Deletion of a single β-catenin allele in osteocytes abolishes the bone anabolic response to loading - Wikidata - wikimedia - TriplyDB

Deletion of a single β-catenin allele in osteocytes abolishes the bone anabolic response to loading

Deletion of a single β-catenin allele in osteocytes abolishes the bone anabolic response to loading

http://www.wikidata.org/entity/Q34228323

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New Insights into Wnt-Lrp5/6-β-Catenin Signaling in Mechanotransduction Physiological mechanisms and therapeutic potential of bone mechanosensing.Osteocytes mediate the anabolic actions of canonical Wnt/β-catenin signaling in bone.Activation of Wnt Signaling by Mechanical Loading Is Impaired in the Bone of Old Mice Sex steroid actions in male bone Activation of β-catenin signalling leads to temporomandibular joint defects.Planar cell polarity aligns osteoblast division in response to substrate strain HIF-1α regulates bone formation after osteogenic mechanical loading.Cx43 and mechanotransduction in bone.Impaired bone homeostasis in amyotrophic lateral sclerosis mice with muscle atrophy.In vivo mechanical loading rapidly activates β-catenin signaling in osteocytes through a prostaglandin mediated mechanism.Follistatin-like 3 is a mediator of exercise-driven bone formation and strengthening.Beta-Catenin Haplo Insufficient Male Mice Do Not Lose Bone in Response to Hindlimb Unloading.Knee loading inhibits osteoclast lineage in a mouse model of osteoarthritis Postnatal β-catenin deletion from Dmp1-expressing osteocytes/osteoblasts reduces structural adaptation to loading, but not periosteal load-induced bone formation Pathogenic LRRK2 variants are gain-of-function mutations that enhance LRRK2-mediated repression of β-catenin signaling Estrogen receptor α in osteocytes regulates trabecular bone formation in female mice.Dmp1 Promoter-Driven Diphtheria Toxin Receptor Transgene Expression Directs Unforeseen Effects in Multiple Tissues Evidence of the Role of R-Spondin 1 and Its Receptor Lgr4 in the Transmission of Mechanical Stimuli to Biological Signals for Bone Formation.Muscle-Bone Crosstalk in Amyotrophic Lateral Sclerosis.Bone Homeostasis and Repair: Forced Into Shape.The role of the wnt/β-catenin signaling pathway in formation and maintenance of bone and teeth.Comparison of strain measurement in the mouse forearm using subject-specific finite element models, strain gaging, and digital image correlation.Strain uses gap junctions to reverse stimulation of osteoblast proliferation by osteocytes.Exploiting the WNT Signaling Pathway for Clinical Purposes.Adverse Effects of Osteocytic Constitutive Activation of ß-Catenin on Bone Strength and Bone Growth.Degeneration of the osteocyte network in the C57BL/6 mouse model of aging.Recombinant sclerostin antagonises effects of ex vivo mechanical loading in trabecular bone and increases osteocyte lacunar size.ALS-associated mutation SOD1G93A leads to abnormal mitochondrial dynamics in osteocytes.Different bone remodeling levels of trabecular and cortical bone in response to changes in Wnt/β-catenin signaling in mice.Transient peak-strain matching partially recovers the age-impaired mechanoadaptive cortical bone response.Sost Deficiency does not Alter Bone's Lacunar or Vascular Porosity in Mice.

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Deletion of a single β-catenin allele in osteocytes abolishes the bone anabolic response to loading

http://www.wikidata.org/entity/Q34228323

description

2014 nî lūn-bûn

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2014 թուականի Մարտին հրատարակուած գիտական յօդուած

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2014 թվականի մարտին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Deletion of a single β-catenin ...... e anabolic response to loading

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Deletion of a single β-catenin ...... e anabolic response to loading

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Deletion of a single β-catenin ...... e anabolic response to loading

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Deletion of a single β-catenin ...... e anabolic response to loading

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Deletion of a single β-catenin ...... e anabolic response to loading

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Deletion of a single β-catenin ...... e anabolic response to loading

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prefLabel

Deletion of a single β-catenin ...... e anabolic response to loading

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Deletion of a single β-catenin ...... e anabolic response to loading

@en

Deletion of a single β-catenin ...... e anabolic response to loading

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P1433

Journal of Bone and Mineral Research

P1476

Deletion of a single β-catenin ...... e anabolic response to loading

@en

P2093

Amber Rath Stern

http://www.w3.org/2001/XMLSchema#string

Hong Zhao

http://www.w3.org/2001/XMLSchema#string

Lynda F Bonewald

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Mark Dallas

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Mark L Johnson

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Nuria Lara

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Ying Liu

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P2860

NIH Image to ImageJ: 25 years of image analysis

A mutation in the LDL receptor-related protein 5 gene results in the autosomal dominant high-bone-mass trait

Wnt signaling and cancer

The Wnt signaling pathway in development and disease

High bone mass in mice expressing a mutant LRP5 gene

LDL receptor-related protein 5 (LRP5) affects bone accrual and eye development

High bone density due to a mutation in LDL-receptor-related protein 5

The Wnt co-receptor LRP5 is essential for skeletal mechanotransduction but not for the anabolic bone response to parathyroid hormone treatment

Matrix-embedded cells control osteoclast formation

Evidence for osteocyte regulation of bone homeostasis through RANKL expression

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705-715

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scholarly article

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10.1002/JBMR.2064

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3

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29

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Behzad Javaheri

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2014-03-01T00:00:00Z

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23929793

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4171742

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