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The ploidy conveyor of mature hepatocytes as a source of genetic variation.

The ploidy conveyor of mature hepatocytes as a source of genetic variation.

http://www.wikidata.org/entity/Q34259413

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Cell Fusion in the War on Cancer: A Perspective on the Inception of Malignancy Wound-Induced Polyploidy Is Required for Tissue Repair Different Facets of Copy Number Changes: Permanent, Transient, and Adaptive Tissue Regeneration in the Chronically Inflamed Tumor Environment: Implications for Cell Fusion Driven Tumor Progression and Therapy Resistant Tumor Hybrid Cells Genome management and mismanagement--cell-level opportunities and challenges of whole-genome duplication Endoreplication and polyploidy: insights into development and disease Centrosomes, chromosome instability (CIN) and aneuploidy A novel source of cultured podocytes Increased asymmetric and multi-daughter cell division in mechanically confined microenvironments A tetraploid intermediate precedes aneuploid formation in yeasts exposed to fluconazole Essential role for a novel population of binucleated mammary epithelial cells in lactation.Phyllanthus emblica Fruit Extract Activates Spindle Assembly Checkpoint, Prevents Mitotic Aberrations and Genomic Instability in Human Colon Epithelial NCM460 Cells.E2F8 is essential for polyploidization in mammalian cells TGFbeta Induces Binucleation/Polyploidization in Hepatocytes through a Src-Dependent Cytokinesis Failure Degeneracy allows for both apparent homogeneity and diversification in populations Interphase cytofission maintains genomic integrity of human cells after failed cytokinesis.Chromosome missegregation rate predicts whether aneuploidy will promote or suppress tumors.Incomplete cytokinesis and re-fusion of small mononucleated Hodgkin cells lead to giant multinucleated Reed-Sternberg cells.Ploidy variation in multinucleate cells changes under stress.Transient endoreplication down-regulates the kinesin-14 HSET and contributes to genomic instability.Somatic polyploidy is associated with the upregulation of c-MYC interacting genes and EMT-like signature Susceptibility to Plasmodium liver stage infection is altered by hepatocyte polyploidy Three steps to the immortality of cancer cells: senescence, polyploidy and self-renewal.Deletion of the MAD2L1 spindle assembly checkpoint gene is tolerated in mouse models of acute T-cell lymphoma and hepatocellular carcinoma.Non-viral FoxM1 gene delivery to hepatocytes enhances liver repopulation.Mechanisms and consequences of aneuploidy and chromosome instability in the aging brain Rapid growth of a hepatocellular carcinoma and the driving mutations revealed by cell-population genetic analysis of whole-genome data Use of array CGH to detect exonic copy number variants throughout the genome in autism families detects a novel deletion in TMLHE Clonal evolution through loss of chromosomes and subsequent polyploidization in chondrosarcoma.Single-cell analysis of ploidy and centrosomes underscores the peculiarity of normal hepatocytes.Variation in chromosome copy number influences the virulence of Cryptococcus neoformans and occurs in isolates from AIDS patients.p53 dependent centrosome clustering prevents multipolar mitosis in tetraploid cells Single cell sequencing reveals low levels of aneuploidy across mammalian tissues.Principles of liver regeneration and growth homeostasis.Bursty gene expression in the intact mammalian liver Three-dimensional genome architecture: players and mechanisms.Controlled induction of DNA double-strand breaks in the mouse liver induces features of tissue ageing.Restricted diet delays accelerated ageing and genomic stress in DNA-repair-deficient mice In vitro alterations do not reflect a requirement for host cell cycle progression during Plasmodium liver stage infection.New concepts in liver regeneration.

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The ploidy conveyor of mature hepatocytes as a source of genetic variation.

http://www.wikidata.org/entity/Q34259413

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

The ploidy conveyor of mature hepatocytes as a source of genetic variation.

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The ploidy conveyor of mature hepatocytes as a source of genetic variation.

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The ploidy conveyor of mature hepatocytes as a source of genetic variation.

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type

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The ploidy conveyor of mature hepatocytes as a source of genetic variation.

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The ploidy conveyor of mature hepatocytes as a source of genetic variation.

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The ploidy conveyor of mature hepatocytes as a source of genetic variation.

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The ploidy conveyor of mature hepatocytes as a source of genetic variation.

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The ploidy conveyor of mature hepatocytes as a source of genetic variation.

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The ploidy conveyor of mature hepatocytes as a source of genetic variation.

@nl

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The ploidy conveyor of mature hepatocytes as a source of genetic variation

@en

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Amy E Hanlon Newell

http://www.w3.org/2001/XMLSchema#string

Andrew W Duncan

http://www.w3.org/2001/XMLSchema#string

Matthew H Taylor

http://www.w3.org/2001/XMLSchema#string

Michelle L Lenzi

http://www.w3.org/2001/XMLSchema#string

Milton J Finegold

http://www.w3.org/2001/XMLSchema#string

Raymond D Hickey

http://www.w3.org/2001/XMLSchema#string

Susan B Olson

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Cyclin A is required at two points in the human cell cycle

Promoter traps in embryonic stem cells: a genetic screen to identify and mutate developmental genes in mice

A mechanism linking extra centrosomes to chromosomal instability

Ploidy reductions in murine fusion-derived hepatocytes.

In vivo suppressor mutations correct a murine model of hereditary tyrosinemia type I.

Some flow-cytofluorimetric studies of the nuclear ploidy of mouse hepatocytes: iii. further observations on early changes in nuclear ploidy of mouse hepatocytes following various experimental procedures.

Fluorescence in situ Hybridization (FISH).

Widespread monoallelic expression on human autosomes.

The mutagenicity of the tyrosine metabolite, fumarylacetoacetate, is enhanced by glutathione depletion.

Aneuploidy underlies rapid adaptive evolution of yeast cells deprived of a conserved cytokinesis motor.

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707-710

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scholarly article

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10.1038/NATURE09414

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467

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1044509580

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20861837

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2967727

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