Hepatitis C virus infection of T cells inhibits proliferation and enhances fas-mediated apoptosis by down-regulating the expression of CD44 splicing variant 6.
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Direct effects of hepatitis C virus on the lymphoid cellsInterleukin-27 is differentially associated with HIV viral load and CD4+ T cell counts in therapy-naïve HIV-mono-infected and HIV/HCV-co-infected ChineseHCV infection enhances Th17 commitment, which could affect the pathogenesis of autoimmune diseases.Hepatitis C virus protects human B lymphocytes from Fas-mediated apoptosis via E2-CD81 engagementLymphotropic HCV strain can infect human primary naïve CD4+ cells and affect their proliferation and IFN-γ secretion activity.Complementary role of HCV and HIV in T-cell activation and exhaustion in HIV/HCV coinfection.1(OH) vitamin D3 supplementation improves the sensitivity of the immune-response during Peg-IFN/RBV therapy in chronic hepatitis C patients-case controlled trial.Dysfunction of Immune Systems and Host Genetic Factors in Hepatitis C Virus Infection with Persistent Normal ALT.Activins and Follistatin in Chronic Hepatitis C and Its Treatment with Pegylated-Interferon-α Based Therapy.Hepatitis C virus has a genetically determined lymphotropism through co-receptor B7.2.Virologic and immunologic aspects of HIV-hepatitis C virus coinfection.Modulation of IL-2 expression after uptake of hepatitis C virus non-enveloped capsid-like particles: the role of p38 kinase.Biological significance of HCV in various kinds of lymphoid cells.PD-L1+MDSCs are increased in HCC patients and induced by soluble factor in the tumor microenvironment.Sequential immunological analysis of HBV/HCV co-infected patients during Peg-IFN/RBV therapy.Eradication of hepatitis C virus could improve immunological status and pyoderma gangrenosum-like lesions.Authentic Patient-Derived Hepatitis C Virus Infects and Productively Replicates in Primary CD4+ and CD8+ T Lymphocytes In Vitro.Rapid reduction of hepatitis C virus-Core protein in the peripheral blood improve the immunological response in chronic hepatitis C patients.
P2860
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P2860
Hepatitis C virus infection of T cells inhibits proliferation and enhances fas-mediated apoptosis by down-regulating the expression of CD44 splicing variant 6.
description
2009 nî lūn-bûn
@nan
2009 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի մարտին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Hepatitis C virus infection of ...... on of CD44 splicing variant 6.
@ast
Hepatitis C virus infection of ...... on of CD44 splicing variant 6.
@en
Hepatitis C virus infection of ...... on of CD44 splicing variant 6.
@nl
type
label
Hepatitis C virus infection of ...... on of CD44 splicing variant 6.
@ast
Hepatitis C virus infection of ...... on of CD44 splicing variant 6.
@en
Hepatitis C virus infection of ...... on of CD44 splicing variant 6.
@nl
prefLabel
Hepatitis C virus infection of ...... on of CD44 splicing variant 6.
@ast
Hepatitis C virus infection of ...... on of CD44 splicing variant 6.
@en
Hepatitis C virus infection of ...... on of CD44 splicing variant 6.
@nl
P2093
P2860
P356
P1476
Hepatitis C virus infection of ...... on of CD44 splicing variant 6.
@en
P2093
Helene Minyi Liu
Keigo Machida
Koju Kobayashi
Takaji Wakita
Tooru Shimosegawa
Yasuteru Kondo
Yoshiyuki Ueno
P2860
P304
P356
10.1086/596739
P407
P577
2009-03-01T00:00:00Z