Deep RNA sequencing reveals novel cardiac transcriptomic signatures for physiological and pathological hypertrophy
about
Chemokines and Heart Disease: A Network Connecting Cardiovascular Biology to Immune and Autonomic Nervous SystemsOverview of high throughput sequencing technologies to elucidate molecular pathways in cardiovascular diseasesRegulation of chromatin structure in the cardiovascular systemHistone methylations in heart development, congenital and adult heart diseasesTranscriptomic Profile of Whole Blood Cells from Elderly Subjects Fed Probiotic Bacteria Lactobacillus rhamnosus GG ATCC 53103 (LGG) in a Phase I Open Label StudyIntegrated analysis of drug-induced gene expression profiles predicts novel hERG inhibitorsHerbal adaptogens combined with protein fractions from bovine colostrum and hen egg yolk reduce liver TNF-α expression and protein carbonylation in Western diet feeding in ratsIdentification of tissue-enriched novel transcripts and novel exons in mice.Functional networks of nucleocytoplasmic transport-related genes differentiate ischemic and dilated cardiomyopathies. A new therapeutic opportunityBET bromodomains mediate transcriptional pause release in heart failureNucleus accumbens neuronal maturation differences in young rats bred for low versus high voluntary running behaviour.Global microRNA profiling of the mouse ventricles during development of severe hypertrophic cardiomyopathy and heart failure.Gene expression analyses implicate an alternative splicing program in regulating contractile gene expression and serum response factor activity in mice.The switching role of β-adrenergic receptor signalling in cell survival or death decision of cardiomyocytes.Transcriptomic analysis of hepatic responses to testosterone deficiency in miniature pigs fed a high-cholesterol diet.Pik3ip1 modulates cardiac hypertrophy by inhibiting PI3K pathwayThe C2 Domain and Altered ATP-Binding Loop Phosphorylation at Ser³⁵⁹ Mediate the Redox-Dependent Increase in Protein Kinase C-δ ActivityRight ventricular long noncoding RNA expression in human heart failureRapid Alterations in Perirenal Adipose Tissue Transcriptomic Networks with Cessation of Voluntary Running.Long Non-Coding RNA Malat-1 Is Dispensable during Pressure Overload-Induced Cardiac Remodeling and Failure in MiceRNA-seq analysis of glycosylation related gene expression in STZ-induced diabetic rat kidney inner medullaCardiovascular transcriptomics and epigenomics using next-generation sequencing: challenges, progress, and opportunities.RNA binding proteins in the regulation of heart development.Pathological Ace2-to-Ace enzyme switch in the stressed heart is transcriptionally controlled by the endothelial Brg1-FoxM1 complex.Pressure-overload cardiac hypertrophy is associated with distinct alternative splicing due to altered expression of splicing factors.Up-regulated FHL1 expression maybe involved in the prognosis of Hirschsprung's disease.5'RNA-Seq identifies Fhl1 as a genetic modifier in cardiomyopathy.Missing links in cardiology: long non-coding RNAs enter the arena.Insights into RNA transcriptome profiling of cardiac tissue in obesity and hypertension conditions.Murine Myocardial Transcriptome Analysis Reveals a Critical Role of COPS8 in the Gene Expression of Cullin-RING Ligase Substrate Receptors and Redox and Vesicle Trafficking Pathways.Proteomics Research in Cardiovascular Medicine and Biomarker Discovery.Conceptual Foundations of Systems Biology Explaining Complex Cardiac Diseases.BET bromodomain inhibition suppresses innate inflammatory and profibrotic transcriptional networks in heart failure.Nrf2 deficiency prevents reductive stress-induced hypertrophic cardiomyopathy.Impact of external pneumatic compression target inflation pressure on transcriptome-wide RNA expression in skeletal muscle.LINC00341 exerts an anti-inflammatory effect on endothelial cells by repressing VCAM1.Loss of Cdk5 function in the nucleus accumbens decreases wheel running and may mediate age-related declines in voluntary physical activity.Left ventricle transcriptomic analysis reveals connective tissue accumulation associates with initial age-dependent decline in V̇o2peak from its lifetime apex.Understanding Key Mechanisms of Exercise-Induced Cardiac Protection to Mitigate Disease: Current Knowledge and Emerging Concepts.AMPK agonist AICAR delays the initial decline in lifetime-apex V̇o2 peak, while voluntary wheel running fails to delay its initial decline in female rats.
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Deep RNA sequencing reveals novel cardiac transcriptomic signatures for physiological and pathological hypertrophy
description
2012 nî lūn-bûn
@nan
2012 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Deep RNA sequencing reveals no ...... l and pathological hypertrophy
@ast
Deep RNA sequencing reveals no ...... l and pathological hypertrophy
@en
Deep RNA sequencing reveals no ...... l and pathological hypertrophy
@nl
type
label
Deep RNA sequencing reveals no ...... l and pathological hypertrophy
@ast
Deep RNA sequencing reveals no ...... l and pathological hypertrophy
@en
Deep RNA sequencing reveals no ...... l and pathological hypertrophy
@nl
prefLabel
Deep RNA sequencing reveals no ...... l and pathological hypertrophy
@ast
Deep RNA sequencing reveals no ...... l and pathological hypertrophy
@en
Deep RNA sequencing reveals no ...... l and pathological hypertrophy
@nl
P2093
P2860
P1433
P1476
Deep RNA sequencing reveals no ...... l and pathological hypertrophy
@en
P2093
Do Han Kim
Hong Ki Song
Seong-Eui Hong
Taeyong Kim
P2860
P304
P356
10.1371/JOURNAL.PONE.0035552
P407
P577
2012-04-16T00:00:00Z