Overexpression of DDB2 enhances the sensitivity of human ovarian cancer cells to cisplatin by augmenting cellular apoptosis.
about
Genome-wide modulation of gene transcription in ovarian carcinoma cells by a new mithramycin analogueNRIP/DCAF6 stabilizes the androgen receptor protein by displacing DDB2 from the CUL4A-DDB1 E3 ligase complex in prostate cancer.Damaged DNA-binding protein 2 (DDB2) protects against UV irradiation in human cells and Drosophila.DDB2 suppresses tumorigenicity by limiting the cancer stem cell population in ovarian cancer.Damaged DNA-binding protein down-regulates epigenetic mark H3K56Ac through histone deacetylase 1 and 2.Differential contributory roles of nucleotide excision and homologous recombination repair for enhancing cisplatin sensitivity in human ovarian cancer cellsCombination of Aβ Secretion and Oxidative Stress in an Alzheimer-Like Cell Line Leads to the Over-Expression of the Nucleotide Excision Repair Proteins DDB2 and XPCDDB2 modulates TGF-β signal transduction in human ovarian cancer cells by downregulating NEDD4L.Damaged DNA binding protein 2 in reactive oxygen species (ROS) regulation and premature senescenceNucleotide excision repair deficiency in melanoma in response to UVA.Differential DNA lesion formation and repair in heterochromatin and euchromatinDNA damage-binding complex recruits HDAC1 to repress Bcl-2 transcription in human ovarian cancer cells.Stem cell protein Piwil2 modulates chromatin modifications upon cisplatin treatment.A DDB2 mutant protein unable to interact with PCNA promotes cell cycle progression of human transformed embryonic kidney cells.Single nucleotide polymorphisms of nucleotide excision repair pathway are significantly associated with outcomes of platinum-based chemotherapy in lung cancer.Cdt2-mediated XPG degradation promotes gap-filling DNA synthesis in nucleotide excision repair.Damaged-DNA Binding Protein-2 Drives Apoptosis Following DNA Damage.DDB2 association with PCNA is required for its degradation after UV-induced DNA damage.Human CRL4DDB2 ubiquitin ligase preferentially regulates post-repair chromatin restoration of H3K56Ac through recruitment of histone chaperon CAF-1.DDB2 represses ovarian cancer cell dedifferentiation by suppressing ALDH1A1.
P2860
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P2860
Overexpression of DDB2 enhances the sensitivity of human ovarian cancer cells to cisplatin by augmenting cellular apoptosis.
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2010 nî lūn-bûn
@nan
2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
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name
Overexpression of DDB2 enhance ...... augmenting cellular apoptosis.
@ast
Overexpression of DDB2 enhance ...... augmenting cellular apoptosis.
@en
Overexpression of DDB2 enhance ...... augmenting cellular apoptosis.
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type
label
Overexpression of DDB2 enhance ...... augmenting cellular apoptosis.
@ast
Overexpression of DDB2 enhance ...... augmenting cellular apoptosis.
@en
Overexpression of DDB2 enhance ...... augmenting cellular apoptosis.
@nl
prefLabel
Overexpression of DDB2 enhance ...... augmenting cellular apoptosis.
@ast
Overexpression of DDB2 enhance ...... augmenting cellular apoptosis.
@en
Overexpression of DDB2 enhance ...... augmenting cellular apoptosis.
@nl
P2093
P2860
P356
P1476
Overexpression of DDB2 enhance ...... augmenting cellular apoptosis.
@en
P2093
Altaf A Wani
Bassant M Barakat
Chunhua Han
De-Tao Yin
El-Shaimaa A Arafa
Gulzar Wani
Keisha Milum
Mohamed A El-Mahdy
Qi-En Wang
P2860
P304
P356
10.1002/IJC.25112
P577
2010-08-01T00:00:00Z