Amyloid precursor protein (APP) mediated regulation of ganglioside homeostasis linking Alzheimer's disease pathology with ganglioside metabolism.
about
Amyloid-beta Alzheimer targets - protein processing, lipid rafts, and amyloid-beta poresThe Impact of Vitamin E and Other Fat-Soluble Vitamins on Alzheimer´s DiseaseReporter pathway analysis from transcriptome data: Metabolite-centric versus Reaction-centric approach.Cross-talk of membrane lipids and Alzheimer-related proteins.Soluble Aβ oligomers are rapidly sequestered from brain ISF in vivo and bind GM1 ganglioside on cellular membranes.Lipid rafts and Alzheimer's disease: protein-lipid interactions and perturbation of signaling.Upregulated function of mitochondria-associated ER membranes in Alzheimer diseaseNeprilysin and Aβ Clearance: Impact of the APP Intracellular Domain in NEP Regulation and Implications in Alzheimer's DiseaseMetabolic Dysfunction of Astrocyte: An Initiating Factor in Beta-amyloid Pathology?APP Function and Lipids: A Bidirectional Link.Tocotrienol Affects Oxidative Stress, Cholesterol Homeostasis and the Amyloidogenic Pathway in Neuroblastoma Cells: Consequences for Alzheimer's DiseaseEndo-lysosomal and autophagic dysfunction: a driving factor in Alzheimer's disease?Omega-3 fatty acids, lipids and apoE lipidation in Alzheimer's disease: a rationale for multi-nutrient dementia prevention.Effect of Different Phospholipids on α-Secretase Activity in the Non-Amyloidogenic Pathway of Alzheimer's Disease.APP intracellular domain derived from amyloidogenic β- and γ-secretase cleavage regulates neprilysin expression.Expression of B4GALNT1, an essential glycosyltransferase for the synthesis of complex gangliosides, suppresses BACE1 degradation and modulates APP processingThe impact of cholesterol, DHA, and sphingolipids on Alzheimer's disease.Ischemia-modified albumin and advanced oxidation protein products as potential biomarkers of protein oxidation in Alzheimer's disease.Vitamin D and Its Analogues Decrease Amyloid-β (Aβ) Formation and Increase Aβ-Degradation.The Aβ-clearance protein transthyretin, like neprilysin, is epigenetically regulated by the amyloid precursor protein intracellular domain.Deletion of Specific Sphingolipids in Distinct Neurons Improves Spatial Memory in a Mouse Model of Alzheimer's Disease.
P2860
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P2860
Amyloid precursor protein (APP) mediated regulation of ganglioside homeostasis linking Alzheimer's disease pathology with ganglioside metabolism.
description
2012 nî lūn-bûn
@nan
2012 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի մարտին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Amyloid precursor protein (APP ...... y with ganglioside metabolism.
@ast
Amyloid precursor protein (APP ...... y with ganglioside metabolism.
@en
type
label
Amyloid precursor protein (APP ...... y with ganglioside metabolism.
@ast
Amyloid precursor protein (APP ...... y with ganglioside metabolism.
@en
prefLabel
Amyloid precursor protein (APP ...... y with ganglioside metabolism.
@ast
Amyloid precursor protein (APP ...... y with ganglioside metabolism.
@en
P2093
P2860
P1433
P1476
Amyloid precursor protein (APP ...... y with ganglioside metabolism.
@en
P2093
Benjamin Hundsdörfer
Eva G Zinser
Heike S Grimm
Lars Kaestner
Marcus O W Grimm
Peter Lipp
Sven Grösgen
Tatjana L Rothhaar
Thomas A Bayer
Ulrike Müller
P2860
P304
P356
10.1371/JOURNAL.PONE.0034095
P407
P577
2012-03-28T00:00:00Z