized by hepatocytes in vivo.
@en
The pentraxins, C-reactive pro ...... olized by hepatocytes in vivo.
@nl
prefLabel
The pentraxins, C-reactive pro ...... olized by hepatocytes in vivo.
@ast
The pentraxins, C-reactive pro ...... olized by hepatocytes in vivo.
@en
The pentraxins, C-reactive pro ...... olized by hepatocytes in vivo.
@nl
P2093
P2860
P356
P1476
The pentraxins, C-reactive pro ...... olized by hepatocytes in vivo.
@en
P2093
Hawkins PN
Hutchinson WL
P2860
P304
P356
10.1172/JCI117474
P407
P50
P577
1994-10-01T00:00:00Z