Rare chromosomal deletions and duplications in attention-deficit hyperactivity disorder: a genome-wide analysis. - Wikidata - wikimedia - TriplyDB

Rare chromosomal deletions and duplications in attention-deficit hyperactivity disorder: a genome-wide analysis.

Rare chromosomal deletions and duplications in attention-deficit hyperactivity disorder: a genome-wide analysis.

http://www.wikidata.org/entity/Q34247422

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Parent training interventions for Attention Deficit Hyperactivity Disorder (ADHD) in children aged 5 to 18 years The shock of the new: progress in schizophrenia genomics What causes attention deficit hyperactivity disorder?What have we learnt about the causes of ADHD?The genetics of attention deficit/hyperactivity disorder in adults, a review CNV analysis in a large schizophrenia sample implicates deletions at 16p12.1 and SLC1A1 and duplications at 1p36.33 and CGNL1 The neurobiological basis of ADHD Moving towards causality in attention-deficit hyperactivity disorder: overview of neural and genetic mechanisms Genetics and genomics of psychiatric disease Synaptic plasticity, neural circuits, and the emerging role of altered short-term information processing in schizophrenia.Following the genes: a framework for animal modeling of psychiatric disorders Toward developmental models of psychiatric disorders in zebrafish Child development and molecular genetics: 14 years later Raising attention to attention deficit hyperactivity disorder in schizophrenia Genome-wide copy number variation study associates metabotropic glutamate receptor gene networks with attention deficit hyperactivity disorder Novel Findings from CNVs Implicate Inhibitory and Excitatory Signaling Complexes in Schizophrenia The newly identified migration inhibitory protein regulates the radial migration in the developing neocortex Rare Copy Number Variants Identified Suggest the Regulating Pathways in Hypertension-Related Left Ventricular Hypertrophy The Danish 22q11 research initiative The Growing Importance of CNVs: New Insights for Detection and Clinical Interpretation 'Sifting the significance from the data' - the impact of high-throughput genomic technologies on human genetics and health care CNVs leading to fusion transcripts in individuals with autism spectrum disorder Variations of the candidate SEZ6L2 gene on Chromosome 16p11.2 in patients with autism spectrum disorders and in human populations Polygenic transmission and complex neuro developmental network for attention deficit hyperactivity disorder: genome-wide association study of both common and rare variants.Genome-wide association study in German patients with attention deficit/hyperactivity disorder.Detection and correction of artefacts in estimation of rare copy number variants and analysis of rare deletions in type 1 diabetes.Analysis of shared homozygosity regions in Saudi siblings with attention deficit hyperactivity disorder.DCLK1 variants are associated across schizophrenia and attention deficit/hyperactivity disorder Deletions at 22q11.2 in idiopathic Parkinson's disease: a combined analysis of genome-wide association data.DRD4 rare variants in Attention-Deficit/Hyperactivity Disorder (ADHD): further evidence from a birth cohort study.Genomic structural variation in psychiatric disorders.De novo CNV analysis implicates specific abnormalities of postsynaptic signalling complexes in the pathogenesis of schizophrenia.Italian regional health service costs for diagnosis and 1-year treatment of ADHD in children and adolescents.The RNA-binding protein MARF1 promotes cortical neurogenesis through its RNase activity domain Electrophysiological markers of genetic risk for attention deficit hyperactivity disorder Genomic insights into the overlap between psychiatric disorders: implications for research and clinical practice.The Four Causes of ADHD: Aristotle in the Classroom.Biological overlap of attention-deficit/hyperactivity disorder and autism spectrum disorder: evidence from copy number variants Intellectual disability in children with attention deficit hyperactivity disorder.Use of clinical chromosomal microarray in Chinese patients with autism spectrum disorder-implications of a copy number variation involving DPP10

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Rare chromosomal deletions and duplications in attention-deficit hyperactivity disorder: a genome-wide analysis.

http://www.wikidata.org/entity/Q34247422

description

2010 nî lūn-bûn

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2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Rare chromosomal deletions and ...... order: a genome-wide analysis.

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Rare chromosomal deletions and ...... order: a genome-wide analysis.

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Rare chromosomal deletions and ...... order: a genome-wide analysis.

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Rare chromosomal deletions and ...... order: a genome-wide analysis.

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Rare chromosomal deletions and ...... order: a genome-wide analysis.

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Rare chromosomal deletions and ...... order: a genome-wide analysis.

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Irina Zaharieva

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Kiran Mantripragada

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Michael J Owen

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Nigel M Williams

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Olafur O Gudmundsson

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Omar Gustafsson

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Pall Magnusson

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Ragnheidur Fossdal

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P2860

Advances in autism genetics: on the threshold of a new neurobiology

Copy-number variations associated with neuropsychiatric conditions

Rare chromosomal deletions and duplications increase risk of schizophrenia

Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls

Rare structural variants found in attention-deficit hyperactivity disorder are preferentially associated with neurodevelopmental genes

Autism genome-wide copy number variation reveals ubiquitin and neuronal genes

Microduplications of 16p11.2 are associated with schizophrenia

Disruption of neurexin 1 associated with autism spectrum disorder.

Contribution of SHANK3 mutations to autism spectrum disorder

Large recurrent microdeletions associated with schizophrenia

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Hreinn Stefansson

Michael C O'Donovan

Kári Stefánsson

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attention deficit hyperactivity disorder

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