A quantitative study of the mechanisms behind thymic atrophy in Gαi2-deficient mice during colitis development
about
Maturation and migration of murine CD4 single positive thymocytes and thymic emigrantsLoss of Gαi proteins impairs thymocyte development, disrupts T-cell trafficking, and leads to an expanded population of splenic CD4+PD-1+CXCR5+/- T-cellsToward quantifying the thymic dysfunctional state in mouse models of inflammatory bowel disease.The regulating function of heterotrimeric G proteins in the immune system.The impact of RGS and other G-protein regulatory proteins on Gαi-mediated signaling in immunity.
P2860
A quantitative study of the mechanisms behind thymic atrophy in Gαi2-deficient mice during colitis development
description
2012 nî lūn-bûn
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2012 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի մայիսին հրատարակված գիտական հոդված
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2012年の論文
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2012年論文
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2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
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2012年論文
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2012年论文
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name
A quantitative study of the me ...... ice during colitis development
@ast
A quantitative study of the me ...... ice during colitis development
@en
A quantitative study of the me ...... ice during colitis development
@nl
type
label
A quantitative study of the me ...... ice during colitis development
@ast
A quantitative study of the me ...... ice during colitis development
@en
A quantitative study of the me ...... ice during colitis development
@nl
prefLabel
A quantitative study of the me ...... ice during colitis development
@ast
A quantitative study of the me ...... ice during colitis development
@en
A quantitative study of the me ...... ice during colitis development
@nl
P2093
P2860
P1433
P1476
A quantitative study of the me ...... ice during colitis development
@en
P2093
Andreas Jansson
Elisabeth Hultgren-Hörnquist
Kristina Elgbratt
P2860
P304
P356
10.1371/JOURNAL.PONE.0036726
P407
P577
2012-05-10T00:00:00Z