Oral-resident natural Th17 cells and γδ T cells control opportunistic Candida albicans infections. - Wikidata - wikimedia - TriplyDB

Oral-resident natural Th17 cells and γδ T cells control opportunistic Candida albicans infections.

Oral-resident natural Th17 cells and γδ T cells control opportunistic Candida albicans infections.

http://www.wikidata.org/entity/Q34230506

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Mucosal Interactions between Genetics, Diet, and Microbiome in Inflammatory Bowel Disease Mechanistic Insights into the Role of C-Type Lectin Receptor/CARD9 Signaling in Human Antifungal Immunity γδ T Cells and NK Cells - Distinct Pathogenic Roles as Innate-Like Immune Cells in CNS Autoimmunity Interleukin 17-Mediated Host Defense against Candida albicans IL-17-Mediated Immunity to the Opportunistic Fungal Pathogen Candida albicans Oropharyngeal Candidiasis in HIV Infection: Analysis of Impaired Mucosal Immune Response to Candida albicans in Mice Expressing the HIV-1 Transgene The Kallikrein-Kinin System: A Novel Mediator of IL-17-Driven Anti-Candida Immunity in the Kidney T-Bet independent development of IFNγ secreting natural T helper 1 cell population in the absence of Itk.Immunity against fungi.CX3CR1 is dispensable for control of mucosal Candida albicans infections in mice and humans.Signaling through IL-17C/IL-17RE is dispensable for immunity to systemic, oral and cutaneous candidiasis.C/EBPβ Promotes Immunity to Oral Candidiasis through Regulation of β-Defensins.Antigen-Specific Th17 Cells Are Primed by Distinct and Complementary Dendritic Cell Subsets in Oropharyngeal Candidiasis Immune Interactions with Pathogenic and Commensal Fungi: A Two-Way Street A Candida albicans Strain Expressing Mammalian Interleukin-17A Results in Early Control of Fungal Growth during Disseminated Infection.Sequential Dysfunction and Progressive Depletion of Candida albicans-Specific CD4 T Cell Response in HIV-1 Infection.Natural Th17 cells are critically regulated by functional medullary thymic microenvironments.Neutrophils Do Not Express IL-17A in the Context of Acute Oropharyngeal Candidiasis.Adaptive immune responses to Candida albicans infection.Nociceptive Sensory Fibers Drive Interleukin-23 Production from CD301b+ Dermal Dendritic Cells and Drive Protective Cutaneous Immunity Inhibiting the immunoproteasome exacerbates the pathogenesis of systemic Candida albicans infection in mice.Skin Immunity to Candida albicans.Antibody blockade of IL-17 family cytokines in immunity to acute murine oral mucosal candidiasis.Interleukin-23 Secreted by Activated Macrophages Drives γδT Cell Production of Interleukin-17 to Aggravate Secondary Injury After Intracerebral Hemorrhage.Brothers in arms: Th17 and Treg responses in Candida albicans immunity The role of IL-17 in CNS diseases.The unusual suspects--innate lymphoid cells as novel therapeutic targets in IBD.Influence of nutrient-derived metabolites on lymphocyte immunity.Beyond Candida albicans: Mechanisms of immunity to non-albicans Candida species Immune defence against Candida fungal infections.IL-1β and IL-23 Promote Extrathymic Commitment of CD27(+)CD122(-) γδ T Cells to γδT17 Cells Ncf1 polymorphism reveals oxidative regulation of autoimmune chronic inflammation.Could the PI3K canonical pathway be a common link between chronic inflammatory conditions and oral carcinogenesis?Antifungal Immunological Defenses in Newborns.On-going Mechanical Damage from Mastication Drives Homeostatic Th17 Cell Responses at the Oral Barrier The contrasting roles of Th17 immunity in human health and disease.IL-17+ γδ T cells as kick-starters of inflammation.Interleukin 17 is a chief orchestrator of immunity.The dichotomous nature of T helper 17 cells.The Goldilocks model of immune symbiosis with Mycobacteria and Candida colonizers.

P2860

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P2860

Oral-resident natural Th17 cells and γδ T cells control opportunistic Candida albicans infections.

http://www.wikidata.org/entity/Q34230506

description

2014 nî lūn-bûn

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2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Oral-resident natural Th17 cel ...... c Candida albicans infections.

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Oral-resident natural Th17 cel ...... c Candida albicans infections.

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Oral-resident natural Th17 cel ...... c Candida albicans infections.

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Oral-resident natural Th17 cel ...... c Candida albicans infections.

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Oral-resident natural Th17 cel ...... c Candida albicans infections.

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Oral-resident natural Th17 cel ...... c Candida albicans infections.

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Oral-resident natural Th17 cel ...... c Candida albicans infections.

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Oral-resident natural Th17 cel ...... c Candida albicans infections.

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Journal of Experimental Medicine

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Oral-resident natural Th17 cel ...... c Candida albicans infections.

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P2093

Abhishek V Garg

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Alanna C Peterson

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Anna J Mamo

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Anna R Huppler

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David Artis

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Gregory A Gibson

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Heather R Conti

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Lucas Brane

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Mandy J McGeachy

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Michelle R Simpson-Abelson

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P2860

Evolution of pathogenicity and sexual reproduction in eight Candida genomes

Selective regulatory function of Socs3 in the formation of IL-17-secreting T cells

The interleukin 23 receptor is essential for the terminal differentiation of interleukin 17-producing effector T helper cells in vivo

Fate mapping of IL-17-producing T cells in inflammatory responses

Lymphoid tissue inducer-like cells are an innate source of IL-17 and IL-22

Mucocutaneous candidiasis: the IL-17 pathway and implications for targeted immunotherapy

STAT3 mutations in the hyper-IgE syndrome

STAT3 regulates cytokine-mediated generation of inflammatory helper T cells

IL-17RC is required for immune signaling via an extended SEF/IL-17R signaling domain in the cytoplasmic tail

Innate lymphoid cells--a proposal for uniform nomenclature

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2075-2084

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10.1084/JEM.20130877

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25200028

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