PPAR{gamma} regulates hypoxia-induced Nox4 expression in human pulmonary artery smooth muscle cells through NF-{kappa}B.
about
Oxidases and peroxidases in cardiovascular and lung disease: new concepts in reactive oxygen species signalingEnhanced depolarization-induced pulmonary vasoconstriction following chronic hypoxia requires EGFR-dependent activation of NAD(P)H oxidase 2Brd4 inhibition attenuates unilateral ureteral obstruction-induced fibrosis by blocking TGF-β-mediated Nox4 expressionNADPH oxidases in vascular pathology.PPARγ attenuates hypoxia-induced hypertrophic transcriptional pathways in the heart.Decoding NADPH oxidase 4 expression in human tumors.Time-dependent PPARγ Modulation of HIF-1α Signaling in Hypoxic Pulmonary Artery Smooth Muscle Cells.The Role of NOX4 and TRX2 in Angiogenesis and Their Potential Cross-Talk.FSP27 promotes lipid droplet clustering and then fusion to regulate triglyceride accumulation.The Nox4 inhibitor GKT137831 attenuates hypoxia-induced pulmonary vascular cell proliferationPeroxisome proliferator-activated receptor α protects capillary pericytes in the retina.Increased oxidative stress in the nucleus caused by Nox4 mediates oxidation of HDAC4 and cardiac hypertrophy.The Nox1/4 Dual Inhibitor GKT137831 or Nox4 Knockdown Inhibits Angiotensin-II-Induced Adult Mouse Cardiac Fibroblast Proliferation and Migration. AT1 Physically Associates With Nox4.Is peroxisome proliferator-activated receptor gamma (PPARγ) a therapeutic target for the treatment of pulmonary hypertension?Circadian clock control of Nox4 and reactive oxygen species in the vasculature.Nox4: a hydrogen peroxide-generating oxygen sensor.Impact of diabetes in patients with pulmonary hypertensionHyperglycemia enhances IGF-I-stimulated Src activation via increasing Nox4-derived reactive oxygen species in a PKCζ-dependent manner in vascular smooth muscle cells.Inhibition of extracellular signal-regulated kinases ameliorates hypertension-induced renal vascular remodeling in rat modelsPPARγ Ligands Attenuate Hypoxia-Induced Proliferation in Human Pulmonary Artery Smooth Muscle Cells through Modulation of MicroRNA-21.Ethanol induces oxidative stress in alveolar macrophages via upregulation of NADPH oxidases.The NOX toolbox: validating the role of NADPH oxidases in physiology and disease.Peroxisome proliferator-activated receptor gamma (PPARγ) regulates thrombospondin-1 and Nox4 expression in hypoxia-induced human pulmonary artery smooth muscle cell proliferationRelaxation of human pulmonary arteries by PPARγ agonists.Rosiglitazone attenuates NF-κB-mediated Nox4 upregulation in hyperglycemia-activated endothelial cellsRole of nicotinamide adenine dinucleotide phosphate-reduced oxidase proteins in Pseudomonas aeruginosa-induced lung inflammation and permeability.Proline-rich tyrosine kinase 2 downregulates peroxisome proliferator-activated receptor gamma to promote hypoxia-induced pulmonary artery smooth muscle cell proliferation.Peroxisome Proliferator-Activated Receptor γ Regulates Chronic Alcohol-Induced Alveolar Macrophage Dysfunction.Reactive oxygen and nitrogen species in pulmonary hypertension.Chronic inhibition of PPAR-γ signaling induces endothelial dysfunction in the juvenile lambGlutathione attenuates ethanol-induced alveolar macrophage oxidative stress and dysfunction by downregulating NADPH oxidases.Mammalian target of rapamycin complex 2 (mTORC2) coordinates pulmonary artery smooth muscle cell metabolism, proliferation, and survival in pulmonary arterial hypertension.Rosiglitazone preserves pulmonary vascular function in lambs with increased pulmonary blood flow.Galectin-3 inhibition ameliorates hypoxia-induced pulmonary artery hypertension.Pediatric pulmonary arterial hypertension: current and emerging therapeutic options.Differential roles of NADPH oxidases in vascular physiology and pathophysiologyPhysiological roles of NOX/NADPH oxidase, the superoxide-generating enzyme.NADPH oxidases as therapeutic targets in ischemic stroke.Lung oxidative damage by hypoxiaFrom form to function: the role of Nox4 in the cardiovascular system.
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PPAR{gamma} regulates hypoxia-induced Nox4 expression in human pulmonary artery smooth muscle cells through NF-{kappa}B.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
PPAR{gamma} regulates hypoxia- ...... cle cells through NF-{kappa}B.
@ast
PPAR{gamma} regulates hypoxia- ...... cle cells through NF-{kappa}B.
@en
PPAR{gamma} regulates hypoxia- ...... cle cells through NF-{kappa}B.
@nl
type
label
PPAR{gamma} regulates hypoxia- ...... cle cells through NF-{kappa}B.
@ast
PPAR{gamma} regulates hypoxia- ...... cle cells through NF-{kappa}B.
@en
PPAR{gamma} regulates hypoxia- ...... cle cells through NF-{kappa}B.
@nl
prefLabel
PPAR{gamma} regulates hypoxia- ...... cle cells through NF-{kappa}B.
@ast
PPAR{gamma} regulates hypoxia- ...... cle cells through NF-{kappa}B.
@en
PPAR{gamma} regulates hypoxia- ...... cle cells through NF-{kappa}B.
@nl
P2093
P2860
P1476
PPAR{gamma} regulates hypoxia- ...... cle cells through NF-{kappa}B.
@en
P2093
C Michael Hart
Mark S Nanes
Tamara C Murphy
Xianghuai Lu
P2860
P304
P356
10.1152/AJPLUNG.00090.2010
P577
2010-07-09T00:00:00Z