Genes are often sheltered from the global histone hyperacetylation induced by HDAC inhibitors.
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Histone Deacetylase (HDAC) Inhibitor Kinetic Rate Constants Correlate with Cellular Histone Acetylation but Not Transcription and Cell ViabilityHistone acetyltransferases and histone deacetylases in B- and T-cell development, physiology and malignancyHistone deacetylase inhibitors for cancer therapy: An evolutionarily ancient resistance response may explain their limited successHistone deacetylase inhibitors potentiate photodynamic therapy in colon cancer cells marked by chromatin-mediated epigenetic regulation of CDKN1A.Psammaplin a improves development and quality of somatic cell nuclear transfer mouse embryos.HDAC inhibitors as cognitive enhancers in fear, anxiety and trauma therapy: where do we stand?Epigenetic mechanisms underlying the role of brain-derived neurotrophic factor in depression and response to antidepressants.Cells adapt to the epigenomic disruption caused by histone deacetylase inhibitors through a coordinated, chromatin-mediated transcriptional response.HDAC inhibition attenuates cardiac hypertrophy by acetylation and deacetylation of target genes.MicroRNA-222 regulates MMP-13 via targeting HDAC-4 during osteoarthritis pathogenesis.Interindividual Variability in Stress Susceptibility: A Role for Epigenetic Mechanisms in PTSD.Histone deacetylase inhibitors facilitate partner preference formation in female prairie voles.Epigenetic origin of adaptive phenotypic variants in the human blood fluke Schistosoma mansoniSuberoylanilide hydroxamic acid induces limited changes in the transcriptome of primary CD4(+) T cells.The histone deacetylase inhibitor sodium valproate causes limited transcriptional change in mouse embryonic stem cells but selectively overrides Polycomb-mediated Hoxb silencingDeacetylase-independent function of HDAC3 in transcription and metabolism requires nuclear receptor corepressor.Epigenetic modulation with histone deacetylase inhibitors in combination with immunotherapy.Cell Polarization and Epigenetic Status Shape the Heterogeneous Response to Type III Interferons in Intestinal Epithelial Cells.Histone deacetylase inhibitors as cancer therapeutics.HDAC inhibitors attenuate the development of hypersensitivity in models of neuropathic pain.Suppression of Spleen Tyrosine Kinase (Syk) by Histone Deacetylation Promotes, Whereas BAY61-3606, a Synthetic Syk Inhibitor Abrogates Colonocyte Apoptosis by ERK Activation.
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P2860
Genes are often sheltered from the global histone hyperacetylation induced by HDAC inhibitors.
description
2012 nî lūn-bûn
@nan
2012 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի մարտին հրատարակված գիտական հոդված
@hy
2012年の論文
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2012年論文
@yue
2012年論文
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2012年論文
@zh-hk
2012年論文
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2012年論文
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2012年论文
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name
Genes are often sheltered from ...... on induced by HDAC inhibitors.
@ast
Genes are often sheltered from ...... on induced by HDAC inhibitors.
@en
Genes are often sheltered from ...... on induced by HDAC inhibitors.
@nl
type
label
Genes are often sheltered from ...... on induced by HDAC inhibitors.
@ast
Genes are often sheltered from ...... on induced by HDAC inhibitors.
@en
Genes are often sheltered from ...... on induced by HDAC inhibitors.
@nl
prefLabel
Genes are often sheltered from ...... on induced by HDAC inhibitors.
@ast
Genes are often sheltered from ...... on induced by HDAC inhibitors.
@en
Genes are often sheltered from ...... on induced by HDAC inhibitors.
@nl
P2093
P2860
P1433
P1476
Genes are often sheltered from ...... on induced by HDAC inhibitors.
@en
P2093
Bryan M Turner
Karl P Nightingale
Laura P O'Neill
Vibhor Gupta
P2860
P304
P356
10.1371/JOURNAL.PONE.0033453
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P50
P577
2012-03-30T00:00:00Z