Pseudomonas and neutrophil products modify transferrin and lactoferrin to create conditions that favor hydroxyl radical formation.
about
Pathogenic triad in COPD: oxidative stress, protease-antiprotease imbalance, and inflammationRole of oxidants in microbial pathophysiologySalivary Markers for Periodontal and General DiseasesWhat really happens in the neutrophil phagosome?Interaction of Candida albicans with adherent human peripheral blood mononuclear cells increases C. albicans biofilm formation and results in differential expression of pro- and anti-inflammatory cytokines.Enhanced Pseudomonas aeruginosa biofilm development mediated by human neutrophilsLaccase protects Cryptococcus neoformans from antifungal activity of alveolar macrophagesCharacterization of an endoprotease (PrpL) encoded by a PvdS-regulated gene in Pseudomonas aeruginosaProtease-cleaved iron-transferrin augments oxidant-mediated endothelial cell injury via hydroxyl radical formation.Clinical significance of microbial infection and adaptation in cystic fibrosis.Oxidants, antioxidants, and respiratory tract lining fluids.Binding and surface exposure characteristics of the gonococcal transferrin receptor are dependent on both transferrin-binding proteins.Antioxidants in cystic fibrosis. Conclusions from the CF antioxidant workshop, Bethesda, Maryland, November 11-12, 2003.Low-density lipoprotein modification by normal, myeloperoxidase-deficient and NADPH oxidase-deficient granulocytes and the impact of redox active transition metal ions.Protease cleavage of iron-transferrin augments pyocyanin-mediated endothelial cell injury via promotion of hydroxyl radical formation.Augmentation of oxidant injury to human pulmonary epithelial cells by the Pseudomonas aeruginosa siderophore pyochelin.Transferrin and lactoferrin undergo proteolytic cleavage in the Pseudomonas aeruginosa-infected lungs of patients with cystic fibrosis.Interaction of the Pseudomonas aeruginosa secretory products pyocyanin and pyochelin generates hydroxyl radical and causes synergistic damage to endothelial cells. Implications for Pseudomonas-associated tissue injury.Cleavage of human transferrin by Porphyromonas gingivalis gingipains promotes growth and formation of hydroxyl radicals.Increased expression of senescence markers in cystic fibrosis airwaysIncreased oxidative stress and depleted antioxidant capacity in chronic obstructive pulmonary disease: Searching for applications.
P2860
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P2860
Pseudomonas and neutrophil products modify transferrin and lactoferrin to create conditions that favor hydroxyl radical formation.
description
1991 nî lūn-bûn
@nan
1991 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1991 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1991年の論文
@ja
1991年学术文章
@wuu
1991年学术文章
@zh-cn
1991年学术文章
@zh-hans
1991年学术文章
@zh-my
1991年学术文章
@zh-sg
1991年學術文章
@yue
name
Pseudomonas and neutrophil pro ...... or hydroxyl radical formation.
@ast
Pseudomonas and neutrophil pro ...... or hydroxyl radical formation.
@en
Pseudomonas and neutrophil pro ...... or hydroxyl radical formation.
@nl
type
label
Pseudomonas and neutrophil pro ...... or hydroxyl radical formation.
@ast
Pseudomonas and neutrophil pro ...... or hydroxyl radical formation.
@en
Pseudomonas and neutrophil pro ...... or hydroxyl radical formation.
@nl
prefLabel
Pseudomonas and neutrophil pro ...... or hydroxyl radical formation.
@ast
Pseudomonas and neutrophil pro ...... or hydroxyl radical formation.
@en
Pseudomonas and neutrophil pro ...... or hydroxyl radical formation.
@nl
P2860
P356
P1476
Pseudomonas and neutrophil pro ...... or hydroxyl radical formation.
@en
P2093
B E Britigan
B L Edeker
P2860
P304
P356
10.1172/JCI115408
P407
P577
1991-10-01T00:00:00Z