Insulin-like growth factor I treatment reduces demyelination and up-regulates gene expression of myelin-related proteins in experimental autoimmune encephalomyelitis.
about
Glial lineages and myelination in the central nervous systemHuman conditions of insulin-like growth factor-I (IGF-I) deficiencyGlial development: the crossroads of regeneration and repair in the CNSPromoting myelin repair and return of function in multiple sclerosisProNGF induces p75-mediated death of oligodendrocytes following spinal cord injuryProspect of brain-machine interface in motor disabilities: the future support for multiple sclerosis patient to improve quality of lifeAbsence of spontaneous central nervous system remyelination in class II-deficient mice infected with Theiler's virus.Radiation myelopathy: new perspective on an old problem.A novel microtubule-associated protein-2 expressed in oligodendrocytes in multiple sclerosis lesions.Insulin-like growth factor-I regulation of immune function: a potential therapeutic target in autoimmune diseases?Aerobic fitness is associated with gray matter volume and white matter integrity in multiple sclerosisInsulin-like growth factor-I-stimulated Akt phosphorylation and oligodendrocyte progenitor cell survival require cholesterol-enriched membranes.Surprising pleiotropy of nerve growth factor in the treatment of experimental autoimmune encephalomyelitis.Insulin-like growth factor-1 stimulates regulatory T cells and suppresses autoimmune disease.Investigation of sequential growth factor delivery during cuprizone challenge in mice aimed to enhance oligodendrogliogenesis and myelin repair.Targeting oligodendrocyte protection and remyelination in multiple sclerosis.Genetic programs and responses of neural stem/progenitor cells during demyelination: potential insights into repair mechanisms in multiple sclerosis.Insulin-like growth factor-I ameliorates demyelination induced by tumor necrosis factor-alpha in transgenic mice.Remyelinating and neuroprotective treatments in multiple sclerosis.Glia Disease and Repair-RemyelinationInsulin-like growth factor-1 fails to enhance central nervous system myelin repair during autoimmune demyelination.Redox regulation of cytokine-mediated inhibition of myelin gene expression in human primary oligodendrocytes.Neural stem cells in inflammatory CNS diseases: mechanisms and therapy.The neuregulin, glial growth factor 2, diminishes autoimmune demyelination and enhances remyelination in a chronic relapsing model for multiple sclerosis.Brain regeneration in physiology and pathology: the immune signature driving therapeutic plasticity of neural stem cellsIron supplementation dose for perinatal iron deficiency differentially alters the neurochemistry of the frontal cortex and hippocampus in adult rats.The therapeutic potential of gene transfer for the treatment of peripheral neuropathies.Experimental concepts for toxicity prevention and tissue restoration after central nervous system irradiation.Promoting oligodendrogenesis and myelin repair using the multiple sclerosis medication glatiramer acetate.Regulation of experimental autoimmune encephalomyelitis with insulin-like growth factor (IGF-1) and IGF-1/IGF-binding protein-3 complex (IGF-1/IGFBP3).Insulin-like growth factor 1 regulates developing brain glucose metabolism.Glial response during cuprizone-induced de- and remyelination in the CNS: lessons learned.Definition of a Family of Tissue-Protective Cytokines Using Functional Cluster Analysis: A Proof-of-Concept StudyExtracellular cues influencing oligodendrocyte differentiation and (re)myelinationThe role of growth factors as a therapeutic approach to demyelinating disease.A select combination of neurotrophins enhances neuroprotection and functional recovery following spinal cord injury.Depletion of Olig2 in oligodendrocyte progenitor cells infected by Theiler's murine encephalomyelitis virus.Local recruitment of remyelinating cells in the repair of demyelination in the central nervous system.Inflammation promotes survival and migration of the CG4 oligodendrocyte progenitors transplanted in the spinal cord of both inflammatory and demyelinated EAE rats.Insulin-like growth factor-I treatment reduces immune cell responses in acute non-demyelinative experimental autoimmune encephalomyelitis.
P2860
Q24544790-C0211F40-B12D-421D-8D5F-7878B74832DDQ26822806-A263E83E-72CE-4A2C-B80E-2E84703B09D0Q26823697-15065A87-B2F3-49FF-A579-3FC7E5598609Q26862803-9F1CF4FA-CAEA-4905-806F-F666B4C1AE7FQ28574864-A660AE5B-A581-43FF-BB65-CBDFB1537BD0Q28657453-D1D66526-11DE-4EEF-9B46-C542D017B0F2Q33727410-B084D616-19D8-4A5E-96EE-0E22B6224FC8Q33735227-7DA3736F-B690-4B07-83AB-66AB6F45AC05Q33882319-5DC3359A-D3AA-4B50-95FC-E2F120C61A7FQ33890792-F63867EE-08CC-4FDF-A938-3A61D45CB03FQ33907522-DE9A850C-F850-4F43-9B1E-B7A1E6C02F46Q33910877-1ADFD3AD-5D8F-4967-84EE-3DD08E3AC1ECQ33919193-D021A396-DCBC-4B46-B78D-F29850799DC1Q34542675-F314B25E-5E13-4C0C-AD69-7419F5F3B395Q34708578-06C0FF99-E645-467C-B66B-DFE2A3CB45CFQ34782075-92C08726-1E07-4C16-8965-422753F18021Q35201289-FBC12A6B-51FF-43BC-A1CC-4E38D66C7319Q35699248-83390013-9FDC-4488-A72A-6F876F5ECE31Q35753458-CD073282-40DF-48D6-9C4C-F3AA90C291C2Q35795587-3EC04843-18E3-4AAE-B0BF-7325D0A18472Q35829445-77BBB501-0027-4D45-8554-8B0EEF7FD3FBQ35956011-E120AABC-C298-4DBF-B979-B3C06E6B9470Q36166488-EA58973F-9AC8-4FED-A522-22B3E71A9B31Q36285550-72F4F8F1-9FE2-4DAB-B589-B93C0CDB7AD2Q36554467-99BE2D97-DF73-45D4-9B5C-5511CC1347DAQ36584697-8FD306A9-9CCD-4357-9C68-688819F4A4C4Q36762639-8D37C26D-CD7F-4B0F-AF91-3972382BE842Q36866199-46EF9817-4167-4B0B-B126-E25617B8D93DQ37378494-21FC8E19-A64A-48EB-9C6D-F9FF5A0899E0Q37381231-B94D9D34-C3BB-4610-8935-D6E26D5DAB30Q37438813-22FAEB9C-03DF-4542-BA64-45F2E675D68BQ37634375-818732A5-6759-43B7-BC51-9D65044EFF7CQ38286765-84129B82-9FE6-412F-A7B0-335A41B980C3Q38789322-C5EFF651-1F94-4EA6-8CA8-E7A22D4ECF4FQ38789326-482DD975-27F7-4B35-A47E-917F926BF928Q40036024-F7DBCA40-FFBD-4E45-97B8-5030700145B9Q40248517-3E6475F3-8898-4DEA-A792-3A6D0A59CC23Q41066214-A411100E-A765-4545-838A-E8A4887E982FQ41072947-900A6E5E-A436-4D46-81D3-A8B6354766D9Q41124612-16931BD8-E404-4EFD-B9BE-9D62BF2E8511
P2860
Insulin-like growth factor I treatment reduces demyelination and up-regulates gene expression of myelin-related proteins in experimental autoimmune encephalomyelitis.
description
1995 nî lūn-bûn
@nan
1995 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Insulin-like growth factor I t ...... autoimmune encephalomyelitis.
@ast
Insulin-like growth factor I t ...... autoimmune encephalomyelitis.
@en
Insulin-like growth factor I t ...... autoimmune encephalomyelitis.
@nl
type
label
Insulin-like growth factor I t ...... autoimmune encephalomyelitis.
@ast
Insulin-like growth factor I t ...... autoimmune encephalomyelitis.
@en
Insulin-like growth factor I t ...... autoimmune encephalomyelitis.
@nl
prefLabel
Insulin-like growth factor I t ...... autoimmune encephalomyelitis.
@ast
Insulin-like growth factor I t ...... autoimmune encephalomyelitis.
@en
Insulin-like growth factor I t ...... autoimmune encephalomyelitis.
@nl
P2093
P2860
P356
P1476
Insulin-like growth factor I t ...... autoimmune encephalomyelitis.
@en
P2093
P2860
P304
P356
10.1073/PNAS.92.13.6190
P407
P577
1995-06-01T00:00:00Z