Alterations in brain glucose utilization accompanying elevations in blood ethanol and acetate concentrations in the rat
about
Laboratory models available to study alcohol-induced organ damage and immune variations: choosing the appropriate model1H-NMR-based metabolomic profiling of CSF in early amyotrophic lateral sclerosisAlcohol decreases baseline brain glucose metabolism more in heavy drinkers than controls but has no effect on stimulation-induced metabolic increasesAcute alcohol intoxication decreases glucose metabolism but increases acetate uptake in the human brain.Succinate-to-fumarate ratio as a new metabolic marker to detect the presence of SDHB/D-related paraganglioma: initial experimental and ex vivo findings.A ketone ester diet increases brain malonyl-CoA and Uncoupling proteins 4 and 5 while decreasing food intake in the normal Wistar Rat.Addiction research centres and the nurturing of creativity: the National Institute on Alcohol Abuse and Alcoholism.Acetate-dependent mechanisms of inborn tolerance to ethanol.Ethanol, not detectably metabolized in brain, significantly reduces brain metabolism, probably via action at specific GABA(A) receptors and has measureable metabolic effects at very low concentrations.The sequenced rat brain transcriptome--its use in identifying networks predisposing alcohol consumptionMicrowave irradiation decreases ATP, increases free [Mg²⁺], and alters in vivo intracellular reactions in rat brain.Increased brain uptake and oxidation of acetate in heavy drinkers.Oxidation of ethanol in the rat brain and effects associated with chronic ethanol exposure.Focal thalamic degeneration from ethanol and thiamine deficiency is associated with neuroimmune gene induction, microglial activation, and lack of monocarboxylic acid transporters.Neuroimaging in Alcohol and Drug DependenceThe "first hit" toward alcohol reinforcement: role of ethanol metabolites.Metabolic products of [2-(13) C]ethanol in the rat brain after chronic ethanol exposure.Effects of a dietary ketone ester on hippocampal glycolytic and tricarboxylic acid cycle intermediates and amino acids in a 3xTgAD mouse model of Alzheimer's disease.
P2860
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P2860
Alterations in brain glucose utilization accompanying elevations in blood ethanol and acetate concentrations in the rat
description
2009 nî lūn-bûn
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2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
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2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
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name
Alterations in brain glucose u ...... tate concentrations in the rat
@ast
Alterations in brain glucose u ...... tate concentrations in the rat
@en
Alterations in brain glucose u ...... tate concentrations in the rat
@nl
type
label
Alterations in brain glucose u ...... tate concentrations in the rat
@ast
Alterations in brain glucose u ...... tate concentrations in the rat
@en
Alterations in brain glucose u ...... tate concentrations in the rat
@nl
prefLabel
Alterations in brain glucose u ...... tate concentrations in the rat
@ast
Alterations in brain glucose u ...... tate concentrations in the rat
@en
Alterations in brain glucose u ...... tate concentrations in the rat
@nl
P2093
P2860
P1476
Alterations in brain glucose u ...... tate concentrations in the rat
@en
P2093
Calvin Crutchfield
George Kunos
Michael T King
Richard L Veech
Robert J Pawlosky
Shireesh Srivastava
Ting-Kai Li
Yoshihiro Kashiwaya
P2860
P304
P356
10.1111/J.1530-0277.2009.01099.X
P577
2009-11-24T00:00:00Z