Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death - Wikidata - wikimedia - TriplyDB

Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death

Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death

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Neurotrophic factor small-molecule mimetics mediated neuroregeneration and synaptic repair: emerging therapeutic modality for Alzheimer's disease Multiple signaling factors and drugs alleviate neuronal death induced by expression of human and zebrafish tau proteins in vivo Astrocytic and microglial nicotinic acetylcholine receptors: an overlooked issue in Alzheimer's disease.Exercise-Mediated Increase in Nigral Tyrosine Hydroxylase Is Accompanied by Increased Nigral GFR-α1 and EAAC1 Expression in Aging Rats Potential therapeutic action of natural products from traditional Chinese medicine on Alzheimer's disease animal models targeting neurotrophic factors.Inhibitory Effect of Memantine on Streptozotocin-Induced Insulin Receptor Dysfunction, Neuroinflammation, Amyloidogenesis, and Neurotrophic Factor Decline in Astrocytes.Growth factor therapy sequesters inflammation in affording neuroprotection in cerebrovascular diseases.The Non-Survival Effects of Glial Cell Line-Derived Neurotrophic Factor on Neural Cells.Proteomic Dissection of Nanotopography-Sensitive Mechanotransductive Signaling Hubs that Foster Neuronal Differentiation in PC12 Cells.GDNF/GFRα1 Complex Abrogates Self-Renewing Activity of Cortical Neural Precursors Inducing Their Differentiation.Sensory and Motor Behavior Evidences Supporting the Usefulness of Conditioned Medium from Dental Pulp-Derived Stem Cells in Spinal Cord Injury in Rats

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P2860

Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death

http://www.wikidata.org/entity/Q34233456

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2014 nî lūn-bûn

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2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death

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Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death

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Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death

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type

label

Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death

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Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death

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Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death

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prefLabel

Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death

@ast

Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death

@en

Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death

@nl

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JNEUROSCI.2582-13.2014

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Journal of Neuroscience

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Deficiency of GDNF Receptor GFRα1 in Alzheimer's Neurons Results in Neuronal Death

@en

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Bai Lu

http://www.w3.org/2001/XMLSchema#string

Kristina Lindholm

http://www.w3.org/2001/XMLSchema#string

Li-Bang Yang

http://www.w3.org/2001/XMLSchema#string

Ping He

http://www.w3.org/2001/XMLSchema#string

Rena Li

http://www.w3.org/2001/XMLSchema#string

Yong Shen

http://www.w3.org/2001/XMLSchema#string

Yoshihiro Konishi

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P2860

Artemin, a novel member of the GDNF ligand family, supports peripheral and central neurons and signals through the GFRalpha3-RET receptor complex

Alzheimer mechanisms and therapeutic strategies

Neuropathological stageing of Alzheimer-related changes

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

Structure of artemin complexed with its receptor GFRalpha3: convergent recognition of glial cell line-derived neurotrophic factors

Ret is a multifunctional coreceptor that integrates diffusible- and contact-axon guidance signals

The Consortium to Establish a Registry for Alzheimer's Disease (CERAD). Part II. Standardization of the neuropathologic assessment of Alzheimer's disease

A quantitative method for analysis of in vitro neurite outgrowth.

Efficient generation of recombinant adenoviruses using adenovirus DNA-terminal protein complex and a cosmid bearing the full-length virus genome.

Characterization of a multicomponent receptor for GDNF.

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13127-13138

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scholarly article

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10.1523/JNEUROSCI.2582-13.2014

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39

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34

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2014-09-01T00:00:00Z

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266151561

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25253858

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Alzheimer's disease

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4172805

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