A multi-cancer mesenchymal transition gene expression signature is associated with prolonged time to recurrence in glioblastoma - Wikidata - wikimedia - TriplyDB

A multi-cancer mesenchymal transition gene expression signature is associated with prolonged time to recurrence in glioblastoma

A multi-cancer mesenchymal transition gene expression signature is associated with prolonged time to recurrence in glioblastoma

http://www.wikidata.org/entity/Q34228481

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Molecular biomarkers of cancer stem/progenitor cells associated with progression, metastases, and treatment resistance of aggressive cancers Key nodes of a microRNA network associated with the integrated mesenchymal subtype of high-grade serous ovarian cancer High-throughput 3D screening reveals differences in drug sensitivities between culture models of JIMT1 breast cancer cells Overcoming therapeutic resistance in glioblastoma: the way forward The Huntington disease protein accelerates breast tumour development and metastasis through ErbB2/HER2 signalling.Biphasic Dependence of Glioma Survival and Cell Migration on CD44 Expression Level Cancer stem cell plasticity and tumor hierarchy.Biomolecular events in cancer revealed by attractor metagenes.Dynamic transcription factor networks in epithelial-mesenchymal transition in breast cancer models.Correlation analysis connects cancer subtypes.Cadherin-11 regulates motility in normal cortical neural precursors and glioblastoma Acetate supplementation induces growth arrest of NG2/PDGFRα-positive oligodendroglioma-derived tumor-initiating cells Personalized medicine for gliomas An integrated mRNA and microRNA expression signature for glioblastoma multiforme prognosis.Whole genome sequencing of glioblastoma multiforme identifies multiple structural variations involved in EGFR activation KITENIN promotes glioma invasiveness and progression, associated with the induction of EMT and stemness markers.Coexpression analysis of CD133 and CD44 identifies proneural and mesenchymal subtypes of glioblastoma multiforme.Loss of ATM accelerates pancreatic cancer formation and epithelial-mesenchymal transition.Computational analysis of the mesenchymal signature landscape in gliomas.RTVP-1 promotes mesenchymal transformation of glioma via a STAT-3/IL-6-dependent positive feedback loop.Ginsenoside Rg3 inhibits epithelial-mesenchymal transition (EMT) and invasion of lung cancer by down-regulating FUT4.Genotypes of cancer stem cells characterized by epithelial-to-mesenchymal transition and proliferation related functions.JUN dependency in distinct early and late BRAF inhibition adaptation states of melanoma Altered gene products involved in the malignant reprogramming of cancer stem/progenitor cells and multitargeted therapies.Identifying causal networks linking cancer processes and anti-tumor immunity using Bayesian network inference and metagene constructs.Recent advances and prospects in the isolation by size of epithelial tumor cells (ISET) methodology.Cancer stem cell contribution to glioblastoma invasiveness.Extra-CNS metastasis from glioblastoma: a rare clinical entity.Mithramycin inhibits epithelial-to-mesenchymal transition and invasion by downregulating SP1 and SNAI1 in salivary adenoid cystic carcinoma.Epithelial-mesenchymal transition in glioblastoma progression.A COL11A1-correlated pan-cancer gene signature of activated fibroblasts for the prioritization of therapeutic targets.The Epithelial-to-Mesenchymal Transition-Like Process in Glioblastoma: An Updated Systematic Review and In Silico Investigation.The KDM1A histone demethylase is a promising new target for the epigenetic therapy of medulloblastoma.Expression/activation of α5β1 integrin is linked to the β-catenin signaling pathway to drive migration in glioma cells.Supra- and infratentorial pediatric ependymomas differ significantly in NeuN, p75 and GFAP expression.Aberrantly activated Cox-2 and Wnt signaling interact to maintain cancer stem cells in glioblastoma.Epigenetic Regulation of ZBTB18 Promotes Glioblastoma Progression.Silencing of BAG3 inhibits the epithelial-mesenchymal transition in human cervical cancer.Developmentally regulated signaling pathways in glioma invasion.Analysis of expression and prognostic significance of vimentin and the response to temozolomide in glioma patients.

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A multi-cancer mesenchymal transition gene expression signature is associated with prolonged time to recurrence in glioblastoma

http://www.wikidata.org/entity/Q34228481

description

2012 nî lūn-bûn

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2012 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2012 թվականի ապրիլին հրատարակված գիտական հոդված

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2012年論文

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2012年論文

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A multi-cancer mesenchymal tra ...... to recurrence in glioblastoma

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A multi-cancer mesenchymal tra ...... to recurrence in glioblastoma

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A multi-cancer mesenchymal tra ...... to recurrence in glioblastoma

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A multi-cancer mesenchymal tra ...... to recurrence in glioblastoma

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A multi-cancer mesenchymal tra ...... to recurrence in glioblastoma

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A multi-cancer mesenchymal tra ...... to recurrence in glioblastoma

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A multi-cancer mesenchymal tra ...... to recurrence in glioblastoma

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A multi-cancer mesenchymal tra ...... to recurrence in glioblastoma

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A multi-cancer mesenchymal tra ...... to recurrence in glioblastoma

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A multi-cancer mesenchymal tra ...... to recurrence in glioblastoma

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Darrell J Yamashiro

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Dimitris Anastassiou

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Jessica J Kandel

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Peter Canoll

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Wei-Yi Cheng

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P2860

Gene set enrichment analysis: a knowledge-based approach for interpreting genome-wide expression profiles.

The epithelial-mesenchymal transition generates cells with properties of stem cells

Integrated genomic analysis identifies clinically relevant subtypes of glioblastoma characterized by abnormalities in PDGFRA, IDH1, EGFR, and NF1

Epithelial-mesenchymal transitions in tumour progression

Epithelial-mesenchymal transitions in development and disease

TGF-β Receptor Inhibitors Target the CD44(high)/Id1(high) Glioma-Initiating Cell Population in Human Glioblastoma

Snail, Zeb and bHLH factors in tumour progression: an alliance against the epithelial phenotype?

EMT, cancer stem cells and drug resistance: an emerging axis of evil in the war on cancer

Molecular subclasses of high-grade glioma predict prognosis, delineate a pattern of disease progression, and resemble stages in neurogenesis

Generation of breast cancer stem cells through epithelial-mesenchymal transition

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