Synergistic epigenetic reactivation of estrogen receptor-α (ERα) by combined green tea polyphenol and histone deacetylase inhibitor in ERα-negative breast cancer cells
about
Epigenetic regulation of caloric restriction in agingEpigenetic effects of green tea polyphenols in cancerSuppressive Effects of Tea Catechins on Breast CancerPhytochemicals modulate carcinogenic signaling pathways in breast and hormone-related cancersEpigenetic diet: impact on the epigenome and cancerBetter Living through Chemistry: Caloric Restriction (CR) and CR Mimetics Alter Genome Function to Promote Increased Health and LifespanDietary phytochemicals, HDAC inhibition, and DNA damage/repair defects in cancer cellsSynergistic Anticancer Effects of Vorinostat and Epigallocatechin-3-Gallate against HuCC-T1 Human Cholangiocarcinoma Cells.ERRβ signalling through FST and BCAS2 inhibits cellular proliferation in breast cancer cells.Effects of physiological levels of the green tea extract epigallocatechin-3-gallate on breast cancer cellsBreast cancer chemopreventive and chemotherapeutic effects of Camellia Sinensis (green tea): an updated reviewZ-ligustilide restores tamoxifen sensitivity of ERa negative breast cancer cells by reversing MTA1/IFI16/HDACs complex mediated epigenetic repression of ERaBioactive dietary supplements reactivate ER expression in ER-negative breast cancer cells by active chromatin modificationsGreen tea and breast cancer.Epigenetic regulation by selected dietary phytochemicals in cancer chemoprevention.Epigenetic linkage of aging, cancer and nutrition.Epigenetic regulation of multiple tumor-related genes leads to suppression of breast tumorigenesis by dietary genisteinCurrent and upcoming approaches to exploit the reversibility of epigenetic mutations in breast cancer.Epigenetic therapy for breast cancer.Epigenetics in breast and prostate cancerEpigenetic-based combinatorial resveratrol and pterostilbene alters DNA damage response by affecting SIRT1 and DNMT enzyme expression, including SIRT1-dependent γ-H2AX and telomerase regulation in triple-negative breast cancerPhytochemical antioxidants modulate mammalian cellular epigenome: implications in health and disease.The transposon-driven evolutionary origin and basis of histone deacetylase functions and limitations in disease prevention.A Novel Combinatorial Epigenetic Therapy Using Resveratrol and Pterostilbene for Restoring Estrogen Receptor-α (ERα) Expression in ERα-Negative Breast Cancer Cells.Oestrogen receptors interact with the α-catalytic subunit of AMP-activated protein kinase.EGCG prevents PCB-126-induced endothelial cell inflammation via epigenetic modifications of NF-κB target genes in human endothelial cells.Epigenetic reactivation of estrogen receptor-α (ERα) by genistein enhances hormonal therapy sensitivity in ERα-negative breast cancer.Natural compound-derived epigenetic regulators targeting epigenetic readers, writers and erasers.Going Green: The Role of the Green Tea Component EGCG in Chemoprevention.Cell cycle regulators cyclin D1 and CDK4/6 have estrogen receptor-dependent divergent functions in breast cancer migration and stem cell-like activityAberrant promoter CpG methylation and its translational applications in breast cancer.Epigenetic reactivation of p21CIP1/WAF1 and KLOTHO by a combination of bioactive dietary supplements is partially ERα-dependent in ERα-negative human breast cancer cells.Green tea and anticancer perspectives: updates from last decade.Synergistic enhancement of anticancer effects on numerous human cancer cell lines treated with the combination of EGCG, other green tea catechins, and anticancer compounds.Impact of Epigenetic Dietary Components on Cancer through Histone Modifications.Histone and Non-Histone Targets of Dietary Deacetylase Inhibitors.Epigallocatechin-3-gallate inhibits growth and induces apoptosis in esophageal cancer cells through the demethylation and reactivation of the p16 gene.Dietary phytochemicals as epigenetic modifiers in cancer: Promise and challenges.Histone deacetylase inhibitors promote the expression of ATP2A3 gene in breast cancer cell lines.Chronic oxidative stress causes estrogen-independent aggressive phenotype, and epigenetic inactivation of estrogen receptor alpha in MCF-7 breast cancer cells.
P2860
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P2860
Synergistic epigenetic reactivation of estrogen receptor-α (ERα) by combined green tea polyphenol and histone deacetylase inhibitor in ERα-negative breast cancer cells
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Synergistic epigenetic reactiv ...... α-negative breast cancer cells
@ast
Synergistic epigenetic reactiv ...... α-negative breast cancer cells
@en
Synergistic epigenetic reactivation of estrogen receptor-α
@nl
type
label
Synergistic epigenetic reactiv ...... α-negative breast cancer cells
@ast
Synergistic epigenetic reactiv ...... α-negative breast cancer cells
@en
Synergistic epigenetic reactivation of estrogen receptor-α
@nl
prefLabel
Synergistic epigenetic reactiv ...... α-negative breast cancer cells
@ast
Synergistic epigenetic reactiv ...... α-negative breast cancer cells
@en
Synergistic epigenetic reactivation of estrogen receptor-α
@nl
P2093
P2860
P356
P1433
P1476
Synergistic epigenetic reactiv ...... α-negative breast cancer cells
@en
P2093
Syed M Meeran
Trygve O Tollefsbol
Yih-Ying Yuan
Yuanyuan Li
P2860
P2888
P356
10.1186/1476-4598-9-274
P577
2010-10-14T00:00:00Z
P5875
P6179
1024795555