Hypertrophic stimuli induce transforming growth factor-beta 1 expression in rat ventricular myocytes.
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MicroRNA-34a regulates cardiac fibrosis after myocardial infarction by targeting Smad4MiR-155 Knockout in Fibroblasts Improves Cardiac Remodeling by Targeting Tumor Protein p53-Inducible Nuclear Protein 1Activation of TGF-beta1-TAK1-p38 MAPK pathway in spared cardiomyocytes is involved in left ventricular remodeling after myocardial infarction in ratsHistopathological study of time course changes in inter-renal aortic banding-induced left ventricular hypertrophy of mice.Plasma levels of transforming growth factor-beta1 reflect left ventricular remodeling in aortic stenosisIntramyocardial fibroblast myocyte communication.Inhibition of transforming growth factor-beta signaling induces left ventricular dilation and dysfunction in the pressure-overloaded heartRNA-binding protein RBM20 represses splicing to orchestrate cardiac pre-mRNA processing.Evolution and pathophysiology of chronic systolic heart failure.Deficiency of cardiomyocyte-specific microRNA-378 contributes to the development of cardiac fibrosis involving a transforming growth factor β (TGFβ1)-dependent paracrine mechanism.The MEKK1-JNK pathway plays a protective role in pressure overload but does not mediate cardiac hypertrophy.Delivery of an engineered HGF fragment in an extracellular matrix-derived hydrogel prevents negative LV remodeling post-myocardial infarctionTransforming growth factor-beta 1 hyperexpression in African-American hypertensives: A novel mediator of hypertension and/or target organ damage.Progenitor cell therapy in a porcine acute myocardial infarction model induces cardiac hypertrophy, mediated by paracrine secretion of cardiotrophic factors including TGFbeta1.The lipid peroxidation product 4-hydroxy-trans-2-nonenal causes protein synthesis in cardiac myocytes via activated mTORC1-p70S6K-RPS6 signaling.Excessive Dpp signaling induces cardial apoptosis through dTAK1 and dJNK during late embryogenesis of Drosophila.3-Hydroxy-3-methylglutaryl CoA reductase inhibitors up-regulate transforming growth factor-beta signaling in cultured heart cells via inhibition of geranylgeranylation of RhoA GTPase.Interleukin-1 beta modulates the growth and phenotype of neonatal rat cardiac myocytesBeneficial effects of long-term administration of an oral formulation of Angiotensin-(1-7) in infarcted rats.A cardiac myocyte vascular endothelial growth factor paracrine pathway is required to maintain cardiac function.Mechanical stretch up-regulates the B-type natriuretic peptide system in human cardiac fibroblasts: a possible defense against transforming growth factor-β mediated fibrosis.Myocyte-fibroblast communication in cardiac fibrosis and arrhythmias: Mechanisms and model systems.Fate of undifferentiated mouse embryonic stem cells within the rat heart: role of myocardial infarction and immune suppression.IL-9 Inhibits Viral Replication in Coxsackievirus B3-Induced Myocarditis.Critical role for IL-6 in hypertrophy and fibrosis in chronic cardiac allograft rejection.Thrombospondins in the heart: potential functions in cardiac remodeling.TGF-beta, IL-6, IL-17 and CTGF direct multiple pathologies of chronic cardiac allograft rejection.The cardiogenic niche as a fundamental building block of engineered myocardium.Regulatory RNAs and paracrine networks in the heart.Elevated Plasma Transforming Growth Factor β1 Levels Predict the Development of Hypertension in Normotensives: The 14-Year Follow-Up Study.TGF-beta1 mediates the hypertrophic cardiomyocyte growth induced by angiotensin II.Nitric oxide, atrial natriuretic peptide, and cyclic GMP inhibit the growth-promoting effects of norepinephrine in cardiac myocytes and fibroblasts.Kinetics of B-type natriuretic peptide plasma levels in patients with left-sided breast cancer treated with radiation therapy: Results after one-year follow-up.Deletion of phd2 in myeloid lineage attenuates hypertensive cardiovascular remodeling.Pivotal role of cardiomyocyte TGF-β signaling in the murine pathological response to sustained pressure overload.TGF-beta(1) and prepro-ANP mRNAs are differentially regulated in exercise-induced cardiac hypertrophy.Mechanisms of blood pressure variability-induced cardiac hypertrophy and dysfunction in mice with impaired baroreflex.Cardiac fibroblasts arrest at the G1/S restriction point in response to interleukin (IL)-1beta. Evidence for IL-1beta-induced hypophosphorylation of the retinoblastoma protein.Autocrine/paracrine determinants of strain-activated brain natriuretic peptide gene expression in cultured cardiac myocytes.Fibroblasts and the extracellular matrix in right ventricular disease.
P2860
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P2860
Hypertrophic stimuli induce transforming growth factor-beta 1 expression in rat ventricular myocytes.
description
1994 nî lūn-bûn
@nan
1994 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1994 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
name
Hypertrophic stimuli induce tr ...... n in rat ventricular myocytes.
@ast
Hypertrophic stimuli induce tr ...... n in rat ventricular myocytes.
@en
Hypertrophic stimuli induce tr ...... n in rat ventricular myocytes.
@nl
type
label
Hypertrophic stimuli induce tr ...... n in rat ventricular myocytes.
@ast
Hypertrophic stimuli induce tr ...... n in rat ventricular myocytes.
@en
Hypertrophic stimuli induce tr ...... n in rat ventricular myocytes.
@nl
prefLabel
Hypertrophic stimuli induce tr ...... n in rat ventricular myocytes.
@ast
Hypertrophic stimuli induce tr ...... n in rat ventricular myocytes.
@en
Hypertrophic stimuli induce tr ...... n in rat ventricular myocytes.
@nl
P2093
P2860
P356
P1476
Hypertrophic stimuli induce tr ...... n in rat ventricular myocytes.
@en
P2093
P2860
P304
P356
10.1172/JCI117485
P407
P577
1994-10-01T00:00:00Z